Cardiology Flashcards
3 Is, 4 Ps, DTU
Causes of pericarditis
Infectious - usually viral (80% are viral or idiopathic)(coxsackie, COVID), can be bacterial, fungal, TB (poorer prognosis)
Immunological - SLE, rheumatic fever, vasculitis
Idiopathic
Post MI (Dressler’s syndrome)
Post cardiac surgery
Post RTx
Paraneoplastic
Trauma
Drug induced (isoniazid, cyclosporin)
Uraemia
ECG changes in pericarditis
Widespread concave STE (esp inferior and precordial) with reciprocal STD in aVR and V1
Widespread PR depression with reciprocal PR elevation in aVR and V1
Sinus tachycardia
Spodick’s sign
Differences between ECG for BER and pericarditis
Pericarditis:
-STE widespread in limb and precordial leads (BER pre cordial only)
-no “fish-hook” J point pattern (best seen in V4 in BER)
-ST/T wave ratio >0.25 (BER <0.25)
-presence of PR depression
-normal T wave amplitude
-dynamic ECG changes that evolve over time
Differences between ECG for STEMI and pericarditis
STEMI has reciprocal changes (pericarditis reciprocal only in aVR and V1)
Only STEMI causes convex or horizontal STE
STEMI causes STE > in III than II
PR depression in multiple leads is more likely pericarditis
Diagnosis of pericarditis
At least two of:
Chest pain (retrosternal, sharp, pleuritic, can radiate to L shoulder, better with leaning forward)
Pleural rub
Pericardial effusion
ECG changes of widespread STE and PR depression (<60% of patients)
NZ Criteria for Rheumatic Fever
2 major OR 1 major and 2 minor OR several minor
AND
Evidence of recent GAS infection (2-3 weeks)
Major:
Carditis (echo/new murmur)
Chorea (can be standalone diagnostic)
Polyarthritis (or aseptic mono arthritis)
Erythema marginatum
Subcutaneous nodules
Minor:
Fever
Raised ESR/CRP
Polyarthralgia
Prolonged PR interval on ECG
What is the OR of acute rheumatic fever in Maori and Pasifika cf other ethnicities?
> 20x more likely in Maori
40x more likely in Pasifika
Describe some key features of HFpEF
-EF >50%, LV normal volume
-usually females, older adults
-LV wall is thickened/stiff
-poorer prognosis/less amenable to drugs
List some causes of high output cardiac failure
Thyrotoxicosis
Obesity
Pregnancy
Anaemia
Shunting
Vit B1 deficiency
Describe the NYHA levels of HF
Class I - no limitation
Class II - ordinary activity causes SOB
Class III - less than ordinary activity causes SOB
Class IV - SOBAR
Hx and exam in HF
Hx:
Cough
SOB
Wheeze
Collapse
Palpitations
Decreased exercise tolerance
Orthopnoea/PND
Chest pain
Lethargy
Exam:
Elevated JVP
Tachycardia
Tachypnoea
Sweaty/clammy
Hypoxia
Hepatomegaly
Gallop rhythm/abnormal rhythm
Crackles
Oedema
Cyanosis
Treatment for acute HF
Oxygen
SL nitrates
Diuretics
Morphine
B blocker
ACE inhibiter/ARB
Aspirin
Risk factors for HF
IHD
Arrythmia
Smoking
HTN
High cholesterol
Increased age
Male
FHx heart disease
Treatment for chronic HF
If overloaded start with frusemide, add thiazide if required
Start ACE-i/ARB
Start B-blocker once fluid overload settled
If insufficient add spironolactone
If insufficient add entresto
If insufficient refer cardiology
Add SGLT2 inhibitor if diabetic
Digoxin if AF + consider anticoagulation
High risk features of syncope
Age >65
ANY ECG changes (esp ST/T changes, any conduction abnormalities, any QT prolongation or shortening, delta waves, brugada syndrome, LVH, RVH, bradycardia, abnormal Q waves, arrhythmia)
Palpitations
Dyspnoea
HCT <30
Abdo pain/back pain
Headache
Chest pain
SOB
Occurred while supine or exercising
No prodrome
Heart failure/IHD/structural heart disease/new murmur
Hypotension
Evidence of haemorrhage (malena)
Male
FHx sudden cardiac death <50yo