ECG Flashcards
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Rate
Rhythm
Axis
Ischaemia
Intervals
Others
R R intervals
300 / large box
P wave = SR
Axis = I and aVF
Territories:
Q S
V1 V2 septal
V3 4 anterior
V5 6 lateral
aVL I lateral
II III aVF
Interval
PR 1 box
QRS < 120ms
QT 440 460
J point
STE
S V2
R V5
LVH
Concentric hypertrophy
Rate: 80-85
Rhythm: SR
Axis: Normal
Ischaemia:
II III aVF - STEMI
Q wave: 6-8 hrs to develop = transmural infarction
Inferolateral STE - reperfusion - infarct
Q wave preceding STE - subacute infact
Reciprocal changes in anterior (V1 V2) lateral (avL) lead
Bottom strip
Mobitz I PR progressively increading
RCA infarct - supplies purkinje
- STE III > II + STD aVL - favours RCA infarct
- SII > III - favour Circumflex
Rate: 75
Rhythm: SR
Axis: LAD
Ischaemia: STE V2-V4 V5-V6
TWI III V1
Tombstone STE across praecordium
Q wave
V1 - widening QRS - efficiency of depolarisation
V1 Positive deflection - RBBB, posterior infarct, RVH/strain, WPW
Rate: 100, SR, normal
STE
Saddle shape morphology
PR
High takeoff V2-V6
Pleuritic CP
PR depression - pericardial
PERICARDIAL effusion
Rate: irregular >100 (120)
Rhythm: irregular, relationship P to QRS
V1 - P but does not mean SR
Irregular irregular
HTN –> LVH –> LVEDP inc –> atrial strain –> remodel –> AF
Degenerative
Nothing to ablate
3 P waves for every QRS
Fixed relationship
Macro Reentract Circuit
Ablatable / Cardiovert
Hard to control rate
AF rules
- Find the cause
- infection/TFT/ischaemia - Fix the cause
- Control rate
- anticoagulate
CHADVASc 2 = 2% / annum
No bridging required
V1 RSR TWI
V2
SR
Normal axis
Widened QRS in V2
RBBB
RBBB + LAD
LAFB
RBBB + RAD
LPFB
Bifascicular block
RV strain
PE
PHTN
Brugada: electrical condition ~ SCD
Different morphology of STE
Downsloping pattern
SR
Broadened QRS
LAD
Broad complex tachycardia
Monomorphic VT
RV OT VT
Narrow complex tachy
Rate: 150
AVNRT
Treat with adenosine
SVT - include AF/A flutter
Look for retrograde P wave
CHB
Complete dissociation P and QRS
2ND degree AV block
WPW
slurred upstroke
VT
HoCM
J point
Diffuse deep TWI across V1-V6
