Chapter 4 Flashcards

1
Q

humoral immunity

A
  • production of antibodies

- main defense against bacteria and bacterial toxins

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2
Q

cell-mediated immunity

A
  • formation of a population of lymphocytes that attack and destroy foreign material
  • main defense again viruses, fungi, parasites, and some bacteria
  • mechanism by which body rejects transplants
  • eliminates abnormal cell division
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3
Q

t lymphocyte

A
  • thymus-dependent
  • precursor cells that migrated from the marrow to the thymus
  • proliferate to form a diverse population of cells that regulate the immune response and generate a cell-mediated immune reaction to eliminate antigen
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4
Q

b lymphocyte

A
  • bone marrow
  • precursor cells that remained within the bone marrow
  • proliferate and mature into antibody-forming plasma cells
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5
Q

cause of events triggered by foreign antigen

A
  • recognition of foreign antigen
  • proliferation of lymphocytes that are programmed to respond to the antigen form a large group of cells
  • destruction of foreign antigen by the responding lymphocytes
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6
Q

regulator t cells

A
  • helper t cells that regulate immune system by establishing a balance between promoting and inhibiting the immune response
  • in AIDS the virus attacks and destroys helper t lymphocytes
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7
Q

effector t cells

A

-involved in delayed hypersensitivity reactions

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8
Q

Ig G

A
  • smaller antibody
  • principle antibody molecule in response to majority of infectious agents
  • produced after Ig M
  • if increased in blood test, means infection is reoccurring
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9
Q

Ig A

A
  • GI tract
  • produced by antibody-forming cells located in the respiratory and gastrointestinal mucosa
  • combines with harmful ingested or inhaled antigens, forming antigen-antibody complexes that cannot be absorbed, preventing antigens from inducing sensitization
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10
Q

Ig M

A
  • large antibody

- very efficient combining with fungi

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11
Q

Ig E

A
  • allergic reactions

- found in minutes quantities in blood

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12
Q

anaphylactic

A
  • sensitizing antigen circulates throughout the body, triggers widespread mediator release from Ig-coated mast cells and basophils
  • may lead to anaphylaxis
  • prompt treatment required with epinephrine
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