AOTA-Peds Conditions Flashcards

1
Q

Congenital heart disease OT treatment

A

Surgical, activity pacing, education on diet exercise, avoidance of smoking inhalation

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2
Q

Brochopulmonary dysplasia

A

a result of the prolonged use of mechanical ventilation and other traumatic interventions to treat acute respiratory problems.

It results in** thickening of the airway, formation of excess mucus, and restricted growth.**

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3
Q

Asthma

A

characterized by bronchial smooth muscle hyperreactivity that causes airway constriction in the lower respiratory tract, difficulty breathing, and wheezing

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4
Q

Cystic fibrosis (CF)

A

an inherited disorder that causes severe damage to the lungs, digestive system and other organs in the body.

Cystic fibrosis affects the cells that produce mucus, sweat and digestive juices. These secreted fluids are normally thin and slippery. But in people with CF, a defective gene causes the secretions to become sticky and thick. Instead of acting as lubricants, the secretions plug up tubes, ducts and passageways, especially in the lungs and pancreas.

Genetic (inherited) disease that causes sticky, thick mucus to build up in organs.
inherited autosomal recessive disorder, related to chromosome 7

1.Failure to grow properly
2. mucus causes lung infection (d/t clogs)/inability for pancreas to produce enzymes required to break down food.
3. 10-20% of children with CF presents with intestinal blockage
4. Effects on function: exercise intolerance, poor nutrition

complications: reduce life expectancy (30s-40s, some 50s) CF, diabetes, sirroosis, and rectal prolapse rar of complication sof CF

Chronic, progressive lung disease characterized by abnormal mucus produc

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5
Q

Cystic fibrosis (CF) treatment

A

Educate the client about the disease’s progression.
Instruct the client and his or her family in energy conservation.
Teach techniques to promote efficient breathing
.

Med: chest therapy, aerisol, antibiotics, vitamins

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6
Q

Chronic pulmonary disease

A

characterized by a chronic cough, wheezing, and lower respiratory infections.

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7
Q

Erythrocytes

A

characterized by too many red blood cells and elevated levels of white blood cells; it is referred to as transient leukemia occurs

1 in 150 with down syndrome

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8
Q

Hemophilia

A

blood disorder (mostly in males); characterized by the absence or reduction of one of the clotting blood proteins.; longer bleed time; excessive bleeding/bruising; random bleeding with cause; nosebleed

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9
Q

Anemia

A

iron deficiency in blood; symptomatic of other conditions (lead poisoning, vit. def. leukemia, or sickle cell)

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10
Q

Sickle Cell anemia/OT treatment

A

a form of anemia caused by abnormally shaped red blood cells.; mostly in African-Americans

-may demostrate low energy for daily tasks; risk for organ damaage resulting from blocked blood flow; pain

OT treatment: pain management

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11
Q

osteogenesis imperfecta/ OT treatment

A

inherited condition of deformed and abnormally brittle bones; minor trauma can cause fx; decreased bone deposition caused by TYPE I collagen

Educate parents on handling/positioning to prevent fx; monitor activities that promote WB encouraged

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12
Q

Marfan syndrome (arachnodactyly)

A

caused by an autosomal dominant trait.
It is characterized by** excessive growth at the epiphyseal plates**.

-Presents with long and slender fingers, skull asymmetries, and tall stature. They may also have differences in their joints, eyes, and heart; lax and hypermobile joints; poor dev. straited muscles

-Delayed walking/dev milestones

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13
Q

Achondroplasia

A
  1. caused by autosomal dominant trait that results in the ephiphyseal plate growth and cartilage formation
  2. 4ft or less in height; limbs have typical width, but shorter in length; prominent forehead and a small nonse and jaw; trunk is typical
  3. Comorbitities (lumbar lordosis, coxa vara, and cubitus varas
  4. Back and leg pain are common

chondrodystrophia/dwarfish

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14
Q

anchodroplasia

A

dwarfism; 4” or less; prominent forehead and a small nose/jaw; typical trunk

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15
Q

arthrogryplasia OT treatment

Arthrogryposis multiplex congenital

A

associated with reduced anterior horn cells in the spinal cord;
-incomplete contracturs of many or all joints (at birth); UE/LE involved, stiff and spindly extremetis, thicken knee/elbow; underdev. muscles/paralysis

OT: increase/maintain ROM/strength by stretching/spliting and serial casting; increase ADL/IADLs, edu, and play through AD.

