Chapter 12 - Disease Flashcards

1
Q

Bacteria diseases

A

TB
Bacterial meningitis
Ring rot

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2
Q

Virus diseases

A

HIV/AIDS
Tobacco virus
Influenza

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3
Q

Protist diseases

A

Malaria
Potato blight

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4
Q

Fungi diseases

A

Black Sigatoka
Athletes foot/ringworm

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5
Q

TB

A

Damages and destroys lung tissue
Suppresses immune system
Body less able to fight other diseases

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6
Q

Ring rot

A

Damages leaves tubers and fruit
No cure

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7
Q

TMV

A

Damages leaves, flowers and fruit
Stunts Growth

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8
Q

Malaria

A

Caused by plasmodium
Female anopheles mosquitoes are the vector
Invade red blood cells

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9
Q

Potato blight

A

Destroy leaves, tuber and fruit

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10
Q

Black Sigatoka

A

In bananas, turn leaves black

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11
Q

Athletes foot/ringworm

A

There are different types of ringworm, different fungi
Athletes foot is a type of ringworm
Digest skin between toes

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12
Q

Animal direct disease transmission

A
  • contact
  • Inoculation
  • ingestion
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13
Q

Animal indirect disease transmission

A
  • formites
  • Droplet
  • vectors
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14
Q

What factors affect transmission of diseases in animals?

A

Overcrowding poor nutrition climate change

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15
Q

Plants direct disease transmission

A
  • Healthy plant in contact with unhealthy plant
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16
Q

Plant indirect disease transmission

A
  • soil contamination
  • Vectors: wind (spores) water, animals, humans
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17
Q

What factors affect transmission of diseases in plants?

A

Crowding poor mineral nutrients, warm damp conditions, climate change

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18
Q

Callose deposition

A
  • plant attacked by pathogen
  • Plant synthesises lots of the polysaccharide callose
  • callose deposited into cell walls and cell surface membranes of neighbouring cell strengthened with lignin
  • Forms a barrier against pathogens
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19
Q

Plant chemical defences

A
  • insect repellent and insecticides
  • Anti- bacterial compound
  • Antifungal compounds
  • Toxins eg. Form Cyanide compounds.
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20
Q

Non-specific defences

A

Skin
Mucous membrane
Hydrochloric acid and stomach
Expulsive reflexes
Inflammatory response
Wound repair
Blood clotting

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21
Q

Blood clotting

A

Platelets come in contact with collagen and adhere and secrete
Thromboplastin - triggers formation of blood clot
Serotonin - smooth muscle contraction to narrow blood vessels and limit blood supply

22
Q

Wound repair

A
  • epidermal cells under the scab grow to seal wound
  • damage blood vessels regrow
  • Collagen gifts, epidermis strength
23
Q

Inflammatory response

A

Mast cells activated and release histamines and cytokines

24
Q

Histamines

A
  • blood vessel dilation raises temperatures to prevent reproduction of pathogens
  • increase leakiness of vessels blood plasma tissue fluid causes swelling
25
Cytokines
Attract phagocytes for phagocytosis
26
Two types of phagocyte
Neutrophil and macrophage
27
Phagocytosis
-pathogens release chemicals that attract phagocyte - phagocyte recognises non-human proteins in pathogen - pathogen encloses in phagosome - phagosome plus lysosome—> phagosome - Enzymes from lysosome digest pathogen
28
What do phagocytes release after engulfing pathogen, what does it do
Cytokines- cell signalling to other phagocytes body is attacked
29
Opsonins
- chemical binds to pathogens so they’re more easily recognised by phagocytes
30
T helper
- produce interleukins that - Stimulate b cells to increase antibody production - stimulate production of other T cells - Attract macrophages
31
T killer
- destroy pathogens
32
T memory
Provide immunological memory Stay in body for a long time Secondary response, divide to form lots of T killer cells
33
T regulator
- insures body recognises self antigens - Stops immune response once pathogen is gone - Prevent auto immune response
34
B effector cell
Divide to form plasma cell
35
Plasma cell
Produce antibodies and release into circulation
36
B memory
Provide immunological memory remember antigen
37
Primary immune response
- Pathogen enters the body and comes in contact with a macrophage - phagocytosis macrophage presents foreign antigen to the immune system(APC) - Virgin Band T cells undergo clonal selection for complimentary shape to antigen - Clonal expansion - Differentiate to different types
38
Secondary response
- memory b and t lymphocytes - Quicker longer stronger
39
Function of antigen biding site
Complementary shape to antigen Binds to antigen
40
Function of disulphide bridge
Stabilises and holds tertiary structure of antibodies
41
Function of constant region
Bonds to receptors on phagocytes
42
Hinge Region function
Flexibility for antibody to bind to more than one antigen on more than one pathogen
43
Antibody acting as opsonin
- makes the antigen antibody complex more easily engulfed by phagocytes
44
Antibody acting as aglutinan
Antigen antibody complexes clump together, preventing them spreading through the body and easy phagocytosis
45
Antibody acting as anti toxin
Bind to toxins to make them harmless
46
Autoimmune disease
Immune system stops recognising self cells and attacks them
47
Example of an autoimmune disease
Type one diabetes Arthritis Lupus
48
Natural active immunity
Body produces own antibodies and memory cells such as primary and secondary immune response
49
Natural passive immunity
In fetus antibodies pass across placenta In newborn babies get antibodies from breastmilk until immune system can make our antibodies
50
Artificial passive immunity
Antibodies extracted from other individuals injected to another can be from animals
51
Artificial active immunity
Vaccination promotes body to make own antibodies