Abdominal Emergencies Flashcards

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1
Q

3 types of pain the abdomen can experience

A

Visceral, somatic and referred

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2
Q

What is visceral pain like

A

Afferent nerve fibres, dull, poorly localised, can be from any area (foregut, midgut, hindgut)

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3
Q

What is somatic pain like

A

Parietal peritoneum, sharp, well localised, made worse by movement, better lying still

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4
Q

What causes referred pain

A

From nerves transmitting visceral and somatic pain

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5
Q

Causes of colicky pain

A

Intestinal obstruction

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6
Q

Causes of constant pain

A

Peritoneal irritation

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7
Q

Pain from small gut

A

More frequent pain, mid abdomen

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8
Q

Pain from large gut

A

Lower abdomen, hindgut structure, frequency of contractions reduced

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9
Q

What is peritonitis

A

It is an examination finding, where there is guarding or percussion tenderness. Can be localised or generalised

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10
Q

Causes of primary peritonitis

A

Spontaneous bacterial peritonitis

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11
Q

Causes of secondary peritonitis

A

Perforation, appendicitis

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12
Q

Causes of important abdominal emegencies

A

Ruptured AAA, cholecystitis, appendicitis, diverticulitis, ischaemic bowel, bowel obstruction, pancreatitis

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13
Q

Risk factors for gallstones

A

Fat, female, fertile, forty, family history - also weight loss, increasing age, metabolic syndrome

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14
Q

Consequences of gallstones

A

Biliary colic, cholangitis, cholecystitis, severe cholecystitis, pancreatitis

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15
Q

Causes of bowel obstruction

A

Adhesions (congenital bands, post-op, inflammatory), hernias, vovulus, intraluminal (tumour or FB), strictures (malignant - colorectal, benign - crohns)

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16
Q

What is a simple obstruction

A

One point of obstruction and no vascular compromise

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17
Q

What is a closed loop obstruction

A

2 points of obstruction such as lower bowel obstruction with competent IC valve, leading to distension and perforation

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18
Q

What is a stangulated obstruction

A

Vascular compromise, ischaemic, perforation, peritonism, fever and raised WCC

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19
Q

Complications of perforated diverticulitis

A

Local/pelvic abscess, purulent peritonitis, faecal peritonitis, purulent and faeculant

20
Q

What makes up an appendix mass

A

Small bowel, omentum, intraabdominal fat, sometimes colon and fallopian tube

21
Q

Why does an appendix mass form

A

Body tries to seal off the infection

22
Q

Treatment of appendix mass

A

Usually treated with antibiotics and avoid surgery as it normally settles.
If >4cm then radiologically drain.

23
Q

What is happening if a patient is moderately well with appendicitis

A

Local infection and abscess, can become walled off with omentum

24
Q

What is happening if a patient is unwell with appendicitis

A

Inflammation, ischaemia, perforation, and widespread peritonitis

25
Q

Symptoms of cholangitis

A

Ascending cholangitis, obstructive jaundice, pain or no symptoms

26
Q

How to rule out AAA

A

US scan

27
Q

Key finding in ischaemic bowel

A

High lactate

27
Q

Key finding in ischaemic bowel

A

High lactate

28
Q

When does amylase peak in pancreatitis

A

12 hours after

29
Q

Symptoms of upper GI bleed

A

Haematemesis, melaena, syncope, dizziness, haematochezia, hypotension, tachycardia

30
Q

Most common causes of upper GI bleeds

A

Peptil ulcer, mallory-weiss tear, erosion, oesophagitis, varices

31
Q

Key history points for upper GI bleed

A

Liver disease, profuse recent vomiting, peptic ulcer disease, gastritis, H.pylori infection, alcohol, NSAIDs

32
Q

What suggests a variceal bleed

A

Evidence of decompensated liver disease, jaundice, ascites, encephalopathy

33
Q

What is the Glawgos-Blatchford score used for

A

Risk assessment tool of choice in ED for management of upper GI bleed

34
Q

What is the Rockall score used for

A

Risk assessment tool for identifying high risk upper GI bleeds

35
Q

Management of somatostatins in variceal bleeds

A

Somatostatins (Octreotide) and vasopressins (Terlipressin), broad spectum antibiotics

36
Q

Management of non-variceal bleeding

A

Endoscopy

37
Q

What is a balloon haemorrhage

A

Effective method of controlling a variceal haemorrhage until definitive intervention

38
Q

Common causes of lower GI bleeding

A

Diverticular disease, IBD, neoplasia, benign anorectal disease, angiodysplasia

39
Q

What is included in the BLEemergency department criteria

A

Ongoing bleeding, hypotension, abnormal clotting, altered mental status, significant co-morbidities

40
Q

What does the BLEED classification stand for

A

Bleeding
Low systolic BP
Elevated prothrombin time
Erratic mental status
Disease - unstable comorbid

41
Q

What is the management of an unstable lower GI bleed

A

Colonscopy +/- angiography if massive

42
Q

Management of stable patient with lower GI bleed

A

Colonscopy

43
Q

Other investigations for lower GI bleeding

A

Proctoscopy, technetium labelled red blood cell staining, upper GI endoscope, CT angiography, recto-sigmoidoscopy

44
Q

When is surgical intervention required for lower GI bleeds

A

Haemodynamically instability persists despite aggressive resuscitation or bleeding continues/recurs