Diabetes Care Plan Flashcards

1
Q

In hospital blood glucose target range

A

5-10 mmol/L

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2
Q

If clinicians fail to recognize adn treat hyperglycemia appropriately patients with diabetes are at risk for

A

-delayed wound healing
-surgical site infections
-hospital acquired infections
-mortality

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3
Q

1 in 5 patients in hospital have diabetes, what is the percentage of type 1 vs type 2

A

Type 2 - 90%

Type 1 - 10%

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4
Q

Hyperglycemia is defined as

A

Blood glucose levels above 10 mmol/L

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5
Q

What is BBIT

A

B- basal insulin
B- bonus insulin
I- insulin correction
T- titrate

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6
Q

Basal insulin

A

Covers the glucose, the liver makes around the clock

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7
Q

Bonus insulin

A

Covers the meal time carbohydrates

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8
Q

Insulin correction

A

Corrects the patients BG back into target range if it is high

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9
Q

Titrate

A

Every patient is different, the BG needs to be checked regularly and insulin doses adjusted every 1-3 days

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10
Q

Pancreas in normal physiology

A

Produces enzymes that digests fats, proteins, and carbohydrates in the food that is consumed

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11
Q

Pancreas endocrine function

A

Produces hormone: insulin (beta cells) and glucagon (alpha cells)

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12
Q

Beta cells

A

Sense glucose levels in the blood when BG rises
-secret insulin and inhibit glucagon

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13
Q

Alpha cells

A

Detect when BG is low
-insulin is inhibited, glucagon is secreted

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14
Q

Insulin

A

Helps cells in our body to take up glucose to be used for energy, or store excess glucose in form of glycogen

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15
Q

Glucagon

A

Increases amount of glucose in bloodstream
-stimulates liver to produce glucose
-breakdown of glycogen into glucose

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16
Q

Type 1 diabetes mellitus is caused by

A

Caused by destruction of insulin producing beta cells, from autoimmune process

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17
Q

Type 1 DM pancreas

A

Produces very little or no insulin since beta cells are destroyed
-so blood glucose rises

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18
Q

What happens in type 1 DM when body cannot break down glucose as an energy source?

A

The body breaks down fat, and produces ketones which are very acidic

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19
Q

High levels of ketones can lead to

A

Diabetic ketoacidosis

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20
Q

What do people with T1DM need to survive

A

-basal bonus insulin regimen

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21
Q

People with T1DM tend to be less

A

Insulin resistant

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22
Q

People with T1DM have a higher risk of

A

Developing severe hypoglycaemia

-where beta cells no longer work in conjunction with alpha cells that produce glucagon

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23
Q

Symptoms of hyperglycemia in T1DM

A

-fatigue
-frequent urination
-dehydration
-thirst
-weight loss despite hunger

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24
Q

T2DM is a

A

Progressive chronic disease

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25
T2DM pancreas
The pancreas produces some insulin, but cells in the body fail tor respond to the insulin properly -insulin resistance
26
T2DM insulin resistance
Pancreas often cannot produce enough insulin to overcome this resistance without treatment
27
T2DM have a lower risk of
Severe hypoglycaemia -since the pancreas is still able to make glucagon
28
What is different from T1DM hyperglycemia and T2DM hyperglycemia
Many people may be asymptomatic
29
Initially when diagnosed with T2DM what is an important part of treatment?
Diet and exercise
30
Basal insulin
Serves to suppress glucose and ketone production in periods of fasting -relatively constant
31
Basal insulin is produced by
Liver and to a lesser extent the kidneys
32
Bolus insulin
After meals, insulin levels rise above the basal level in response to the increase in blood glucose from ingested and absorbed carbohydrate
33
What is the 50/50 rule
Basal insulin accounts for about 50% of the total daily insulin secretion and remaining 50% is secreted as bolus insulin
34
How often is basal insulin ordered to cover needs, and how long does the insulin act
Once or twice daily, and is a long/intermediate acting insulin
35
Patients with __
May require basal insulin
36
Bolus meal insulin length of action and when is it given
-short acting insulin -given prior to meals
37
Correction insulin length, and when is it given
-rapid, short acting insulin -given to correct unanticipated hyperglycemia
38
Correction insulin ensures
High glucose values are not left untreated
39
Goal of basal
Replace the background insulin the body naturally makes with basal long acting insulin
40
Goal of bolus
To replace natural mealtime insulin with bolus short acting insulin to cover carb intake
41
Rapid acting insulin analogues (2)
Aspart and lisp r o
42
Onset of aspart/lispro
Aspart - 10 to 15min Lispro - 10 to 15min
43
Peak of aspart and lispro
Aspart - 1 to 1.5h Lispro - 1 to 2h
44
Duration of aspart and lispro
Aspart - 3 to 5h Lispro - 3.5 to 4.75h
45
Short acting bolus insulin (1)
Regular
46
Onset of regular
30min
47
Peak of regular
2 to 3h
48
Duration of regular
6.5h
49
Basal intermediate acting insulin (1)
NPH
50
Onset of NPH
1 to 3h
51
Peak of NPH
5 to 8h
52
Duration of NPH
Up to 18h
53
Long lasting basal insulin (2)
Detemir and glargine
54
Onset of detemir and glargine
Detemir - 90min Glargine - 90min
55
Peak of detemir and glargine
Detemir - not applicable Glargine - not applicable
56
Duration of detemir and glargine
Detemir - 16 to 24h Glargine - 24h
57
What color are long acting basal insulin
Clear
58
What color is Intermediate acting insulin
Cloudy
59
What color is bolus rapid acting insulin
Clear
60
What color is Bolus short acting insulin
Clear
61
Bolus can be
Rapid acting or short acting
62
Basal can be
Intermediate acting or long acting