CRANIAL NERVE FUNCTION TESTS Flashcards

1
Q

WHAT IS ANOSMIA

A

loss of smell due to a neurological lesion

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2
Q

testing function of olfactory nerve

A

eyes closed, the subject is asked to sniff substances through each nostril in turn and name the odours - coffee , vanilla

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3
Q

optic and oculomotor nerve tests

A

test for visual acuity , check visual fields and inspect optic discs , inspect size and shape of pupil and compare , test pupillary reactions to light

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4
Q

occulomotor trochlear and abducens

A

subject sitting upright , head held by examiner , subject asked to follow examiners finger .
movement of eyes along horizontal plane - > medial and lateral rectus muscles

movement of eyes along vertical planes - looking outwards - superior and inf rectus muscles

movement of eyes along vertical planes - looking inwards - superior and inferior oblique muscles

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5
Q

during exam of occulomotor trochlear and abducens note what

A

Note for any strabismus (squint), ptosis, nystagmus, deviation of the
eye and ask the patient if they have diplopia (double vision )

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6
Q

testing function of CN V

A

cutaneous sensation can be tested in appropriate areas of the face.
jaw muscles can be tested by asking subject to clench teeth and palpating the contracting masseter and temporalis muscles .
jaw jerk reflexes .
pterygoids -> if subject opens their mouth against resistance and there is unilateral weakness , jaw will deviate to one side

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7
Q

test functions of CN V and VI

A

wisp of cotton wool is applied to cornea , results in blinking of both eyes
sensory limb is opthalmic division . motor response is a contraction of orbicularis oculi , recieves its motor innervation form VII

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8
Q

testing function of vestibulocochlear - hearing

A

ƒ Each ear is tested separately
ƒ Vibrating tuning fork is held close to the external auditory
meatus and its base is then pressed gently against the mastoid
bone Æ subject is asked which of these two stimuli is loudest
ƒ Fork is left applied to the mastoid bone until the vibrations are
no longer heard and the fork is the held against the ear Æ
subject is asked if note can still be heard
ƒ For a normal subject, air conduction > bone conduction
ƒ If this is not the case may be due to infection of middle ear
ƒ Weber’s test Æ tuning fork is placed in centre of forehead and
subject is asked which side is louder
ƒ Both equally (normal), left (right sensironeural impairment) and
right (left sensironeural impairment)

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9
Q

vestibulo-occuloreflexes

A

ƒ Reflex movements of the eyes consequent upon rotation of a
subject in a rotating chair
ƒ Horizontal nystagmus (rotate as per normal) -> eyes move
horizontally
ƒ Vertical nystagmus (head bend and tilt to one side) -> eyes
move vertically
ƒ Rotational nystagmus (head bent) -> eyes swing upwards from
left to right
ƒ Ask subject to observe the pictures on the slowly rotating drum
and observe eye movements -> optokinetic nystagmus
(normally observed by looking at objects moving across field of
vision)

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10
Q

testing glossopharyngeal and vagus nerves

A

tests are not very impressive esp in normal subject

- subject has no huskiness , dysphonia, dysphagia and palate moves symmetrically when the subject says AHHHHH

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11
Q

accessory nerve function test

A

subject should be able to lift shoulders against resistance , strenght of trapezius muscle

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12
Q

hypoglossal function test

A

when subject is asked to protrude tongue and it will deviate to weaker side

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13
Q

what compartment is the most easily displaced in the brain when there is raised ICP

A

the CSF
Can be dis-placed from the ventricles and cerebral subarachnoid space into the spinal subarachnoid space.
it can also undergo increased absorption or decreased production

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14
Q

what is the Monro-kellie hypothesis

A

Normally, a reciprocal relationship exist among the three intracranial
volumes such that the ICP is maintained within normal limits.
Because these volumes are practically incompressible, a change in one
component must be balanced by an almost equal and opposite effect
in one or both of the remaining components

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15
Q

clinical effects of raised ICP

A

headache -> worse in the morning and when stooping or bending
vomiting -> acute rise in ICP
papilloedema -> swelling of the optic disc and retina resulting in disc haemorrhages

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16
Q

what is brain herniation

A

consequence of increased ICP
brain is protected by the skull and two supporting septa (falx cerebri and tentorium cerebelli)
brain herniation represents a displacement of brain tissue under the falx cerbri or through tentorial notch or incisuria of the tentorium cerebelli
area of high pressure ,moving contents to low pressure

17
Q

brain herniation is divided into two categories

A

supratentorial

infratentorial

18
Q

causes of hypoxic brain damage

A

extracranial blood loss -> common in association with severe head injury

uncontrolled seizures -> further source of secondary insults following head injury due to increased metabolic rate associated with head seizures and the respiratory impairment that comes with them

19
Q

acute management of head injury

A
ƒ Airways and cervical spine control
ƒ Breathing and ventilation
ƒ Circulation and haemorrhage control
ƒ Disability and neurological status
ƒ Exposure and environment
20
Q

neural plasticity

A

recovery of tissue which had been partially but not irreversibly damaged by the injury and adaptation of uninjured tissue to undertake some of the functions previously subserved by tissue which has been irreparably damage

21
Q

Sequelae

A

pathological condition resulting from a prior disease, injury or
trauma

22
Q

sequelae of head injury

A
incomplete recovery -> cognitive impairment , hemiparesis 
post traumatic epilepsy 
post traumatic syndrome
chronic subdural haemorrhage 
hydrocephalus
23
Q

what is post traumatic syndrome

A

complaints of headache , dizziness, malaise
litigation
depression

24
Q

what is punch drunk syndrome

A

o Consists of cognitive impairment, extrapyramidal and pyramidal signs
o Seen typically in professional boxers

25
Q

Which nerve provides somatosensory innervation to the nasal cavity ?

A

Trigeminal - ophthalmic and maxillary divisions

26
Q

Which intrinsic eye muscles are innervated by the oculomotor nerve

A

Sphincter papillae - pupil constriction

Ciliary muscle - accommodation

27
Q

Where do cranial nerves III, IV and VI enter the orbit?

A

All enter the orbit through the superior orbital fissure

28
Q

Which cranial nerve is involved in the corneal reflex

A

Trigeminal - sensory afferent ,ophthalmic division

Facial nerve efferent - orbicularis oculi

29
Q

Which gland is innervated by the glossopharyngeal nerve

A

Parotid gland via the lesser petrosal nerve

30
Q

Which cranial nerves are involved in the gag reflex

A

Afferent glossopharyngeal nerve

Efferent vagus nerve

31
Q

Deviation of the uvula indicates a weakness of what CN ?

A

Glossopharyngeal and vagus

32
Q

What is shingles

A

Reactivation of the chicken pox virus - varicella - zoster virus - which remains dormant in nerve cell ganglia

33
Q

What is Ramsay hunt syndrome?

A

A particular form of shingles that forms in the geniculate ganglion

34
Q

What is a common cause of jugular foramen compression

A

Paraganglioma

35
Q

How can CSF be identified

A

CSF should have glucose in it , but nasal secretions do not; this is neither sensitive nor specific - beta 2 transferrin level only found in CSF
HALO SIGN on tissue of blood stained fluid

36
Q

What is nystagmus

A

Abnormal eye movements

37
Q

What can lead to oculomotor nerve palsy ?

A

Aneurysms of the posterior communicating artery causing a subarachnoid haemorrhage -> presents with severe head ache, dilated pupil, ptosis