Biological approach to OCD Flashcards

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1
Q

what does the genetic explanation of OCD argue that genetics are the cause of…

A

genetics are the cause of the changes in NEUROTRANSMISSION which in turn causes the ocd behaviour

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2
Q

what does the COMT gene regulate the production of

A

the neurotransmitter, dopamine

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3
Q

what does the COMT gene produce lower levels of

A

catechol-O- methyltransferase (comt)

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4
Q

what does the COMT gene produce higher levels of

A

dopamine

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5
Q

TRUE OR FALSE: low dopamine levels are found in people with OCD

A

FALSE
dopamine levels are thought to be higher in individuals with OCD

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6
Q

explain how dopamine causes people with OCD to perform certain behaviours

A

as dopamine is part of the learning pathway
it could be argued when dopamine is high
people perform a behaviour to respond to an obsession repeating this behaviour developing a compulsion

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7
Q

what does the SERT gene affect the transport of

A

seretonin

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8
Q

TRUE OR FALSE: the SERT gene creates lower levels of seretonin in people with OCD

A

TRUE
there are lower levels of the neurotransmitter

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9
Q

how does lower production of seretonin result in OCD

A

low seretonin = lower connectivity of neural pathways
leading to thoughts not to be entirely complete
leading them to be stuck on certain thoughts
as a result obsessive thoughts are caused

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10
Q

what is seretonin associated with

A

happiness

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11
Q

due to lower levels of seretonin it can argued that people with OCD are..

A

lacking happiness

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12
Q

how is it known that those with OCD lack happiness

A

it can be seen that antidepressants drugs increase seretonin activity and has been shown to reduce OCD symptoms

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13
Q

what is the ‘normal’ neural process of worrying

A

orbitofrontal cortex sends worry signal

the caudate nucleus then suppresses signals from the OFC

this suppression prevents the thalamus from taking action

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14
Q

what is the ‘ocd’ neural process

A

the orbitofrontal cortex sends worrying signals

the caudate nucleus doesnt suppress these signals

this leads to the thalamus sending signals back to the OFC to cause panic and worry

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15
Q

true or false: do people with OCD have the same neurological pathway as everybody else

A

FALSEEE
those with OCD have different neurological pathways in which their brains do not function normally

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16
Q

according to the neural explanation of OCD what do those with OCD fail to suppress

A

worry signals cannot be suppressed from the caudate nucleus causing them to continue worrying

17
Q

according to the neural explanation of OCD what does the lack of suppression of worry signals create within the person with OCD

A

it creates obsession as they are continually worried meaning they must perform compulsions to cope

18
Q

what does the diathesis stress model suggest

A

diathesis stress model suggest that we are genetically predisposed (inc chance of developing) to attain a behaviour however an ENVIRONMENTAL stimuli will cause the actual onset of the disorder

19
Q

what does the diathesis stress model suggest about genetics

A

genetic increase our chance of attaining a disorder but something causes the actual onset (environmental)

20
Q

strength of the biological explanation of OCD

A

P: There is supporting evidence for the role of serotonin in OCD
E: comes from research examining antidepressants (SSRIs) such as that conducted by Piggott et al. (1990)
E: he found that drugs which increase the level of serotonin in the synaptic gap are effective in treating patients with OCD.
L: This therefore increases reliability of the study

21
Q

weakness of the biological explanation of OCD

A

P: A diathesis-stress model may be better suited to explaining OCD
E: as it factors in both genetics and psychological factors such as environmental stressors.
E: This suggests some people may have a genetic vulnerability to develop OCD, providing the right environmental triggers cause its onset.
L:This would be a more appropriate explanation as it explains why identical twins may not share the disorder.