Chapter 18 Flashcards

1
Q

Osteoarthritis is:

A

the progressive deterioration and loss of cartilage and bone in one or more joints

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2
Q

Crepitus:

A

occurs when the cartilage disintegrates and pieces of bone and cartilage “float” in the diseased joint causing crepitus, a grating sound caused by loosened bone and cartilage

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3
Q

Cause of PRIMARY OA:

A

aging

genetic factors

Hips, knees & hands most commonly affected

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4
Q

Cause of SECONDARY OA:

A

Joint injury (excessive use, trauma or RA)

obesity

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5
Q

Typical onset of RA:

A

35-45 YR

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6
Q

Typical onset of OA:

A

Older than 60 yrs

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7
Q

Risk factors of RA

A

Autoimmune (genetic basis)

Emotional stress (triggers exacerbation)

Environmental factors

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8
Q

Risk factors of OA:

A

Aging

genetic factor (possible)

Obesity

Trauma

Occupation

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9
Q

Disease process for RA & OA

A

RA: Inflammatory

OA: Degenerative

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10
Q

Disease pattern for RA:

A

Bilateral, symmetric, multiple joints

Usually affects upper extremities first

Systemic

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11
Q

Disease pattern for OA:

A

May be unilateral, single joint

Affects weight bearing joints, hands, spine

Nonsystemic

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12
Q

Common drug therapy for RA:

A

NSAIDS (Short term use)

Methotrexate

Leflunomide (Arava)

Corticosteroids

Immunosuppresive agents

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13
Q

Common drug therapy for OA:

A

NSAIDS (Short term use)

Acetaminophen

Analgesics

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14
Q

clinical manifestations of OA

A

Joint pain and stiffness

Crepitus

Herberden’s nodes

Bouchard’s nodes

joint effusions

Atrophy of skeletal muscle

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15
Q

Early: clinical manifestations of RA (joint/systemic)

A

Joint stiffness/inflammation

Low grade fever

weakness

fatigue

anorexia

paresthesias

swelling

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16
Q

Late clinical manifestations of RA (JOINT/SYSTEMIC)

A

Joints become progressively inflamed and quite painful

deformities (swan neck or ulnar deviation)

moderate to severe pain and morning stiffness

osteoporosis

severe fatigue

anemia

weight loss

17
Q

Lab tests for OA (2)

A

ESR

HsCRP

18
Q

Lab tests for RA/CTD

A

Rheumatoid factor

ANA

ESR

Serum complement (C3 &C4)

SPEP

Serum immunoglobulins

19
Q

RA Nonpharmacologic interventions:

A

adequate rest

proper positioning

ice and heat application

plasmapheresis (not common)

complementary and alternative therapies

promotion of self-management

management of fatigue

enhance body image

20
Q

OA Chronic Pain: Nonsurgical management

A

Drug therapy (Tylenol) drug of choice

Rest

Immobilization

Positioning

Thermal modalities

Weight control

Integrative therapies (glucosamine, chondroitin)

21
Q

Inflammatory rheumatology

A

RA

Systemic lupus erythematosus (SLE)

Autoimmune disease

22
Q

OA is

A

most common arthritis type

progressive loss of cartilage

joint pain
- loss of function characterized by progressive deterioration

Cartilage disintegrates; bone and cartilage “float” into joint, causing crepitus

23
Q

Nursing interventions to prevent dislocation:

A

position correctly

for hip: keep leg slightly abducted

for hip, prevent hip flexion beyond 90 degrees

assess for acute pain, rotation, and extremity shortening

24
Q

Nursing interventions to prevent infection:

A

aseptic techniques for wound care and emptying of drains

hand hygiene

culture drainage fluid, if change

monitor temperature

25
Q

Nursing interventions to prevent venous thromboembolism

A

have patient wear elastic stockings or SCDs

teach leg exercises

encourage fluid intake

observe for signs of thrombosis (redness, swelling or pain)

observe patient for changes in mental status

administer anticoagulant as prescribed

do not massage legs

do not flex knees for a prolonged period of time

26
Q

Nursing interventions to prevent hypotension, bleeding, infection

A

take vital signs q4hr

observe patient for bleeding

report excessively low bp or bleeding