CEP Exam Flashcards
signs of myocardial ischemia (3)
- ECG abnormalities
- Angina
- Stiffening left ventricle
Angina pectoris
Chest pain, heart cramp
Modifiable factors to prevent CVD?
High Blood pressure
hyperlipidemia
obesity
smoking
physical inactivity
myocardial infarction
Heart attack. Not enough blood to the hear muscle
myocardial ischemia
reduced blood flow to the heart
chronic heart failure
function of the heart fails to meet the requirements
stroke
obstruction of bloodflow to the brain
Marie Tooth disease
muscle athrophy
Post-polio syndrome
muscle weakness, fatigue and pain
sarcopenia
loss of muscle mass and strength
geriatric
ilness and disease of old people
frailty
failure of multiple systems
What delays the process of frailty?
Physical activity, especially higher intensity
What happend during systolic heart faile
- less blood pumped out of the ventricles
- weakened heart muscle can’t squeeze as well
What happend during diastolic heart failure
- less blood fills the ventricles
- stiff heart can’t relax normally
Equation of Stroke Volume
SV = EDV - ESV
What is ejection fraction
How much blood is pumped away per pump.
Consequences for exercise measurements in CVD / Structural/functional changes in the heart in CVD? (5)
- Cardiac athrophy
- reduced blood volume
- reduced vascular compliance(increased arterial stiffness)
- weakened contractility heart
- reduced capillarisation of active tissue
Consequences for exercise measurements in CVD / Structural/functional changes in the lungs in CVD? (5)
- increase pulmonary vascular pressures
- higher stiffnes
- higher ventilation (VE)
- higher respiratory muscle work
- lower diffusion capacity
Consequences for exercise measurements in CVD / Structural/functional changes in the skeletal muscle in CVD? (4)
- lower blood flow
- lower mitochondrial volume
- lower % type 1 fibers
- higher glycolytic enzymes
What are the consequences of a CVD for the respiratory chain or energy metabolism
- The fick’s equation predicts the consequences for energy expenditure
- The lungs, muscles and heart affect this equation
- a-v O2diff is smaller
- aterial: lungs give less oxygen
- venous: muscles use less oxygen
CPET results in CVD - cardiac responses
- HRpeak not affected
- O2 puls (VO2/HR) –> VO2 is lower
CPET results in CVD - functional capacity
- VO2 peak is reduced
- Peak WR is decline
CPET results in CVD - ventilation/gas exchange
- MVV (no change)
- VT1 & VT2 (lower exercise intensity)
What are the exercise responses in chronic heart failure?
- reduced VO2peak
- Abnormal delta VO2/WR
- High HR/WR
- Low VO2/WR
- increase in HR vs VO2
What are the 5 Electrical phases heart cycle
- SA node activity
- stimulus spread to AV node
- Atria contraction begins
- impulse travels from purkinje fibers to right ventricle
- atria contraction completed. ventricular contraction begins
right atria
deoxygenated blood from body
right ventricle
pums deoxygenated blood to the lungs
left atria
receives oxygenated blood from the lungs
right atria
pumps oxygenated blood to the body
function of the SA-node
generates electrical activity
function of the AV-node
generates delay in the conduction system
What says an abnormality in the P-wave
something wrong with atria
What says an abnormality in the QRS-complex
slower conduction of the ventricular depolarization
What says an abnormality in the PR-interval
less/more time between contraction of atria and ventricles
bradycardia
abnormal low HR < 60
tachycardia
abnormal high HR > 100
Objective criteria to determine exercise intensity? (3)
- VO2max Reserve or METs
- HRReserve (karvonen)