Arrhythmias Flashcards

1
Q

what are the 2 locations for arrhythmias

A

supra ventricular: SAN, atria, AVN or his

ventricular: ventricular muscle

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
2
Q

name 3 supraventricular tachycardic arrhythmias…

A

A.fib, atrial flutter, ectopic atrial tachycardia (EAT)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
3
Q

name 2 supraventricular bradycardic arrhythmias…

A

sinus bradycardia, sinus pauses

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
4
Q

name 3 supraventricular AVN arrhythmias…

A

AVN re-entry tachycardia (AVNRT), AV re-entrant tachycardia (AVRT), AV block

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
5
Q

what are 4 ventricular arrhythmias…

A

ventricular ectopics/ premature ventricular complexes (PVC), ventricular tachycardia, ventricular fibrillation, asystole

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
6
Q

what causes arrhythmias

A

anatomical: LV hypertrophy, accessory pathways, congenital heart disease
ANS: sympathetic stimulation e.g. stress
metabolic: hypoxia, ischaemic myocardium, electrolyte imbalance
inflammation: viral myocarditis
genetic: e.g. LQTS

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
7
Q

what are the 5 pathological mechanisms that cause arrhythmias

A
automaticity 
ectopic beats 
triggered activity 
tract pathway 
re-entry
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
8
Q

what does automaticity consist of

A

occurs if latent pacemaker takes over SAN usually due to tissue damage e.g. hypoxia

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
9
Q

what type of pathological mechanism for arrhythmia is beats that originate in places other than SAN?

A

Ectopic beats

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
10
Q

“normal AP triggers abnormal oscillations in membrane potentials that reach threshold triggering a sustained train of premature APs” is…

A

Triggered activity

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
11
Q

what are Tract pathways?

A

alternate electrical pathways to AVN that are faster than normal conduction

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
12
Q

what type of arrhythmia do tract pathways usually cause

A

tachyarrhythmias

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
13
Q

what is re-entry?

A

more than 1 conduction pathway which have different speeds

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
14
Q

what can cause re-entry mechanisms

A

structural: MI scar, CHD
Functional: conditions that depress conduction velocity or prolong refractory period e.g. WPW syndrome

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
15
Q

in what type of syndrome does pre-excitation (accessory pathway causes AP during repolarisation of heart) occur?

A

Wolf Parkinson White Syndrome

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
16
Q

what 2 arrhythmias are life-threatening

A

Vtac & A.Fib

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
17
Q

what are general presenting symptoms of arrhythmias?

A

asymptomatic, palpitations, SOB, dizziness, pre-syncope, syncope, tachy/brady, SCD, angina

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
18
Q

what ix for arrhythmias?

A

12 lead ECG, exercise ECG, 24hr ECG

echo

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
19
Q

sinus bradycarda…

  • how many bpm
  • aetiology
  • tx
A

<60bpm
physiological, drugs, ischameia
tx: acute- atropine, cardiac pacing if haemodynamically compromised

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
20
Q

what does haemodynamically compromised mean?

A

hypotension etc

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
21
Q

sinus tacycardia…

  • bpm
  • aetiology
  • tx
A

> 100bpm
physiological, drugs
tx: underlying cause, BBs

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
22
Q

what findings may you find on the ECG of someone with supraventrcular ectopic beats?

A

narrow QRS complex

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
23
Q

what is the pathological mechanism responsible for supraventricular tachycardias

A

re-entry, ectopic beats

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
24
Q

what is the tx for supraventricular tachycardias ?

A

acute: inc vagal tone (valsalva monouvre, carotid massage), slow conduction to AVN (IV adenosine, verapamil)
chronic: BBs, anti-arrhythmic drugs. avoid stimulants

