Chapter 42 Worksheet Flashcards

1
Q

what is acute hypoxemia?

A

hypoxemia is a decreased level of oxygen in the blood. “acute” means sudden

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2
Q

What are the threshold criteria for defining hypoxemia in adults according to the AARC clinical?

A

PaO2 < 60 mm Hg

SaO2 <90%

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3
Q

Describe the two compensatory mechanism for the cardiopulmonary system when faced with hypoxemia

A

Lungs: increased ventilation: faster rate and increased depth
Heart: Increased cardiac output: faster heart rate, increased stroke volume equals increased CO2

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4
Q

in what acute cardiac condition is oxygen therapy especially important?

A

Myocardial infarction, because when the heart is stressed or damaged, it is especially important to reduce workload

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5
Q

What effects does hypoxemia have on the pulmonary blood vessels? What are long term consequences of this effect?

A

alveolar hypoxemia causes vasoconstriction of pulmonary vessels. if prolonged, this results in pulmonary hypertension, increased work of the right heart, and eventually cor pulmonale or right heart failure

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6
Q

List 3 basic ways to determine if a patient needs O2?

A
  1. laboratory evidence
  2. patient’s specific condition or problem
  3. bedside assessment
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7
Q

List 6 common acute clinical situations where hypoxemia is so common that oxygen therapy is usually provided

A
  1. carbon monoxide poisoning
  2. cyanide poisoning
  3. shock
  4. trauma
  5. acute myocardial infarction
  6. post-op patients
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8
Q

oxygen toxicity can affect what two organ systems?

A
  1. lungs

2. central nervous system

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9
Q

the harm caused by oxygen is influenced by what two factors?

A
  1. PO2

2. exposure time

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10
Q

describe the effect on lung tissue by breathing excessive O2

A

tracheobronchitis and substernal chest pain develop during the first 12 hours. Next, vital capacity and lung compliance being to decrease (12 to 30 hours) A state similar to bronchopneumonia develop. The alveoli and capillaries are damaged, resulting in interstitial edema. Type 1 cells are destroyed. In the end stages, hyaline membranes form, the pulmonary fibrosis and hypertension develop

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11
Q

what is meant by “viscous circle” in reference to oxygen toxicity?

A

high FiO2 caused O2 toxicity, which causes shunting, which results in lower PaO2 levels, which require high FiO2 levels for treatment

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12
Q

although every patient is unique, what general rule of thumb can be applied to prevent oxygen toxicity

A

limit exposure to 100% oxygen to less than 24 hours whenever possible. High FiO2 levels are acceptable if the concentration can be lowered to 70% within 2 days, and 50% within 5 days

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13
Q

what specific type of COPD patient is likely to experience depression of ventilatory drive while breathing oxygen

A

a small percentage of severe COPD patients with carbon dioxide retention tend to ventilate less when exposed to moderate to high levels of oxygen

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14
Q

when should oxygen be withheld from a hypoxic COPD patient to avoid depressing ventilation

A

never

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15
Q

during what time period after birth is a preemie likely to develop ROP

A

premature infants and neonates up to 1 month

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16
Q

how can we reduce the risk of ROP

A

maintain PO2 levels less than 80 mm Hg. Minimize other factors such as acidosis

17
Q

how can O2 cause atelectasis

A

FiO2 levels above 0.50 cause atelectasis by washing nitrogen out of poorly ventilated units. The oxygen diffuses into the blood, and the alveoli collapse

18
Q

how can we reduce the risk of absorption atelectasis

A

use the lowest FiO2 levels. Identify at risk patients. Use hyperinflation therapies to avoid or reverse the atelectasis

19
Q

what is the fire triangle

A

oxygen, heat, fuel

20
Q

what is the biggest hazard in the home setting

A

smoking

21
Q

when should we attach a cannula to a bubble humidifier

A

when the flows are greater than 4 L/min (also infants and children)

22
Q

what maximum flow does the text suggest for newborns

A

2 L/min

23
Q

how does the patient breathing pattern affect the FiO2 delivered to the lungs when using a low flow device such as a cannula

A

a patient has a saturation of 94% on 2 L by cannula. he exercise and increases rate and depth, drawing in more room air while the O2 flows constantly at 2L. The additional airs drops the FiO2 in the trachea and the saturation

24
Q

what is the primary disadvantage of the transtracheal catheter

A

requires surgical procedure; maintenance

25
Q

what FiO2 range is usually delivered by low-flow devices

A

low 24% to 45%

26
Q

because you cant tell exactly how much oxygen a patient is receiving at any given moment from a cannula or any low-flow device, how can you assess the effects of administering the drug

A

assess the patient response (clinical, SpO2)

27
Q

in what setting are reservoir cannulas usually used

A

the home or ambulatory patients who need increased mobility

28
Q

what is the primary difference between the partials rebreathing and non-rebreathing masks

A

valves; particularly between the bag and the mask

29
Q

how can you tell if a non-breathing mask has an adequate flow rate

A

the bag does not fully deflate on inspiration

30
Q

why does the AEM have larger opening on the side of the mask than the simple oxygen mask

A

allow excess flow and patient exhalation to escape

31
Q

what effect does raising the delivered flow from the flowmeter have on the FiO2 delivered by an AEM?

A

little or no effect

32
Q

how do you boost the total flow when using an AEM

A

increase the input flow

33
Q

describe an easy way to tell if an air entrainment nebulizer is providing sufficient gas to meet the patients needs

A

observe the mist on the expiratory side when the patient inhales. calculate the flow delivered by the device. measure the patient minute ventilation, and multiply by 3. compare the two values

34
Q

give on example of a specialized flow generator that produces an aerosol

A

a gas injection nebulizer

35
Q

what effect does downstream resistance to flow have on FiO2 and total flow delivered by a typical AEM or nebulizer entrainment system

A

downstream resistance increases the FiO2 and decreased total flow delivered