Cardiology Flashcards
bradycardia examples
sinus bradycardia
sick sinus (tachy-brady)
sinus arrest vasovagal
sinus bradycardia causes
athletes
drug toxicity - beta blockers, calcium channel blockers, digoxin
asymptomatic sinus bradycardia management
NIL
symptomatic sinus bradycardia management
permanent pacemaker
sinus bradycardia origin
SAN / atria
first degree heart block ECG features
elongated PR interval
1D HB management
check for drug toxicity (digoxin)
NIL management if asymptomatic
if symptomatic - prov cardiac monitoring
Type I second degree heart block ECG features
longer, longer, longer, drop!
PR gradually elongating, then dropped QRS complex
2D HB Type I causes
high vagal tone ie. young, fit, healthy
post inferior MI
2D HB Type I management
NIL
Type II second degree heart block ECG features
sudden failure of P wave to be conducted to ventricles
ie. randomly dropped QRS
Type II 2D HB management
permanent pacing
third degree heart block ECG features
no relationship between p waves and QRS complexes
can have broad complex escape (beneath AVN) or narrow complex escape (above AVN)
third degree heart block causes
digoxin toxicity
post inferior STEMI
sev hyperkalaemia
third degree heart block management
atropine IV
isoprenaline
calcium chloride IV (hyperkalaemia)
permanent pacing
what is the most common cardiac arrhythmia encounter in clinical practice
AF
AF presentation
not typically presenting with symptoms
BUT - will present as a complication of another condition eg.
haemodynamic instability (due tachy / brady)
ACS
CCF
cardioembolic stroke
complications of AF
cardioembolic stroke
cardiac instability
death
AF diagnosis
manual pulse checks
ECG
(SOB, palpitations, syncope/dizziness, CP, stroke, TIA)
? paroxysmal - cardiac monitoring (24 hour cardiac monitor)
AF classifications
paroxysmal > persistent > permanent
Echocardiogram indications
structural heart disease
consideration of rhythm control strategy (eg. cardioversion)
baseline needed for LT treatment
AF management
- anticoagulation (stroke / systemic embolism prophylaxis) eg. apixaban
- Rate control
- Rhythm control
- CHAD2S2 VASC Score / HAS-BLED Score
DOACS MOA
apixaban = inhibit factor Xa dabigatran = inhibit direct thrombin inhibitor
DOACs excretion
kidney
monitor renal function yearly
DOAC benefit
less need for regular monitoring (INR)
lower rates of bleeding (vs warfarin)
better reduction in strokes
Rhythm control options
Haemodynamically unstable = electrical cardioversion
Haemodynamically stable =
+ SHD = IV amiodarone
+ no SHD = IV flecainide
Rate control options
beta blocker
digoxin
supraventricular tachycardia pathophysiology
AVNRT (AVN reentry tachy)
AVRT (atria-ventricular reentry tachy)
first line treatment SVT
vagal manoeuvres
- breath holding
- Valsalva manoeuvre
- carotid massage
vagal manoeuvre MOA
slow AVN conduction > interruption of reentrant circuit
carotid massage risks
stroke / emboli
- always ascultate for bruits
- mostly used in younger patients
second line / 1st line drug treatment SVT
IV adenosine OR IV verapamil / diltiazem
adenosine = rapid IV bolus 6 mg stat + long saline flush in antecubital fossa
(if unsuccessful 12 mg stat, 12mg stat)
adenosine SEs
chest discomfort
transient hypotension
flushing
adenosine contraindications
reversible airways disease
significant bradycardia / tachycardias
adenosine MOA
reduces HR and conduction velocity @ AVN
atropine MOA
reduces vagal tone > ^ AVN conduction
synchronised cardioversion indication for SVT
SVT \+ hypotension \+ pulmonary oedema \+ chest pain and ischaemia \+ unstable Nb. under GA / sedation
verapamil contraindications
pts on BXs
flecainide MOA
Na channel blocker
flecainide contraindications
MI - past or present
2nd line drug treatment for SVT
flecainide
amiodarone
sotalol
HTN Target low-mod risk
<140 mmHg systolic
HTN target DM / stroke / TIA / IHD / CKD
< 130/80
HTN target >80 years old
140-150 systolic
HTN diastolic target
<90
HTN + DM diastolic target
<85
HTN first line tx <55 y/o
ACEi / ARB
HTN first line >55 y/o OR black (of any age)
CCB