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
25
what is heart block
AVN conduction disease
26
causes of heart block?
ageing, acute MI, myocarditis, drugs, CHD, post aortic valve surgery
27
how many degrees of heart block are there?
3
28
what does 1st degree heart block mean?
no block, no tx
29
what are ECG signs of 1st degree heart block
PR interval is long (>0.2secs)
30
what does 2nd degree heart block mean?
intermittent conduction block at AVN
31
what are the 2 types of 2nd degree heart block?
Mobitz 1 & Mobitz 2
32
True/False... | Mobitz 1 shows a fixed PR interval then a dropped beat on ECG
False... Mobitz 1 shows progressive lengthening of PR interval then a dropped beat Mobitz 2 shows a fixed PR interval then a dropped beat
33
what does 3rd degree heart block mean?
no AP get through to AVN ie. complete heart block
34
what signs are there on ECG that show 3rd degree heart block?
no correlation between P wave & QRS complex
35
no tx is given for 1st degree heart block, so for what types of heart block is tx given and what is it?
2nd Degree Heart Block Mobitz 2 & 3rd degree Heart Block | Tx= ventricular pacing
36
what are the causes of ventricular ectopic or premature ventricular complex?
structural (LV Hypertrophy, HF), metabolic (Ischemia, CHD)
37
tx for ventricular ectopic or premature ventricular complex?
BBs, ablation of focus
38
what are causes for ventricular tachycardia?
pre-existing HD e.g. prev MI, cardiomyopathy
39
what are the 4Ts & 4Hs of reversible ventricular tachycardia?
``` 4Ts= Thrombosis Tension Pneumo Tamponade Toxins ``` ``` 4Hs Hypothermia Hypoxia Hypovolaemia Hyper/Hypokalameia ```
40
what arrhythmia would a finding of a wide QRS complex (>0.12) be?
Ventricular Tachycardia
41
what is Tx for acute ventricular tachycardia
unstable: direct current cardioversion (DCC) stable: AAD, amiodarone
42
what tx is given for torsades de pointes?
IV Magnesium
43
what is Tx for chronic ventricular tachycardia?
treat underlying, Congestive HF therapy e.g. ACEI, anti-arrhythmic drugs, ICD (implantable cardioverter defibrillator), VT catheter ablation
44
what are the classes of ventricular tachycardia
monomorphic (1 circuit), | polymorphic (torsades de pointes)
45
what is V.Fib?
chaotic ventricular activity causing heart to lose its pumping function
46
what are 2 key sings of v.fib
pulseless, unconscious
47
what is tx for v.fib
defibrillate & CPR
48
what is asystole?
absence of ventricular contractions
49
what are pacemakers usually used for
AVN disease
50
when is catheter ablation usually used for
re-entry disease e.g. PVC or A.fib
51
what is the most common arrhythmia?
Atrial fibrillation
52
what are 3 classes of A.Fib
paroxysmal (<48hrs, recurrent), persistent (>48hrs, cardioverted back to NSR) permanent (inability to restore to NSR)
53
what is a.fib?
chaotic atrial activity- irregular supra ventricular tachycardia
54
where does a.fib ectopic foci originate?
ostia of pulmonary vein
55
what are etiological diseases of a.fib?
HT, HF, Coronary HD, thyroid disease, valvular, congenital HD, cardiac surgery, COPD, septicaemia, pericarditis
56
what is a common sign of a.fib
irregular pulse
57
what does ECG show if a.fib present?
>300bpm, irregular rhythm, ventricular rate is variable, absence of p waves but f waves may be present
58
what is the 4 step tx for A.fib
Terminate AF through cardioversion Control Rate Control Rhythm + Anticoagulants
59
what are the 3 ways in which AF can terminate?
1. drugs 2. direct current cardioversion (DCCV) 3. spontaneous reversion to sinus rhythm
60
how do you control rate and hence slow down AVN conduction through Rx?
digoxin, b-blockers, verapamil
61
how does digoxin work
inc force of contraction of heart by inhibiting ATPase hence inc Ca++ movement into heart
62
how do you gain control of the rhythm?
restore NSR via amiodarone or DCCV & | maintain NSR via amiodarone, catheter ablation of focus
63
why MUST anticoagulants be given after A.fib?
A.fib poses an inc risk of thromboembolisms forming
64
what is the biggest complication of A.fib?
PE and stroke
65
what is the definition of congestive heart failure?
heart failure due to lost atrial kick and slow filling time
66
what is atrial flutter?
rapid, REGULAR, atrial tachycardia
67
True/False... | atrial flutters usually progress to AF?
True!
68
what is a typical ECG of someone with an atrial flutter?
regular rate with saw tooth F wave
69
tx for atrial flutter?
radio frequency ablation, BBs, cardioversion, anticoags to prevent thromboembolism
70
what is cardiac arrest?
cessation of heart
71
how to recognise someone in cardiac arrest?
unresponsive, no pulse, no breathing
72
what are causes of cardiac arrest- think ABCDE...
A: CNS depression, blocked airway, swelling, muscle spasm B: dec res drive, lung disorders (e.g. haemothroax) C: 1y- ACS, arrhythmias, drugs, valvular HD. 2y- asphyxia, hypoxia, hypovolaemia, sepsis D: hypoglycaemia E: trauma
73
how to assess A of ABCDE?
talking?, SOB?
74
how to assess B of ABCDE?
look, listen, feel - chest expansion
75
how to assess C of ABCDE?
general exam, BP, pulse, perfusion, bleeding
76
how to assess D of ABCDE?
AVPU or GCS + Pupils
77
tx for cardiac arrest
start CPR 30:2 shock if rhythm is VF/pulseless VT every 2 mins administer adrenaline/amiodarone after 3rd shock and thereafter every 3-5mins
78
when should you not shock?
asystole or PEA (pulseless electrical activity)
79
True/False... | Cardioversion & Defibrillation are the same thing
True & False... defibrillation is only applied to emergencies and only to VT and pulseless V.Fib Cardioversion given only if pt has a pulse but is unstable or chemical cardioversion has failed however- same in sense that both shock heart to try return arrhythmia back to NSR