Assessment HISTOMTLE Flashcards
This type of epithelium lines most of the respiratory tract:
Simple squamous epithelium
Simple cuboidal epithelium
Simple columnar epithelium
Pseudostratified ciliated columnar epithelium
Pseudostratified ciliated columnar epithelium
Thyroid follicles are lined by this type of epithelium:
Simple squamous epithelium
Simple cuboidal epithelium
Simple columnar epithelium
Transitional epithelium
Simple cuboidal epithelium
It forms the linings of the urinary bladder, the ureters and part of the urethra (organs which are subject to considerable stretching):
Stratified squamous epithelium
Transitional epithelium
Stratified cuboidal epithelium
Stratified columnar epithelium
Transitional epithelium
Long, cylindrical and multinucleate, they have obvious striations, and can be controlled voluntarily:
Skeletal muscle
Visceral muscle
Cardiac muscle
Skeletal muscle
Uninucleate branching cells that fight tightly together at junctions called intercalated disks:
Skeletal muscle
Visceral muscle
Cardiac muscle
Cardiac muscle
With single nucleus and are spindle-shaped, no striations are visible:
Skeletal muscle
Visceral muscle
Cardiac muscle
Visceral muscle
What is the stem cell precursor of most connective tissues?
Macrophage
Mesenchymal
Adipocytes
Plasma cells
Mesenchymal
Elastic cartilage EXCEPT:
Intervertebral discs
External ear
Walls of the Eustachian tubes
Epiglottis
Intervertebral discs
Intervertebral discs: FIBROCARTILAGE
Which one of the listed statements is the best histologic definition of an
abscess?
A circumscribed collection of neutrophils with necrotic cellular debris
A localized defect that results from the sloughing of necrotic inflammatory tissue
from the surface of an organ
A localized proliferation of fibroblasts and small blood vessels
An aggregate of two or more activated macrophages
The excessive secretion of mucus from a mucosal surface
a. A circumscribed collection of neutrophils with necrotic cellular debris
An abscess is a localized collection of neutrophils and necrotic debris. It is basically a localized form of suppurative (purulent) inflammation, which is associated with pyogenic bacteria and is characterized by edema fluid admixed with neutrophils and necrotic cells (liquefactive necrosis or pus).
Cells provide a structural framework for the skin and play a critical role in
WOUND HEALING:
Basophils
Fibroblasts
Lymphocytes
Monocytes and macrophages
b. Fibroblasts
The cardinal sign of inflammation called rubor is mainly the result of:
Decreased interstitial hydrostatic pressure
Decreased vascular permeability of capillaries
Increased vascular permeability of venules
Vasoconstriction of muscular arteries
Vasodilation of arterioles
e. Vasodilation of arterioles
Inflammation can be defined as the reaction of vascularized living tissue to local injury.
Celsus originally described four cardinal signs of inflammation: rubor (redness), tumor
(swelling), calor (heat), and dolor (pain). Virchow later added a fifth sign, loss of function
(functio laesa).
Redness (rubor) and heat (calor) are primarily the result of increased blood flow secondary
to vasodilation of arterioles.
During acute inflammation, histamine-induced increased vascular
permeability causes the formation of exudates (inflammatory
edema).Which one of the listed cell types is the most likely source of the
histamine that causes the increased vascular permeability?
Endothelial cells
Fibroblast
Lymphocytes
Mast cells
Neutrophils
Mast cells
Inflammation characterized by the presence of large amount of pus:
Serous
Fibrinous
Hemorrhagic
Suppurative or purulent
Suppurative or purulent
Inflammation characterized by extensive outpouring of a watery, low-protein fluid from blood:
Serous
Fibrinous
Hemorrhagic
Suppurative or purulent
Serous
It is usually observed in skeletal muscles, heart, kidneys, endocrine organs and smooth muscles of hollow viscera due to increased workload and endocrine stimulation (e.g. during exercise and pregnancy)
True hypertrophy
False hypertrophy
Compensatory hypertrophy
None of these
True hypertrophy
It is due to edema fluid and connective tissue proliferation (e.g. in cirrhosis and chronic hypertrophic salphingitis or appendicitis):
True hypertrophy
False hypertrophy
Compensatory hypertrophy
None of these
False hypertrophy
An increase in size of tissues or organs due to increase in size of the individual cells:
Anaplasia
Hypoplasia
Hyperplasia
Hypertrophy
Hypertrophy
A reversible change involving the transformation of one type of cell to
another:
Anaplasia
Dysplasia
Metaplasia
Neoplasia
Metaplasia
Microscopically, the hallmark of this type necrosis is the conversion of normal cells into ‘TOMBSTONES’ :
Caseous necrosis
Coagulation necrosis
Fat necrosis
Liquefaction necrosis
Coagulation necrosis
Outlines of the cells are retained so that the cell type can still be recognized but their cytoplasmic and nuclear details are lost.
A 49-year-old man develops an acute myocardial infarction because of the sudden occlusion of the left anterior descending coronary artery. The areas of myocardial necrosis within the ventricle can best be described as:
Coagulative necrosis
Liquefactive necrosis
Fat necrosis
Caseous necrosis
Fibrinoid necrosis
Coagulative necrosis
A special form of cell death produced by the tubercle bacillus:
Liquefaction necrosis
Coagulation necrosis
Fat necrosis
Caseous necrosis
Caseous necrosis
Organ most commonly affected by FATTY DEGENERATION:
Heart
Liver
Lungs
Kidney
Liver
Cytological picture suggestive but not conclusive of malignancy:
Class II
Class III
Class IV
Class V
Class III
Cytological picture strongly suggestive of malignancy:
Class II
Class III
Class IV
Class V
Class IV