Cardiac Flashcards
Cardiac Output (shock) for Pediatrics
Initial: Tachycardia
Late: Hypotension
Threatening: Bradycardia
Stroke Volume is influenced by
Contractility
Preload
Afterload
What is preload?
How is it decreased & increased?
Volume! Measured by central venous pressure on right.
< decreases from hypovolemia and vasodilators ie Lasix
> increases with fluid, blood, vasoconstrictors
What is afterload?
How is it decreased and increased?
Ventricular emptying evaluated by systemic vascular resistance
<decreased> increased with HTN and vasopressin’s ie norepinephrine
</decreased>
What increases your heart rate in response to shock?
Sympathetic nervous system (SNS) cause the release of epi and norepi
What decreases heart rate in shock? (seen in neurogenic shock)
Parasympathetic nervous system
Mean Arterial Pressure (MAP)
Equation
DBP x 2 + SBP / 3
Narrow Pulse Pressure
Early Shock
Widened Pulse Pressure
Seen in ICP
Cushing Triad
An indication of >ICP
Widening pulse pressure
Bradycardia
Irregular breathing pattern
Chronotropes
Affect the heart rate at the SA node
ex: cardizem
Inotropes
Affect contractility by force of contraction
Ex: Dopamine
Dromotropes
Affect automaticity of electrical impulses at the AV node
ACE inhibitors
(angiotensin-converting enzyme)
Reduces BP by blocking conversion of angiotensin 1 to angiotensin 2
Ex: Lisnopril
beta-blockers
(olols)
*Mask signs of shock & hypoglycemia
Beta 1 meds affect the heart
Beta 2 meds affect the heart & lungs
Ex: Labetalol, metoprolol, propranolol
adverse effects of ACE Inhibitors
Dry nonproductive cough which leads to noncompliance
Angioedema
ARBS
(angiotensin receptor blockers)
*Reduces BP by inhibiting angiotensin 2 receptors
Ex: Avapro, Cozaar, Diovan
calcium channel blockers
Controls ventricular rate in Afib and HTN
Ex: Cardizem, Norvasc, Nifedipine
What does Nitroglycerin
do?
*Vasodilates
< preload & afterload
< BP
< O2 consumption
-contradicted with Cialis
What does Nitroprusside do?
*Vasodilates
<preload & afterload used in HTN crisis
What are examples of vasopressors?
Epi & Norepi
stable angina pectoris
-occurs with physical exertion
-short duration
-relieved by rest
- neg trop
unstable angina
-chest pain with little physical exertion,
-longer duration
-not relieved by rest
-neg trop
NSTEMI
*plaque rupture
*absent ST elevation
- pos trop
STEMI
*obstruction with thrombosis
*ST elevation
- pos trop
Prinzmetals
(variant angina)
-cyclical pain at rest
-ischemia d/t coronary vasospasm which is precipitated by stress or stimulants