Cardiovascular Emergencies Flashcards

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1
Q

Diseases which increase cardiac preload / volume overload

A

Fluid overload, renal dysfunction, poor compliance with duietics, acute valvular regurgitation

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2
Q

Diseases which decrease cardiac preload / impair filling

A

Tachycardia, pericardial constrictive disease, restrictive cardiomyopathy, left ventricular hypertrophy, myocardial fibrosis, hyvolaemic shock

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3
Q

Diseases which increase afterload / pressure overload

A

Uncontrolled HTN and acute PE

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4
Q

Diseases which impair contractility / myocardial loss

A

Acute myocardial infarction, drug overdose, dilated cardiomyopathy, fever

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5
Q

Disease which cause high output failure

A

Fever,anaemia, thyrotoxicosis

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6
Q

What is the result of low cardiac output and hypotension on ischaemia

A

Sympathetic stimulation and RAAS causing tachycardia, vasoconstriction, Na/H2O retention. This causes O2 consumption of myocytes exacerbating ischaemia

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7
Q

What is the result of systemic hypoperfusion on fluid retention

A

Leads to lactic acidosis and pre-renal failure, which then exacerbates fluid retention.

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8
Q

Clinical presentations of heart failure

A

Decompensated, pulmonary oedema, cardiogenic shock, hypertensive HF, RHF, AHF associated with ACS

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9
Q

Different types of classifications of heart failure

A

Heart failure can be classified into perfusion and congestion, clinical presentation, NYHA functional class, LVEF

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10
Q

Way to work out the perfusion/congestion classification of HF

A

If there is evidence of raised filling pressures or congestion - no or yes
If there is evidence of systemic hypoperfusion - no or yes

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11
Q

Outcomes of perfusion / congestion classifications

A

No congestion or hypoperfusion - Warm and dry
Congestion but no hypoperfusion - warm and wet
Congestion and hypoperfusion - cold and wet
No congestion but hypoperfusion - cold and dry

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12
Q

Causes of myocardial injury

A

Acute myocardial ischaemia, acute myocardial ischaemia from O2 supply/demend imbalance, non ischaemic myocardial injury

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13
Q

Causes of acute myocardial injury from decreased supply

A

Coronary artery spasm, embolism, dissection, bradydysrhythmias, hypotension, shock, resp failure, severe anaemia

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14
Q

Causes of acute myocardial injury from increased demand

A

Sustained tachy-dysrhythmias, severe hypertension

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15
Q

Causes of non-ischaemic myocardial injury

A

HF, myocarditis, cardiomyopathy, catheter ablation, defibrilator shocks, cardiac contusion, sepsis, infectious diseases, CKD, infiltrative diseases, chemotherapeutic agents

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16
Q

How to spot inferior coronary infarct on ECG

A

II, III and aVF

17
Q

How to spot anteroseptal coronary infarct on ECG

A

V1 and V2

18
Q

How to spot an anterior or anteroapical coronry infarct on ECG

A

V3 and V4

19
Q

How to spot anterolateral coronary infarct on ECG

A

V5, V6, I and aVL

20
Q

Which condiction can mimic ACS

A

Aortic dissection

21
Q

How can you differentiate between ACS and aortic dissection

A

Check blood pressure in both arms -> if these measurements are different it can be an aortic dissection

22
Q

Revascularisation options for non ST elevated MI

A

Risk assessment using TIMI/GRACE then either PCI, CABG, or conservative

23
Q

Revascularisation options for ST elevated MI

A

pPCI or thrombolysis

24
Q

Eg of anti-thrombotics

A

Dual antiplatelets and anti coagulants

25
Q

Adjuvants of ACS management

A

ACEI, statins, plaque stabilisation, vent remodelling, morphine, oxygen if hypoxaemic

26
Q

Eg of anti-ischaemic agents

A

B blockers, nitrates, CCB

27
Q

Common conditions in cardio ED

A

ACS, aortic dissection, HF

28
Q

Reversible causes of cardiac arrest (4H’s and 4T’s)

A

Hypoxia
Hypovolaemia, Hyperkalaemia/hypo, hypoglycaemia, hypocalcaemia
Hypothermia
Thrombosis
Tension pneumothorax
Tamponade
Toxics