Bundle branch blocks Flashcards

1
Q

What is appearance of RBBB and LBBB in V1?
What limitations of diagnosing underlying disease when RBBB/LBBB present?

A
  • RBBB severely limits the ability to diagnose RVH
  • LBBB severely limits the ability to diagnose LVH, and an ST elevation MI
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2
Q

What is appearance of RBBB/LBBB in V6?

A
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3
Q

What is criteria for RBBB?

A
  • QRS duration >120ms
  • An M shaped QRS complex in V1
  • Prominent S wave in lead I and lead aVL
  • Secondary repolarization abnormalities are usually seen in V1-V2
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4
Q
A

In lead V1: R’ is more prominent than R
In lead I and lead aVL: prominent S wave

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5
Q

What is the criteria for LBBB?

A
  • QRS duration >120ms
  • Broad R wave in lead I, aVL, and V6
  • Lack of septal q waves in lead I, and V6

Widespread secondary repolarization abnormalities should also be present
* Leads I, aVL, V6 usually display a downsloping zst depression leading to an inverted T wave
* Leads V1-V3 usually display a deep S wave, with upsloping ST elevation leading into a upright and prominent T wave

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6
Q
A

LBBB
* Broad R wave and deeply inverted T waves in lead I, aVL and V6
* Deep S wave and ST elevation and prominent T wave in V1-V3

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7
Q

Causes of RBBB

A
  • Coronary artery disease
  • Pulmonary hypertension
  • Acute pulmonary embolism
  • Idiopathic
  • Iatrogenic during right sided cardiac catheterization
  • Congenital heart disease
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8
Q

Causes of LBBB

A
  • Acute anterior MI
  • Coronary artery disease
  • LVH (any cause)
  • Heart failure (any cause)
  • idiopathic
  • HyperK
  • Digoxin toxicity
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9
Q

What is criteria of left anterior fascicular block (LAFB)?

A
  • LAD (usually beyond -45): as only depolarization is from inferior septal/medial rotation upwards
  • Small q in leads I and aVL
  • Small r in leads II, III and aVF
  • Intrinsicoid deflection in aVL >45ms

Precordial leads are not typically affected

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10
Q

What is criteria of left posterior fascicular block (LPFB)?

A

Not as common as left anterior fascicular block
* RAD: depolarization only from anterolateral rotation (goes down and back)
* Small r in leads I and aVL
* Small q in leads II, III and aVF
* Intrinsicoid deflection in aVF >45ms
* No additional evidence of RVH (tall R wave in V1 or RAE)

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11
Q

Define bifascicular block?
What is the causes?

A

RBBB + either LAFB/LPFB

Causes
* CAD (most common)
* Degenerative disease of conduction system
* Hypertension
* Aortic stenosis
* Congenital heart disease

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12
Q
A

Bifascicular block: RBBB combined with LAFB

Axis: Lead I +ve, lead aVF negative, lead II -ve = left axis deviation

RBBB results:
Prominent S wave in lead V1 and lead aVL
M shaped QRS complex in lead V1
Secondary repolarization abnormalities in lead V1-2: T wave inversion in lead V1

LAFB results:
Small q waves in lead I and aVL
Small r waves in leads II, III and aVF

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13
Q
A

RBBB:

LPFB results:
Axis: lead I is -ve, lead aVF is +ve = right axis deviation
rS (small r) pattern in leads I and aVL
qR (small q) pattern in leads II, III and aVF

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14
Q

What are the progressive cardiac conduction diseases causing bundle branch block?

A

Levs disease: sclerosis and calcification of the cardiac skeleton
Lenegres disease: primary degenerative disease of the conduction system

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15
Q
A

Mobitz type 2 second degree heart block: same PR interval than drops
Mobitz type 2 (below AVN): widened QRS complex

Goes from 3:1 type 2 mobitz second degree heart block to mobitz type 2 2nd degree heart block with a 2:1 AV block

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