Challenging Populations Flashcards

1
Q

Describe the ABR waveform for ANSD.

A
  • Flat line

- Typically absent of abnormal

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2
Q

Describe the recording parameters for a typical waveform.

A
  • Amplitude: 1-10 uV
  • Spectrum: 0.1-30 Hz
  • Number of averages needed: 100
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3
Q

Describe hearing levels for ANSD.

A
  • Can range from normal to profound (continuum from no auditory complaints to total lack of sound awareness)
  • HL can fluctuate
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4
Q

Describe ANSD and speech understanding.

A
  • May be okay in Q but definitely poor in N

- Delayed N1 latency (associated with worse speech perception in noise)

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5
Q

Compare CAEPs between AN and NH listeners.

A
  • Poor CAEP morphology, reduced peak amplitude, and delayed latencies compared to NH CAEPs
  • As SNR worsens, AN cortical potentials have delayed latencies/reduced amplitudes much faster than NH cortical potentials
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6
Q

Describe P1 latency is a biomarker for speech perception development.

A
  • Normal P1 latencies associated with normal P1 latency
  • Earlier intervention is associated with normal P1 latency
  • Children fitted earlier with HAs, the more normal the brain was able to normalize P1 latencies as a result of amplification
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7
Q

Why would there be a cortical response when ABR is absent?

A
  • ABR reflects highly synchronous discharges with precision on the order of fraction of ms
  • Synchrony is necessary for speech perception in noise
  • Cortex is able to make use of varied and limited input
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8
Q

How do cochlear implants provide the opportunity to study central auditory pathways?

A
  • Maturation
  • Relationships with speech perception
  • Relationship with speech production
  • Sensitive time periods/cortical reorganization
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9
Q

What is the sensitive period for cochlear implantation?

A
  • 3.5 years, during which implantation occurs in a highly plastic central auditory system
  • Implantation after 7;0 occurs in an already re-organized central auditory system
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10
Q

How rapidly does the auditory pathway change following the onset of stimulation of early implanted children?

A
  • Approx. 8 months
  • P1 latencies decrease more rapidly post-implantation in children implanted under 3;0 than in children implanted over 7;0
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11
Q

What is the relationship between development of central auditory pathways and development of speech perception skills?

A

-Lower performance on LNT associated with exaggerated/abnormal P1 latency

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12
Q

What is the relationship between development of central auditory pathways and development of early communicative behavior?

A
  • Examined the link between babbling and auditory pathway development following implantation
  • Is there a relationship between the rate of change in the auditory system and early communication in young CI recipient?
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13
Q

Describe cross-modal cortical reorganization.

A
  • Same sound, different brain (same sound/implant but different outcome)
  • The variable: what the brain does with the sound
  • Brain activation with sound:
    a. NH: auditory cortex
    b. Early implanted: auditory and parietal cortices
    c. Late implanted: parietal cortex
  • Differences in structure/connectivity determine how the brain responds to sound stimulation through the CI
  • MEG activity in response to vibrotactile stimulation shows the cortical re-organization underlies the end of the sensitive period
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14
Q

Why would a clinician want to incorporate evoked potentials in a hearing aid fitting?

A
  • Can be used for evaluation of aided functioning

- Objective hearing aid evaluation for: young infants, difficult-to-test people

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15
Q

Describe the automatic detection of cortical responses.

A
  • Desirable characteristics:
  • No reliance on a template
  • Able to use information from contributing portions of waveforms
  • Able to discount non-contributing portions of waveforms

-Recorded to three speech sounds: /m, g, t/

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16
Q

What are the clinical implications of corticals?

A

-Test with and without amplification to determine how much gain is necessary

  • If noise is low:
    a. No /t/ response–> review HF gain or loss estimate
    b. No /g/ response–> review mid-frequency gain or loss estimate
    c. No /m/ response–> review LF gain or loss esimate
  • If noise is high: draw no conclusions from missing response
  • If responses are absent even with gain, the child would not benefit from HAs
17
Q

What can evoked potentials tell you about neural speech processing?

A
  • Older adults often have difficulty understanding speech in noise, even with the use of digital amplification technology
  • The primary objective methods of HA verification/ REM ensures appropriate sound levels at the eardrum but cannot provide information about what the brain does with the signal
  • FFR: amplification increases detection of speech components in older adults with HL
  • Higher detection and amplitude relate to improved speech discrimination and sound quality ratings
18
Q

How did the HA study show the effects of amplification on the FFR?

A
  • Fitted older adults with H90.3 with bilateral Widex Dream RITEs
  • Worn for 8+ hours daily for 6 months
  • FFR measured to /ga/ at 65 dB SPL, 80 dB SPL, and 80 dB SPL with +10 SNR
  • After 6 months, reduced age-related delays in LLR(increased phase-locking, earlier latencies, increased amplitude in TR region)
  • More robust PLFs for aided vs. unaided
  • Also demonstrated improved working memory
19
Q

Why would evoked potentials be useful in challenging populations?

A
  • Document benefit

- Evaluate neuroplastic changes over time