CVA Flashcards
Risk Factors
HTN, Smoking , Obesity, SM, sedentary lifestyle, contreseptives w/ estrogen
OT push for primary care (lifestyle redesign)
CVA Survivors
10 have no impairment
40 have mild residual deficits
40 have long teem disabilities
10 need instutional care
Stroke Types: Ischemic
(Blockage in artery by clot, 87%)
Thrombotic: Stationary Clot
Embolic: traveling clot
Stroke Types: Hemorrhagic
Weakened vessel ruptures 13%
Usually due to increased HTN results in major surgery
Stroke Types: Transient Ischemic Attack
(TIA)
Sx. Resolve in 24 Hrs.
Lacunar Infarct : Deficits
Small holes
Pure motor ataxic of sensory loss
Good prognosis
Anterior Cerebral Artery Infarct: ACA Deficits
Contralateral UE hemiparesis Sensory loss LE > UR or face LE Paralysis Frontal lobe damage Apraxia Aphasia Neglect **Sx. also include denile, decreased safety awarnesss, insight, propioseptive input in BLE Apraxia is noted by hesitation use hand over hand A Use blocked practice
Middle Cerebral Artery : MCA Deficits
Most common, more complicated, tx involves a lot of sensory input *Vision screens are very important Sensory loss of face and Ue > LE Contralateral hemiplegia Homonymous Hemianopsia (loss of 1/3 of visual) Aphasia Apraxia *Body scheme *Spatial Relations *Agnosia *Impulsivity
Posterior Cerebral Artery Infarct: PCA Deficits
More rare **Contralateral sensory loss homonyous hemianopsia contralatral hemiplegia
Vertebral-Basilar Artery: VBA Deficits
Locked in syndrome LOC Brainstem or cranial nerve damage hemi or quadriplegia memory loss agitation comatose or vegetative state
Acute Medical Management
Airway protection
Restore cerebral circulation : thrombolytic therapy, antiplatelet drugs, anticoagulation drugs, neuroprotective agents
Maintain BP
Correct Anemia
The Hemi Arm: Decreased postural tone and control
Can be weakness or sensory based
Hemi Arm: Decreased biomechanical alignment
Can be either increased or decreased tone which affect scapular alignment
Hemi Arm: Inefficent muscle recruitment
Either increased or decreased tone
Hemi Arm: Decreased automatic adjustments & reactions
Difficulty with WS and decreased fluididty & mvmt
Hemi Arm: Pain
Pain in sh. sublexations, gravity & poor handeling
Sh, Pain types
Joint paint (where is pain coming from, what type aka harp dull)
Muscle pain (affected by speed of stretch)
Altered sensativity pain (tholamic pain) = distroffla program aka load and carry
Sh. hand syndrome ( CRPD) = increase pt mvmt
Brachial plexus traction (poor handeling)
Rotator cuff tear (ask about sh. hx) related to quick mvmts = use moist heat
Adhesive Changes = decreased AROM
Sh. Pain Eval
Hx.
Age
Transfer Techniques (avoid using sh to mobilize)
Activity level (HEP for stretching & ROM)
Available mvmt
Eval of Pain : nature, location, elements that intensify & deminish pain.
Sh Pain Eval : Visual Inspection
Posture Atrophy Early signs of RSD GH malaignmant neglect tone PROM Motor Performance
Sh. Pain Tx
Normalize tone Proper handling ROM Functional use Sh. support address primary pain causes
Sh. Subluxation
Partial Dislocation or separation of joint surfaces
Causes of Subluxations
Abnormal scapular position
Loss of bilateral muscle firing
Weakness
Spasticity
Inferior Subluxation
Scapula = Downward rotation
Humerus = IR, ADD, below inferior lip
Elbow = ext, pronation
(pic of guy in W/C)
Inferior Subluxation TX ideas
ROM & Scap mobility
Rotate Scap upward & hold hand placement to maintain correct alignment
Trays/Armrest (pt must be able to unstrap it)
Support affected Sh. so gravity does not continue to over stretch muscles
Use estim tapping