CV TEST ONE: HYPERTENSION Flashcards

1
Q

Hydrochlorothiazide

A

Diuretic HTN Med, potassium wasting (no potassium) potassium rich diet suggested

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2
Q

Peripheral resistance

A

Arteries’ resistance to blood flow, arteries constrict=increased resistance , arteries dilate= decreased resistance

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3
Q

Modifiable/non modifiable HTN factors

A

•Modifiable—diet, lifestyle, exercise
•non modifiable—race, gender, heredity, age, pre existing conditions

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4
Q

BP categories

A

*normal–less than 120/80 mmHg
*elevated–120-129/<80 mmHg
*stage 1–130-139/80-89 mmHg, poss. Med. intervention, lifestyle change
*stage 2–BP < 140/< or = 90 mmHg, meds (ANTIHYPERTENSIVES) lifestyle change
*urgent–180/120 or <, immediate reduction of BO to prevent organ damage, PT resting while 911 contacted

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5
Q

HTN diagnosis includes:

A

Confirmed >2 occasions of hypertensive measurements, risk factors, presence/absence of symptoms, history of heart/kidney disease, current meds used

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6
Q

HTN S/S

A

Headache, bloody nose, anxiety, SOB

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7
Q

Cardiac catheterization

A

•Studies heart’s anatomy/physiology, assesses pressure in coronary arteries/chambers/great vessels, assesses C.O./O2 saturation
•dye injected into catheter>inserted into femoral vein

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8
Q

Pre & post cardiac catheter

A

•pre– consent, assess for iodine/dye allergies, NPO/conscious sedation
•post–monitor for bleeding, pressure device at site, bedrest, no flexion of extremity for hrs, V.S. Taken, peripheral pulses taken

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9
Q

PTs radial pulse is different than their apical. What should nurse proceed to do and why

A

Report pulse deficit to doctor, could indicate arrhythmia/cardiac dysfunction

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10
Q

Trans esophageal Echocardiogram (TEE Test)

A

•Clearer picture, transducer probe in esophagus (doesn’t cross lung/rib tissue), sedation/local anesthetic in oropharynx, NPO 6 hrs pre-test
•complications—esophageal perforation, throat bleeding, hypoxia, dysthymias

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11
Q

Pre/Post TEE

A

•Pre TEE—assess allergy hx/sedation reactions/local & general anesthesia
•post TEE–asses VS, monitor bleeding, MONITOR GAG REFLEX!, keep suction & resuscitation equipment available

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12
Q

Pericardial rub

A

Heard in inflammatory processes (pericarditis), sounds like rubbing tissue papers, can be heard closely if PT sitting up & leaning forward

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13
Q

S3 & S4 sounds

A

•S3—ventricular gallop or LUBB DUBB DUBB
•S4— atrial gallop or LUBB LUBB DUBB

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14
Q

3 membranes of heart

A

•fibrous pericardium– outermost layer, forms sac around heart
•parietal pericardium–middle layer
•visceral pericardium aka epicardium– innermost layer

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15
Q

Where does blood start at in the heart ?

A

Superior and inferior vena cava

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16
Q

If a PT is struggling financially to pay for care costs they should be redirected to:

A

The financial dept within the clinical setting

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17
Q

The left ventricle pumps __x times more force because _____

A

5, accommodate systemic circulation

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18
Q

Cardiac pathway

A

AV node>SA node>Bundle of His>purkinje

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19
Q

SA node

A

Pacemaker of the heart, where cardiac impulse is first carried out, heartbeat initiation, located in R.A.’s wall, gives normal rhythm

20
Q

Cardiac cycle

A

One heartbeat, 0.8 seconds

21
Q

During cardiac cycle, both atrias and ventricles contract and relax, known as

A

•systole–contraction(emptying occurs)
•diastole–relaxation phase

22
Q

Cardiac output

A

•amount of blood ejected from left ventricle in a minute
•calculation –stoke volume X heart rate

23
Q

Too much or too little potassium can cause

A

Dysrhythmia

24
Q

ANP (Atrial natriuetic) is secreted in response to

A

Vessels stretching from increased volume

25
Q

Uretic

A

Sodium and water loss, BP increased

26
Q

If there’s a different reading on the PTs arms go with

A

The higher reading for ongoing use

27
Q

Complications of HTN

A

CAD, atherosclerosis, M.I., H.F., stroke, kidney damage, eye damage, L.V. hypertrophy

28
Q

Meds to lower Bp (diuretics)

A

•K-sparing: Spironolactone (Aldactone)
•k-wasting: thiazide (hydrochlorothiazide) (chlorothiazide)
•Loop: furosemide (lasix), torsemide (demadex)
•oldest most studied antiHTN med
•PTs should swing legs on bed’s edge for some minute before standing to avoid syncope

29
Q

most common heart disease manifestation

A

chest pain (angina)

30
Q

Dyspnea may be a sign of

A

LV failure or transient CHF

31
Q

Heart sounds produced by closing valves start at

A

the aortic

32
Q

murmur

A

turbulent blood flow through heart/vessels, prolonged sound>narrowed valve closing

33
Q

cardiac enzyme elevated values

A

CPK and troponin

34
Q

C.O., blood viscosity, blood volume, PVR

A

factors determining BP

35
Q

What is HTN? what could it cause?

A

Increase in blood pressure against vessel walls…

coronary heart disease, CHF, stroke, M.I., eye damage, LV hypertrophy

36
Q

can a clot be caused by HTN

A

Yes due to blood being forced thru narrow vessels

37
Q

Increased C.O., PVR, blood viscosity, blood volume, hormone imbalances, kidney dysfunction

A

pathophysiologic changes of HTN

38
Q

Lifestyle mods. should be done ______ drug therapy unless PT is at high risk

A

before….

39
Q

PTs at risk for urgent HTN

A

untreated HTN, non compliant with med, using nitroprusside as its a vasodilation

40
Q

beta 1

A

beta 1 antagonist: iontropic, chronotropic effects
beta 1 blocker: decreased contractility, HR, decreased C.O.

41
Q

BETA 2

A

agonist: bronchodilation
blocker: bronchoconstriction

42
Q

alpha 1

A

agonist: vasoconstriction
blocker: vasodilation

43
Q

secreted by adrenal medulla, sympathomimetic response

A

epinephrine

44
Q

produced by adrenal cortex, regulates Na & K levels

A

aldosterone

45
Q

ANP

A

secreted by atria, increases Na excretion

46
Q
A