Ch49.ParasiteControlPrograms Flashcards

1
Q

Define egg reappearance periods

A

period of time from anthelmintic treatment until parasite eggs are found in the feces again

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2
Q

Define prepatent period (PPP)

A

time elapsed from teh uptake of the infective stage of the parasite until it reaches patency and starts hseeding eggs to be detected in feces

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3
Q

What is the PPP of equine parasites?

A

Cyathostomins: 2-3 months
Parascaris equorum: 2-3 months
Anaplasma perfoliata: 1.5 to 4 months
Strongylus vulgaris: 6-7months
Strongylus edentatus: 11-12 months

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4
Q

What is the 20/80 rule in regards to parasite shedding?

A

20% of the host animals harbor approximately 80% of the parasites
** ie: about 20% of the hroses are shedding 80% of the total strongyle egg output

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5
Q

define parasite refugia

A

parasite stages that escaped antihelmintic drugs and are viewd as refugees
** important factoraffecting the rate of development of anthelmintic resistance in large animal parasites

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6
Q

Resistance to what anthelmintics is seen in Parascaris species?

A

ivermectin & moxidectin

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7
Q

Larvae from which parasite penetrate the mucosal lining of SI/stomach and migrate the classical hepatoctracheal route, passing through lungs causing airway symptoms in young foals?

A

Parascaris

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8
Q

Which parasite gives foals clinical signs of ill-thrif, weight loss, rough hair coat and pot-bellied appearance?

A

Parascaris spp

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9
Q

What is the major clinical impact seen with Parascaris infections?

A

verminous small intestinal impactions
**survival beyong 1 year after colic incident associated with parascaris reporte d in 11 of 37 cases

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10
Q

What is a significant risk factor for verminous small intestinal impactions associated with Parascaris in foals and why?

A

anthelmintic treatment d/t instant paralysis elicted by dewormers

** USe Benzamidazole t ype drugs

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11
Q

What are the major clinical consequences of Cyathostomins (small strongyles) infections?

A

excystment– L4 penetrate their cysts & migrate short distance back to the intestinal lumen

*acute typholocolitis– larval cyathostominosis

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12
Q

What is the case fatality rate reported with acute typholocolitis caused by small strongyle larval cyathostominosis?

A

50%

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13
Q

What is the anthlemintic drug of choice for treatment of encysted small strongyles?

A

moxidectin (400 microg/kg PO)

**also labeled is the 5 day dose Fenbendazole (10 mg/kg PO once daily)

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14
Q

Which parasites are associated with ileal impactions and ilecoceal intussceptions?

A

Tapeworms (A. perforliata)

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15
Q

What anthelminitics are labeled for treatment of tapeworms?

A

praziquantel & pyrantel pamoate

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16
Q

Which stage of large strongyles (Strongylins) migrate to the cranial mesenteric artery?

A

L4

17
Q

IN the cranial mesenteric artery, what do large strongyles do?

A

Molt to L5– causing a pronounced verminous endarterities with roughened intima, fibrosis of arterial walls & INC diameter of vessels

18
Q

What is the cause/pathogenesis of Strongylus vulgaris cranial mesenteric arterities?

A

Thrombi detach from the arteritis lesions (caused by L4 to L5 larvae) causing occlusion of terminal branches of vessels leading ot ischemia and infarction of intestinal segments–> colic

19
Q

Foals are usually asymptomatic to Strongylus westeri parasites, how are they exposed?

A

lactogenic transmission from the mare, or through skin

20
Q

What is the equine pin worm

A

O. equi

21
Q

What causes the perineal pruritis assocaited with O. equi infections?

A

egg patches dry up that become pruritic

22
Q

what parasite has been confused with the widely occurring culidcoides hypersensitivity?

A

Onchocerca spp– proundounced skinr eactions ventral midline and face

23
Q

Halicephalobus gingiivals larvae have a special affinity for which organs?

A

CNS
Kidneys
**multifocal, esoinphilic lesions causing C/S blindness, ataxia, CP deficits, head pressing, comba, death, etc.
**infection extremely rare

24
Q
A