DM ppt Flashcards
DM risk factors
genetics obesity race/ethnicity age HTN high HDL gestational diabetes, baby >9 lbs
DM 1
FKA brittle diabetes, juvenile diabetes
beta cells in panrease are destroyed
decreased insulin production and unchecked glucose production
5% of DM
DM 2
95% of DM
most common in obese >30 y/o
insulin resistance
impaired insulin secretion
gestational diabetes
hyperglycemia develops with pregnancy 2/2 secretion of placental hormones
r/o HTN during pregnancy
highest risk: obesity, Hx of gestational diabetes, glycosuria, genetics
latent autoimmune diabetes of adults (LADA)
progression of autoimmune beta cell destruction is slower than in DM1/DM2
not insulin dependent initially
3 Ps
polyuria
polydipsia
polyphagia
DM diagnostic findings
casual glucose reading > 200mg/dL
flasting glucose >126 mg/dL
greater than 200mg/dL in oral glucose tolerance
A1C > 6.5%
DM medical mgmt
A1C < 7% nutritional therapy exercise monitoring medication educatoin
nutritional therapy
control of total caloric intake to maintain or attain reasonable body weight
control blood glucose levels
meal planning
maintain pleasure of eating
meal planning
carbs: 50-60%
fat: 30%
cholesterol: <300mg
non animal sources of protein
rapid acting insulin
shorter duration
eat within 5-15 min
short acting insulin
clear solution
given 15 min prior to meal
can be given IV
intermediate acting insulin
NPH or Lente
white and cloudy soltuion
should eat food around onset/peak
long acting “peakless” insulin
absorbed over 24 hrs
given once daily at the same time
conventional insulin therapy
pt should not vary meal patterns 1-4 injections qd appropriate for: terminally ill frail older adult with self-care limitations pts unwilling to do self mgmt