Child Internalising Flashcards

1
Q

Point prevalence of Child Anxiety?

A

2.5 - 5% meet criteria at any given time

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2
Q

How many meet criteria for more than one Anxiety Disorder?

A

40 - 60%

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3
Q

What is homotypic continuity?

A

the development or continuation of symptoms and disorders that are similar to the type shown earlier in life. E.g: anxious children

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4
Q

what is heterotypic continuity

A

the development or continuation of symptoms and disorders that are different to the type shown earlier in life

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5
Q

What are individual environmental factors in twin studies

A

factors that make twins different from each other

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6
Q

what are shared environmental factors in twin studies

A

factors commonly experienced by both twins e.g. school, parents, food

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7
Q

what is the common temperament that predicts childhood anxiety

A

shyness, inhibition and withdrawal

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8
Q

whats the issue with using inhibited temperament as a risk factor for developing anxiety?

A

inhibition and anxiety are highly overlapped measures of the same underlying construct so of course they are closely related. We also don’t know fully what causes temperament. Do the same things cause inhibition and anxiety

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9
Q

how do we make a distinction between anxiety and just inhibition?

A

By looking at impact on their life - inhibition has less of an impact

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10
Q

what is overprotective parenting

A

a pattern of parent-child interaction characterized by parental anticipation of potential threat leading to restriction of child engagement with situations or behaviours.

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11
Q

what is the limitation of looking at parenting as a cause of anxiety etc?

A

We don’t know the direction of effects

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12
Q

dependent life events?

A

major environmental experiences in life that, by their nature may be a result of the behaviour of the child

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13
Q

what are processing biases

A

a variety of cognitive methods of dealing with information from the environment in such a way that the methods tend toward a particular meaning.

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14
Q

whats the key ability anxious children don’t develop?

A

the ability to inhibit attentional focus toward threat

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15
Q

What is bibliotherapy?

A

treatment components are presented via written or computerized media with little or no therapist contact

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16
Q

What are selective interventions?

A

providing intervention to individuals who score high on one or more risk factors for a disorder, regardless of whether the individuals actually have the disorder

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17
Q

What is the increase in risk for depression if a child already has anxiety

A

8-29%

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18
Q

What are vulnerability stress theories for child depression?

A

psychological theories that explain the causation of depression in terms of stress and negative life events and how we react to them.

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19
Q

An example of a Vulnerability Stress Theory?

A

Beck’s Schema Theory 1976

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20
Q

What is Beck’s 1976 Schema Theory

A

We develop schemas in childhood - long term knowledge structures in memory - which activate during stressful life events. Evidence that confirms the Schema is collected and stored.

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21
Q

what is a ruminative response style

A

overthinking an event in a way that is not leading to problem solving - increases risk of depression

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22
Q

How can a depressed child’s behaviour lead to a Negative Response style?

A

People avoid the child because they tend to have poor eye contact, disclose negative info about themselves, seek excessive reassurance. This leads to rejection from others.

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23
Q

What is the stress-generation hypothesis?

A

Depressed children attract more negative life events that are dependant on how they behave e.g. losing your job.

24
Q

what is the insiguous effect of depression?

A

it maintains and exacerbates itself.

25
Q

When was ECT used as the prominent treatment for MDD

A

1938 - 1950s. Used as a last resort today.

26
Q

what are the 3 waves of drug treatments for depression after 1950s?

A

MAOIs, Tricyclic medications, SSRIs

27
Q

How do MAOIs work?

A

increase the availability of serotonin and norepinephrine.

28
Q

How do tricyclic medications work?

A

block the presynaptic reuptake of serotonin and noradrenaline.

29
Q

How do tricyclic medications increase suicide risk?

A

Because they lift people out of vegetative symptoms - they have more energy

30
Q

How do SSRIs work?

A

Block the re-uptake of Serotonin

31
Q

What are the 4 main psychological treatments for depression?

A

Brief psychodynamic therapy, CBT, Interpersonal Psychotherapy and mindfulness based cognitive therapy

32
Q

Which psychological treatment for depression specifically targets relapse?

A

Mindfulness based cognitive therapy

33
Q

What is the point prevalence of MDD

A

25% at age 21

34
Q

Why might there be higher prevalence in females for depression?

A

More socially acceptable to be vulnerable, and males might self-medicate and go into the stats for substance abuse instead. OR the kinds of stressors females experience as adolescents are less controllable (Learned helplessness theory)

35
Q

How does having a depressed parent increase a child’s risk?

A

4-5 times more likely

36
Q

Where would universal prevention for depression take place?

A

In a school environment

37
Q

Who is indicative prevention for

A

children who are going towards a diagnosis

38
Q

Who is selective prevention for?

A

people who are at a high risk e.g. children of depressed parents

39
Q

What is the depressive cognitive triad

A

negative thoughts about the self, the world, the future become dominant in consciousness

40
Q

What does MAOI stand for

A

Monoamine Oxidase Inhibitor

41
Q

at what part in the course of depression should IPT be done?

A

beginning/ exacerbation

42
Q

the 3 cognitive diathesis-stress models

A
  1. Beck - Negative Schema
  2. Seligman - helpessness
  3. cognitive style + negative events => depression
43
Q

Can depressed children be medicated

A

no not approved in Aus. Fluoxetine only in severe MDD

44
Q

how many children actually receive proper services?

A

less than 10%

45
Q

Why do we need to look at what is normative in children?

A

So we can see what behaviours are maladaptive

46
Q

why are internalising disorders less diagnosed in children

A

less disruptive, not as easily observable etc

47
Q

what is Separation Anxiety Disorder

A

Developmentally inappropriate and excessive fear or anxiety concerning separation from individual to whom they are attached

48
Q

what are the duration criteria for separation anxiety in children and adults?

A
children = 4 weeks
adults = 6 months
49
Q

What is the change to separation anxiety disorder in the DSM-5

A

you can diagnose adults (no childhood onset needed)

50
Q

What is GAD

A

Excessive anxiety and worry occurring more days than not for at least 6 months about a number of events or activities

51
Q

According to Szabo what differs in what children and adolescents worry about?

A

children worry more about physical outcomes

adolescents worry more about social outcomes

52
Q

What changed in the Anxiety Disorders from DSM-IV to DSM-5?

A

OCD and PTSD taken out

Selective Mutism, SAD and Agoraphobia added.

53
Q

How many GAD symptoms are required to diagnose a child?

A

1

54
Q

What was Vasey’s developmental analysis for GAD in kids?

A

Children need to be able to imagine chains of catastrophic outcomes. Must switch to verbal thinking from imagery. (age 7/8)

55
Q

how do younger and older children differ in terms of worry related to likelihood and cost

A

older children - likelihood and cost explain worry equally

younger children - cost has unique association with worry