Ch 6: Medical Complications Flashcards

1
Q

How many people in the US are living with a long term disability as the result of brain injury?

A
  1. 2 to 5.3 million

1. 1 to 1.7% of the US population

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2
Q

What is DVT and what is the incidence in TBI? How is it treated?

A

Deep vein thrombosis- blood clot forms in vein
54%
Treatment- prophylaxis, compression stockings, vena cava filters (metal device that traps blood clots), intermittent pneumatic compression (device to prevent blood clot)

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3
Q

What are some musculoskeletal complications of TBI? What are common treatments?

A

Spasticity
Hyperreflexia- involuntary increase in muscle tone and deep tendon reflexes
Contractures- a condition of joints, reduced range of motion
Heterotopic ossification- abnormal bone formation after injury

Tx include:
Exercise, casting/orthotic techniques, ultrasounds/estim, meds, surgery/radiation

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4
Q

Dysphagia level 1 (National Dysphagia Diet Level)

A

Puréed diet
Mod-severe
Foods requiring bolus formation, manipulation and chewing are not allowed

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5
Q

Dysphagia level 2 (National Dysphagia Diet Level)

A

Mechanically altered
Mild to moderate and/or pharyngeal dysphagia

Moist, soft and easily form bolus

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6
Q

Dysphagia level 3 (National Dysphagia Diet Level)

A

Dysphagia advanced
Mild
Includes most textures except hard, sticky, or crunchy foods
Requires chewing ability

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7
Q

In comparison to pre injury, how many calories should a person consume during the acute phase of TBI healing? Why?

A

40% more calories

Metabolic needs increase significantly following a moderate to severe injury as the body works to heal the brain

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8
Q

What is diabetes insipidus and how is it treated?

A

Occurs when too little vasopressin is produced and the person produces significantly more urine leading to increased thirst.
Treated with increased fluid and desmopressin

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9
Q

Syndrome of inappropriate anti diuretic hormone (SIADH)

A

Caused by changes to the hypothalamus or certain medications

Blood sodium level is low and urine is not concentrated

Symptoms: nausea, vomiting, irritability, confusion, seizures, coma

**fluid restriction

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10
Q

Metabolic syndrome

A

Combination of medical disorders that increase the risk for both cardiovascular disease and diabetes.

Marked by abdominal obesity, insulin resistance, HTN, and dyslipidemia

Affects 40% of adults over 60

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11
Q

What is the frequency of UTI after brain injury?

A

60% experience UTI within the first 6 weeks after TBI

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12
Q

What percent of brain injury patients report sleep disorders?

A

30-70% of TBI patients

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13
Q

Post traumatic hypersomnia

A

Excessive sleepiness that occurs as a result of traumatic event involving CNS; daytime sleepiness; cognitive and physical fatigue

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14
Q

What is the treatment for insomnia?

A

Treatment includes:

Lifestyle changes, exercise, reg sleep schedule, avoiding naps, limiting fluid before bed, and decreasing caffeine

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15
Q

What is the reported occurrence of post traumatic seizures and what are the three categories?

A

4-53%

3 different categories of seizures
Immediate post traumatic convulsions (IPTC)
Early post traumatic seizures (EPTS)
Late post traumatic seizures (LPTS)

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16
Q

Immediate post traumatic convulsions (IPTC)

A

+LOC and involuntary movement within seconds of impact

Non epileptic events that are more likely passing out

Brief period of tonic positioning followed by clonic or myoclonic jerks of less than 2-3 min

Altered state of consciousness with associated retrograde and anterograde amnesia

Brief traumatic functional decerebration- loss of cerebral brain function

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17
Q

Early post traumatic seizures (EPTS)

A

Occur within 1st week (up to 7 days) following brain injury

Result from primary direct effects of trauma

Incidence report: 2-10% and can occur in mTBI

50% occur within 24hrs of impact; 25% 1st hr

Strong risk factor for LPTS

18
Q

Status epilepticus

A

Seizure that lasts longer than 5 minutes or seizures that occur close together without recovery between seizures; common in children

19
Q

Late post traumatic seizures (LPTS)

A

Occur later than 1 week post injury , but usually within the first 18-24 months (has been reported later)

Interchangeable with post traumatic epilepsy as the occurrence of one seizure likely leads to more
Common in 65+

20
Q

Post traumatic headache (PTH)- when does it start and how prevalent is it?

A

Starts within 14 days of LOC

More prevalent in mTBI (95%) in comparison to 22% of mod-severe reporting pain

21
Q

Most common type of headache reported by all TBI pts?

A

Migraine

22
Q

Peripheral nocioception

A

Peripheral receptors in the head and neck which are very sensitive to pain

Located on the ends of nerves that initiate near the spinal cord and communicate back to pain centers in the brain
Those involved with post traumatic headache:
CN 5 trigeminal, CN 9 glossopharyngeal, CN 10 vagus nerve, greater occipital nerve, lesser occipital nerve

23
Q

Primary headache

A

No specific cause

24
Q

Secondary headache

A

Identifiable cause that can be determined

25
Q

Chronic headache

A

Occurs at least 15 days per month for at least 3 months, not linked to overuse or withdrawal of medication

26
Q

What is a tension type headache?

How is it treated?

A

Most common form of primary headache

Presents with bilateral pressing head pain

Do not worsen with activity and patients do not present with other symptoms

Treatment- Non-steroidal anti-inflammatory drugs or aspirin or acetaminophen
Long term- low load craniocervical mobilization, botulinum toxin, antidepressants, anticonvulsants

27
Q

Migraine headache

A

usually affects one side of the head, are throbbing, and worsen with physical activity, associated nausea or vomiting

28
Q

Cervicogenic headache

A

Pain generated from the cervical spine; treatment- nerve injections, severing nerves

29
Q

Craniomandibular headache

A

Subtype of tension headache that causes pain in jaw with eating and talking
Treatment- bite blocks, diet changes, surgery

30
Q

Which cranial nerve plays a role in the pathophysiology of headaches?

A

CN5 trigeminal

31
Q

What are the 4 phases of migraine?

A

Prodrome- early symptoms
Aura
Headache
Postdrome- other symptoms following the headache

32
Q

Having low sodium levels is a risk for _______.

A

Seizures

33
Q

Neuralgia

A

Type of pain that is caused by damage or change to a nerve

34
Q

Neuroma

A

Nerve becomes entrapped in scar tissue

35
Q

C-O-L-D-E-R Acronym- Headache review of systems

A

Character: throbbing, sharp, dull etc & severity

Onset: does anything set off the headache? Prior hx of headaches, migraines, neck pain, or other related pain involving the head

Location: one side of the head, or both? Front or back? Start in neck? Shooting or radiating?

Duration/freq: when did it start and how often?

Exacerbation: anything that makes it worse?

Relief: does anything help to relieve/resolve the headache?

36
Q

What are complications with functions of elimination after TBI? How treated?

A

Urinary or fecal incontinence, urinary tract infections

Treatment- suprapubic or foley catheter, timed voiding, anti-cholinergics

37
Q

Dysphagia Level 4 (National Dysphagia Diet Level)

A

Regular diet, all foods as tolerated

38
Q

Why are tube feedings important?

A

provide hydration and calories to promote their metabolism and overall health

39
Q

What is the treatment for pressure sores?

A

keep skin clean and change positions every two hours, use of pressure relieving devices;
Oral antibiotics, antifungal creams

40
Q

Periodic limb movement disorder

A

periodic episodes of repetitive and highly stereotyped limb movements during sleep

41
Q

What is the preferred initial medication for aborting a migraine?

A

AAC- aspirin, acetaminophen and caffeine