Chapters 35, 36 Flashcards

1
Q

What are two classes of antihistamines?

A

H1 and H2

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2
Q

What are the main differenes amongst local and systemic nasal congestants?

A

systemic decongestants can elicit an effect in every body system. whereas local will not

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3
Q

Who should not receive dextromethorphan

A

patients who have vey thick secretions with a weak cough - this can lead the patient to developing pneumonia from retained secretions. Additionally, think of patients where coughing can have negative outcomes like recent surgery

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4
Q

Is chronic bronchitis reversible

A

typically not

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5
Q

Which med works by inhibiting the response of M-3 receptors?

A

tiotroprium

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6
Q

Which dietary instructions will you provide to your patient taking aminophylline-theophylline

A

there is an interaction between xanthines, so you need to avoid xanthine products in addition to coffee, tea, and soda

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7
Q

What occurs when H1 receptors are stimulated?

A

constriction of extravascular smooth muscle and lining of nasal cavity

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8
Q

How do nasal decongestants work?

A

they stimulate the alpha-adrenergic receptors to cause vascular constriction of capillaries, and a shrinkage in mucosa

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9
Q

what does the term expectorate mean?

A

to cough

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10
Q

What makes restrictive lung disease different

A

decreased total lung capacity due to los of elasticity

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11
Q

What class of drug is tiotropoium

A

anticholinergic

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12
Q

How do leukotriene receptor antagonists work?

A

binds with leukotriene receptors to inhibit smooth muscle contraction and bronchoconstriction

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13
Q

What occurs when H2 receptors are stimulated?

A

increase in gastric secretions

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14
Q

What makes intranasal glucocorticoids different from nasal decongestants?

A

they also have anti-inflammatory properties

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15
Q

what purpose to expectorants serve?

A

loosen bronchial secretions which are eliminated by coughing

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16
Q

how do sympathomimetics work for patients with bronchoconstriction

A

increased production of cAMP, which causes bronchial dilation

17
Q

for a ptaient taking tiotropium, what effects do you need to educate them to anticipate?

A

your anticholinergic effects of: can’t see, can’t pee, can’t spit, can’t sh…defecate.

18
Q

what is the serum range for theophylline

A

5-15 mcg/ml

19
Q

what is the main difference between first and second generation antihistamines?

A

first generation can cause drowsiness, dry mouth, and anticholinergic symptoms

20
Q

pulling from the knowledge of edocrine, what can intranasal glucocorticoids also do?

A

increase your blood sugar and immunosupression

21
Q

what is the best natural expectorant?

A

fluids! recommend the patient to increase their water intake if not contraindicated

22
Q

What is different about metaproterenol than albuterol

A

metaproterenol is used for long term asthma treatment

23
Q

what is the main difference in tiotropoim than additional agents

A

this is for maintenance, and not acute attacks. imperative to educate the patietn in the difference so they don’t use the wrong drug during an acute attack

24
Q

which meds will you tell your patient to avoid while taking montelukast?

A

aspirin and NSAIDs, as they can block the action of montelukast

25
what are important assessments to make for a patient receiving diphenhydramine
1. neuro: making sure they aren't too lethartic 2: respiratory: make sure patinet is not in respiratory distress 3. genitourinary: urinary retention
26
whta is the goal of dextromethorphan?
reduce viscosity of tenacious secretions nad provides an effecting but infrequent cough
27
what diseases comprise of COPD
asthma, bronchitis, emphysema, bronchiectasis
28
if oyu need to adminster a beta agonist and an additional agent, which should be administered first
beta agonist always first to dilate and open the airway to improve dlivery of 2nd med
29
how to methylxanthine derivatives work?
stimulate the CNS, resiratoins, dilate coronary and pulmonary vessels and promote diuresis
30
for a patient having an acute attack, what type of medication would you want to administer first?
IV steroids preferred over beta-agonists as they redue bronchial hyper-responsiveness