B7.062 Stroke Syndromes Flashcards

1
Q

symptoms of left internal carotid occlusion

A
similar to MCA but with additional ophthalmic involvement
abulia
contralateral homonymous hemianopia
contralateral hemiplegia
contralateral hemisensory disturbance
gaze preference to side of lesion
aphasia if dominant
neglect if non dominant
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2
Q

abulia

A

absence of willpower

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3
Q

neglect

A

patient does not pay attention to half of their body

due to damage in non-dominant hemisphere

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4
Q

course of ophthalmic artery

A

branches from ICA, below anterior clinoid process
supplies globe, orbit, frontal scalp, and fontal and ethmoidal sinuses
branches anastomose with maxillary artery branches (potential for collateral flow)

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5
Q

what does the ophthalmic artery perfuse

A

optic nerve and retina

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6
Q

what is recurrent transient monocular blindness

A

occurs with infarction of ophthalmic artery
manifests in 25% of patients with symptomatic internal carotid disease
transient ischemic attack

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7
Q

what does the MCA supply

A

most of the temporal lobe
anterolateral frontal lobe
parietal lobe

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8
Q

what do the perforating branches of the MCA supply

A

posterior limb of the internal capsule
part of the head and body of the caudate
globus pallidus

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9
Q

L MCA stem occlusion symptoms (proximal to penetrators)

A

contralateral hemiplegia affecting face, arm, leg
contralateral homonymous hemianopia
ipsilateral head/eye deviation
global aphasia, alexia, agraphia, acalculia, apraxia

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10
Q

R MCA stem occlusion symptoms (proximal to penetrators)

A

contralateral hemiplegia affecting face, arm, leg
contralateral homonymous hemianopia
ipsilateral head/eye deviation
neglect, agnosia, apraxia, aprosody

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11
Q

prosody

A

expressivity in voice

aprosody = lesion on non-dominant hemisphere

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12
Q

lenticulostriate arteries

A

branch of M1 segment of MCA
supplies basal ganglia structures: part of head and body of caudate, globus pallidus, putamen, and the posterior limb of the internal capsule

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13
Q

lenticulostriate lesion symptoms

A

damage to internal capsule: contralateral hemiparesis and sensory deficit

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14
Q

what is a lacunar syndrome

A

occlusion of deep branches of the MCA or basilar artery

preservation of cortical function (language, cognitive, visual)

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15
Q

location and appearance of lacunar infarcts

A

80% : periventricular white matter and basal ganglia
20%: cerebellum and brain stem
0.5-1.5 cm in diameter

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16
Q

symptoms of inferior MCA division infarct

A

superior quadrantanopia / homonymous hemianopia
Wernicke aphasia (deficit in comprehension if dominant hemisphere)
Left sides neglect ( if non dominant hemisphere)
cortical sensory loss

17
Q

symptoms of superior MCA division infarct

A
brachiofacial paralysis
sensorimotor deficit involving face and arm, leg to a lesser extent
foot is spared
ipsilateral deviation of head/eyes
non-fluent aphasia
18
Q

course of ACA

A

supplies most of the medial surface of the cerebral cortex (anterior 3/4), frontal pole, and anterior portions of the corpus callosum

19
Q

unilateral ACA occlusion

A

contralateral sensorimotor deficits mainly involving the lower extremity with sparing of face and hands

20
Q

bilateral ACA occlusion

A

infarction of the anteromedial surface of the cerebral hemispheres

  • paraplegia affecting LEs and sparing face/hands
  • incontinence
  • abulic and motor aphasia
  • frontal lobe symptoms (personality chance, contralateral grasp reflex)
21
Q

what are the PCAs

A

terminal branches of the basilar artery

22
Q

PCA course

A

small perforating branches supply midbrain structures, choroid plexus, and posterior thalamus
cortical branches supply the undersurface of the temporal lobe and occipital and visual cortex

23
Q

2 syndromes associated with PCA occlusion

A

P1: midbrain, subthalamic, and thalamic signs
P2: cortical temporal and occipital lobe signs

24
Q

symptoms of basilar artery occlusion

A

bilateral long tract symptoms
bilateral CN abnormalities
coma

25
Q

artery that supplies the lateral medulla

A

PICA