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16
Q

congential clubfoot &treatment

A

clinical features include unilateral and bilateral foregoot adduction and supination, heel varus

-caused by autosomal dominant gene; bony malformations and under develeoped LE musculature

_ can be corrected if treated early in childhood with taping, casting, splinting, and ortho surg.

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17
Q

congential club hand & treatment

A

-Clinical features include partial or full absence of the radius and bowing of the ulnar shaft with absence or underdevelopment of the upper extremity nerves and musculature.

The person’s hand often remains functional. However, progressive casting, static or dynamic splinting, and surgery may be used for cosmetic reasons.

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18
Q

Dysplasia dev in hip & treatment

A

genetic and environmental; d/t hormal secretions in utero/increased uterine pressure/poor positioning/dislocation in utero; occurs bilaterally; more common in girls

-early diagnosis is critical/ can develop Trendelenburg’s sign if left untreated

-treatment-bracing, casting, or splinting within the first few weeks of life, children typically have good developmental outcomes; if not treated early- may need ortho surg. THR.

19
Q

amelia

A

absent of a limb or distal segment of a limb

20
Q

juvenile rheumatoid arthritis & treatment

A

_joint inflammation, joint stiffness, joint contractures, and change in growth patterns. Weakness around the involved joints

OT treatment: splinting PRN, AROM/PROM; monitor joint function/deformity; educate in EC and AE to put less stress on joints

21
Q

lordosis & OT treatment

A

abnormal anterior curvature of the lumbar spine (sway-back condition)/anterior pelvic tilt

OT treatment: stretching tight hip flexors, strengthening abs; postural training; back bracing

22
Q

kyphosis + treatment

A

excessive outward curvature of the spine, causing hunching of the back; convexity; upper back

-faulty posture d/t skeletal growth outpacing muscular growth

OT treatment: postural training, strengthening, Milwaukee brace; anterial spinal release/postieral spinal fusion in severe cases.

“Round back”- Scheurmann’s disease

23
Q

scoliosis + treatment

A

abnormal lateral curvature of the spine; most serious; poor postural tone, hip contracturs, leg length discrepancy, or pain; curve of 65-80 deg. may result in reduce cardiopulmonary function

bracing (boston brace/thoracolumbosacral orthtic;

OT treatment: postoperative (spinal fusing) strengthening of ab muscles/ ADL adaption

24
Q

cerebral palsy & OT treatment

A

nonprogressive condition that encompasses neurological, motor, and postural deficits; Common comorbidities include language, cognitive, sensory, and psychosocial deficits; seizure disorders and feeding impairments are common;** impaired tone, motor planning, motor control, and coordination**

Treatment: Maintain AROM and PROM through stretching, exercise, and orthotics; use of AE and assistive tech.; positioning/seating; constraint-induced therapy

25
Q

congenitial CP cause

A

increased sensitivity of the CNS before full term; CVA at or around time of birth d/t placemental abruption, fetomaternal hemorrhage, placental infarction, materal exposure to env. toxins

26
Q

acquired CP cause

A

result from trauma, intracranial hemorrhage, CNS infections, near drowning, hypoxia, and metabolic disorders.

27
Q

seizure disorder+treatment

A

Chronic neurological conditions that consist of reoccuring , whether or not the person has other brain abnomalities

Causes: hypoglycemia, fever, trauma, bleeds, tumors, infections, and lack of oxygen

Treatment: antiepileptic medications, hemispherectomy(if progressive), electrical stimulation of the vagus n, or thalamus

28
Q

What does a postive Gower sign indicate?

A

Duchenne’s Muscular dystrophy

A positive test is noted when there is a lack of quads/gluts m in which a person compensates by using their hands to “walk” themselves upright rather than relying on their legs.

29
Q

Duchenne’s muscular dystrophy & OT treatment

A

**most common in boys; typically dev. after birth begins symp. between 2-6 y/o; caused by deficiency in the production of______ ; muscles degnereate without _______.

progressive
-requires w/c by age 9
- death near 20s as result of respiratory problems or cardiovascular complications.

Difficulty going up and down stairs, getting up from lying-dwn position. diffculties with ADLs

OT treatment: maximize/prolong independence; prevent deformities, strength/ROM, psychosocial/vocational support, AE PRN

30
Q

Congenital muscular dystrophy (CMD) & OT treatment

A

-heterogeneous group of disorders w/ onset in utero or 1st yr.
-hypotonia, generalized muscle weakness, contractures.
-comorbidities (clubfoot, torticollis, diaphragmatic involve., congenital heart and spinal defects.)
-floppy-low tone in face, neck, trunk, and limbs, decrease muscle mass and absent deep tendon reflexes.

