Cardiology Flashcards

1
Q

What is the most common symptomatic congenital heart disease

A

VSD’s

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2
Q

Which genetic conditions have increase of VSDs

A

Downs syndrome
DiGeorge syndrome

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3
Q

What type of shunt is VSD

A

left to right

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4
Q

Types of murmurs in VSD

A

small - panystolic at left lower edge, ass. thrill

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5
Q

Medications to treat VSD

A

diuretics
ACEi

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6
Q

What is the pathophysiology of WPW

A

Normal heart - insulating fibre tissue between atria and ventricle - signal has to go via AVN node
In WPW - the band of fibre has myocardial cells which becomes accessory pathway

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7
Q

What is the delta wave?

A

conduction through accessory pathway leads to excitation of the ventricle before conduction of AVN = shortened PR interval
conduction is slower than AVN node which is why it is slurred upstroke of QRS

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8
Q

What is contraindicated in AF and WPW

A

Adenosine - could lead to VF

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9
Q

Treatment for WPW

A

slow conduction down

beta-blockers
amiodarone
flecainide
calcium-channel blocker

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10
Q

What is the most common cardiac condition ass. with Noonan syndrome

A

Hypertrophic cardiomegaly

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11
Q

What does hypertrophy fo LV cause?

A

reduces LV cavity volume
reduced ability of LV to fill in diastole

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12
Q

What does reduce filling in LV cause?

A

Rising left-atrial pressure
pulmonary oedema
congestive cardiac failure

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13
Q

What are the forms of hypertrophic cardiomegaly

A

Obstructive
Non-obstructive - most common

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14
Q

Why does obstructive cardiomegaly form?

A

Dynamic obstruction of LV outflow by anterior leaflet of mitral valve
Due to abnormality of papillary muscle insertion and abnormal flow in LV

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15
Q

What are most common symptoms in HOCM

A

angina
dizziness

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16
Q

Examination findings with patients with HCM

A

Displaced apex and heaving
additional heart sounds S3 S4

17
Q

ECG changes of ventricular hypertrophy

A

left axis deviation and large QRS complexes in chest leads = LVH ST segment changes and t wave inversion in V4-V6 left heart strain

right axis devotions and large qrs in II, III, avF, 1-3 = RVH

18
Q

medications for hypertrophy

A

beta blockers - slow heart rate, improve filling
diuretics
calcium channel blockers
ACE I

19
Q

When does ToF present if not detected antenatally

A

within 1 year with cyanosis and murmur

20
Q

what sign is seen on CXR for coractation of aorta

A

rib notching

21
Q

What murmur is coarctation of aorta associated with

A

ejection systolic murmur radiates to back

22
Q

What happens in TGA

A

Aorta rises from right ventricle
pulmonary artery arises from left ventricle

23
Q

What increases the incidence of TGA

A

DiGeorge syndrome
Infant of mum with diabetes

24
Q

What is CXR sign of TGA

A

egg on side appearance
pulmonary pleathora

25
What type of discordance is TGA
ventriculoaterial discordance
26
What is Marfan's syndrome associated with?
Aortic root dilatation leading to aortic regurgitation
27
Phase of cardiac action potential
Phase 0 - influx of sodium Phase 1 - end of depolarisation - efflux of potatissium pHASE 2 - Plateau phase - slow influx of calcium Phase 3- rapid repolarisations - efflux of potassium Phase 4 - diastole, calcium-sodium efflux, potassium influx
28
Signs of servere aortic stenosis
Soft S2 second heart sound So narrowed and calcified - 2nd here sound becomes inaudible
29
Why is VT broad complex
rhythm starts from ventricle
30
Is there P waves in VT?
No as SAN isn't controlling the rhythm If there is - they will be unrelated to ARS complex
31
Which aortic arch forms right subclavian artery
right 4th aortic arch
32
Which aortic arch forms internal carotid artery
third arches
33
Adenosine doses for SVT
Start adenosine 0.1mg/kg 3mg -> 6mg > 12mg If shocked - synchorous shock 1joules/kg