Dermatology First Aid Flashcards

1
Q

chronic inflammatory skin dz that may be caused by hypersensitivity reaction to Malassezia furfur?

A

Seborrheic dermatitis

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2
Q

treatments for seborrheic dermatitis?

A
  1. selenium sulfate or zinc pyrithione shampoos for the scalp
  2. topical antifungal (ketoconazole)
  3. topical corticosteroid
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3
Q

characteristics of basal cell carcinoma?

A

slow growing, pearly, telangiectatic nodular lesion with rolled borders

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4
Q

clinical presentation of erythema multiforme?

A

target lesions on the palms, soles, and extensor

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5
Q

mech of erythema multiforme?

A

hypersensitivity reaction to a drug, drug metabolite, infectious trigger

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6
Q

What are the 6 ps of Lichen planus

A
Planar
Purple
Polygonal
Pruritic
Papules
Plaques
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7
Q

what path is Lichen planus associated with?

A

hep C (also may induced by thiazides, quinines, beta blockers)

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8
Q

what kind of lines/striae do you see from Lichen planus?

A

Wickham striae (lacy white lines)

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9
Q

what are the most common causative organisms for dermatophyte (Tinea) infection?

A
  1. Trichophyton
  2. Microsporum
  3. Epidermophyton
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10
Q

what is the rules of 9 for body surface area?

A

head and each arm = 9%

back and chest each = 18%

Each leg = 18%

Perneum = 1%

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11
Q

what causes Tinea versicolor?

A

Malassezia spp (Pityrosporum spp.), not dermatophyte despite being called tinea)

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12
Q

what is the most effective treatment for severe psoriasis?

A

methotrexate

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13
Q

side effects for methotrexate?

A

hepatic fibrosis, cirrhosis, leukopenia, anemia, thrombocytopenia, abdominal pain, fatigue, and impaired memory.

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14
Q

skin characteristics of pityriasis rosea?

A

round to oval erthematous plaques covered with fine white scale (cigarette papaer) and often found on the trunk and proximal extremities

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15
Q

treatments for pityriasis rosea?

A

supportive (heals in 6-8 wks without any treatment)

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16
Q

clinical presentations of eythema nodosum?

A

1, painful, erythematous nodules on the pt’s anterior shins and slowly spread, turning brown or gray

  1. pretibial erythematous, tender nodules in a young woman
17
Q

3 common causes for erythema nodosum?

A
  1. MCC: idiopathic
  2. strep pharyngitis
  3. hypersensitivity rxn to drugs (oral contraceptive, NSAIDs)
  4. sarcoidosis, TB, IBD
18
Q

how is SJS different from TEN?

A

the epidermal separation of SJS involves less than 10% of body surface area, whereas in TEN involves more than 30%

19
Q

what are the classical presentations of Stevens Johnson syndrome?

A

flu like prodrome followed by acute fever, typical rash, and ulcerated lesions on at least two mucous membranes

20
Q

clinical presentation of 3rd degree burn?

A

area is painless, white, and charred

21
Q

clinical presentation of 2nd degree burn?

A

the area is painful and blisters present

22
Q

clinical presentation of 1st degree burn?

A

no blisters, capillary refill is intact, looks like sunburn

23
Q

what antibodies are developed in pemphigus vulgaris?

A

intraepidermal desmogleins 1 and 3

24
Q

treatments for pemphigus vulgaris?

A

high dose steroids (prednisone) + immunomodulatory (azathioprine, mycophenolate mofetil, IVIG, rituximab)

25
Q

clinical presentations of Urticaria (Hives)?

A

common dermatologic problems consisting of pruritic edematous papules varying in size that appear/disappear in a matter hours

26
Q

clinical presentation of schistosomiasis?

A

signs of cirrhosis, ascited, ankle edema, organomegaly, caput medusae

27
Q

characteristics of chronic schistosomiasis?

A

eosinophilia, granuloma formation in tissues, peri-portal hepatic fibrosis

28
Q

treatment drug for schistosomiasis?

A

praziquantel

29
Q

choice of antibiotic used along with penicillin or vancomycin for the initial treatment of native valve bacterial endocarditis?

A

gentamicin