OT treatment: Increase mobility and prevent contractures.
Use adaptive equipment and orthotics.
Maintain independent mobility.

31
Q

Neural Tube Defects (3 major form)

A

occur early in fetal development; prevention =importance of folic acid for women in child bearing years.

  1. encephalocele
  2. anecephaly
  3. Spina bifida -most common (myelomeningocele=most severe of SB)
32
Q

encephalocele

A

a protrusion in the occipital region of the brain; severe deficits, such as cognitive impairments, hydrocephalus, motor impairments, and seizures

33
Q

anencephaly

A

Neural development above the level of the brain stem is lacking (Most do not survive infancy)

34
Q

spina bifida; meningocele spina bifida

A

_congenital defect of the vertebral arches and spinal column; mild form=involves no sypm where 1-2 areonly affected;

___________involves an extensive spinal opening with an exposed pouch of cerebrospinal fluid and meninges; nerve root are also exposed

-displayssensoryimotor problems at or below the level of the lesion
-LE paralysis/loss of senseation
-some also have hip, spinal or foot deformities
complications include hydrocephalus and Arnold- Chari syndrome

35
Q

meningocele spina bifida OT treatment/

A

-bowel and bladder management
-cog/learning issues to be addressed
-educate family/client on** skin care, urology, and diet**
-AD should be used for mobility

36
Q

brachial plexus

A

peripheral n injury resulted from birth, affecting nerves C5-C8 AND T1

37
Q

Erb-Duchenne palsy

A

caused by an injury to the upper brachial plexus (the C5 and C6 nerve fibers) such as** extreme shoulder flexion (with arm overhead**; common with breech deliveries;

** weakness or wasting of small muscles of HANDS** and
sensory discrimation of HAND ARMS; typically unilateral= paralysis of UE, shoulder musculature affected than the hand

presentation: hold affected arms in a charactertic posutre;** shoulder adduction and internal rotation, elbow extension, forearm pronation, wrist flexion** (waiter’s tip position)

-recovery depends on extent of nerve damange- occurs within 3- 24 months

38
Q

Klumpke’s palsy

A

results of compression or traction of the lower brachial plexus (C8 and T1; less common)

  • result of mechanical damage to the nerves which causes paralysis of the forearm and hand muscles
39
Q

peripheral nerve injuries -OT assessment and intervention

A

assessment:
observation of occupations (feeding, bathing, dressing); MMT, if able AROM exercises

intervention:
-sling to prevent subluxation/spint-rest
-PROM/AROM
-resistive ex./WB when stregnth improves
-tactile stimulation- texture, massage, vibration)
-bilateral activties to increase body scheme (patty cake, peek-a-boo)
-retrograde massage for edema
-surg. if no improvements between 3-6 months

40
Q

meningocele spina bifida
-self-care maintenance (5-9 y/o)

A

-pressure checks
-educate on injury/illness/changes in body and bowel/bladder habits
-keep track of crutches/w/c with reminders
-perform self-catheterization at school
-carry list of doctor# for emergencies

41
Q

meningocele spina bifida
-self-care maintenance (10-14 y/o)

A

-begins to recognize when feeling poorly has to do with condition
-name doc/med/doses
-keep track of AE and direct others to help; self-catheteriztion in community venues
-take resp. for health (drink water/avoid bladder infec)

42
Q

meningocele spina bifida
-self-care maintenance (15-18 y/o)

A

-take meds/understand side effects
-know how to contact doc/therapists
-know how to get and pay for med supplies/equipment
-regular skin checks
-manage weight
maintain equip

43
Q

Symptoms of Cystic Fibrosis

A

symptoms:

Failure to thrive (inability to gain weight despite having a good appetite and taking in enough calories).
Loose or oily stools.
Trouble breathing.
Recurrent wheezing.
Frequent lung infections (recurrent pneumonia or bronchitis).
Recurrent sinus infections.
A nagging cough.
Slow growth.
Atypical cystic fibrosis

People with atypical cystic fibrosis may be adults by the time they’re diagnosed with atypical CF. Respiratory signs and symptoms may include:

Chronic sinusitis.
Breathing problems, possibly diagnosed as asthma or chronic obstructive pulmonary disease (COPD).
Nasal polyps.
Frequent bouts of pneumonia.
Other signs and symptoms of atypical CF may include:

Dehydration or heat stroke that reveals abnormal electrolyte levels.
Fertility problems.
Diarrhea.
Pancreatitis.
Unintended weight loss.