Burns Flashcards

1
Q

Are acid or alkali chemical burns more serious?

A

ALKALI in general.

Body cannot buffer the alkali.

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2
Q

Why are electrical burns so dangerous?

A

Most of the destruction is internal.

Cardiac dysrythmias, myoglominuria, acidosis and renal failure are common.

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3
Q

Treatment for myoglobinuria

A

To avoid renal injury, think “HAM”.
Hydration
Alkalization with IV HC03
Mannitol diuresis

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4
Q

Epidermis only

A

First degree

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5
Q

Epidermis and varying levels of dermis

A

Second degree

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6
Q

Full thickness

All layers of the skin including the entire dermis

A

Third degree

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7
Q

Burn into bone or muscle

A

Fourth

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8
Q

Painful dry, red areas that do not form blitsters(sunburn)

A

First degree

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9
Q

Painful, hypersensitive, swollen, mottled areas with blisters and open weeping surfaces

A

Second degree

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10
Q

Painless, insensate, swollen, dry, mottled, white, charred areas; DRY LEATHER

A

Third degree

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11
Q

What is the major clinical difference between second and third degree burns?

A

Third degree burns are painless

Second are painful

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12
Q

Which measure is burn severity determined?

A

Depth and TBSA affected by second and thired degree burn

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13
Q

Treatment of second degree burns

A

Remove blisters, apply antibiotic

Silver ion dressings

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14
Q

Third degree burn treatment

A

Early excision of eschar( within first week of postburn) and STSG

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15
Q

what prophylaxis should the burn patient get in the ER?

A

Tetanus

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16
Q

What is used to evaluate the eyes after third degree burn?

A

Fluorescein

17
Q

Diagnostic imaging used for smoke inhilation

A

bronchoscopy

18
Q

Lab test for smoke inhilation

A

Carboxyhemoglobin

19
Q

Loss of fluid from the intravascular space as a result of burn injury, which causes “leaking capillaries” that require crystalloid infusion

A

burn shock

20
Q

What is the Parkland Formula

A

TBSA x wt in kg x 4
1/2 in first 8
1/2 in the next 16 hours

21
Q

Adult urine output goal for burn.

A

30-50cc

22
Q

Why is glucose-containing IVF contraindicated in burn patients in the first 24 hours?

A

Serum glucose will be elevated on its own because of the stress response

23
Q

Minimal urine output for burn patients

A

Adults 30cc; children 1-2 cc/kg/hr

24
Q

Best monitoring tool for volume status

A

Urin output

25
Q

Why do most severely burned patients require nasogastric decompression?

A

Patients with >20% TBSA burns usually develop paralytic ileus –> vomiting –> aspiration risk –> pneumonia

26
Q

What stress prophylaxis must be given to burn patients?

A

PPI to preven burn stress ulcer

27
Q

What is the most common sign of burn wound infection?

A

discoloration of burn eschar

28
Q

What are the common organisms found in burn wound infection?

A

Staph, Psudomonas, strep, candida

29
Q

Why are systemic IV antibiotics contraindicated in fresh burns?

A

Bacteria live in the eschar, which is avascular (the systemic antibiotic will not be delivered to the eschar); thus, apply topical antimicrobial agents

30
Q

Are prophylactic antibiotics administered for burns patients including inhalational injury?

A

NO

31
Q

Circumferential, full-thickness burns to the extremities are at risk for what complication?

A

distal neurovascular impairment

32
Q

What is the major infection complication in burn patients?

A

Pneumonia, central line infection

33
Q

From which burn wound is water evaporation highest?

A

Third-degree

34
Q

Can infection convert partial-thickness injury to a full thickness injury?

A

YES

35
Q

How is carbon monoxide inhalation overdose treated?

A

100% O2

36
Q

Which electrolyte must be closely followed acutely after a burn?

A

Na

37
Q

What is the name of the gastric/duodenal ulcer associated with burn injury?

A

Curling’s ulcer

Think: CURLING iron