AI Flashcards 2 Summary

1
Q

What are the three models for defining abnormal behaviour?

A

Cultural/social/ethical, statistical, and medical. The cultural model looks at differences in norms, the statistical model uses a bell curve, and the medical model, specifically the DSM, treats disorders as discrete.

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2
Q

What is comorbidity?

A

Comorbidity is having multiple psychological/psychiatric disorders at the same time, which is common, particularly with trauma.

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3
Q

What is Conduct Disorder?

A

A psychiatric disorder, usually emerging in childhood or adolescence, characterized by severe antisocial and aggressive behavior. It frequently co-occurs with ADHD, and often leads to antisocial personality disorder in adulthood.

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4
Q

List 4 symptoms of Conduct Disorder

A
  • Aggression to people or animals
  • Destruction of property
  • Deceitfulness or theft
  • Serious violation of rules
  • Other symptoms include lack of remorse or guilt, lack of empathy, and unconcern about performance.
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5
Q

What are the two factors of Psychopathy?

A

Factor 1: Interpersonal/affective (including glibness, lack of remorse, and manipulativeness), and Factor 2: Chronic antisocial lifestyle (including impulsivity and criminal versatility).

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6
Q

How do neurodevelopmental disorders affect the brain?

A

They affect the brain’s structure, function, and connectivity, leading to problems in behaviour, cognition, and emotion. They are driven by genetic and environmental interactions.

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7
Q

Give three examples of environmental factors that can interact with genetic predispositions in neurodevelopmental disorders.

A
  • In the uterus: maternal smoking, stress, drugs and alcohol abuse.
  • During birth: birth complications, parental psychopathology.
  • During childhood/adolescence: harsh discipline, low socioeconomic status, negative peer influences.
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8
Q

What are the three types of gene-environment correlations?

A

Passive (inherited predispositions contribute to the environment), active (child seeks out environments that trigger their genes), and evocative (inherited predispositions evoke a negative response from the environment).

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9
Q

How do genes influence behaviour?

A

Genes influence neuronal formation, which leads to neuronal organization and ultimately impacts behaviour. Disruptions can cause system dysfunctions.

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10
Q

What is the Amygdala-Centered Model of antisociality?

A

This model proposes that in severe antisocial individuals, the amygdala is hyporesponsive to negative stimuli, leading to deficient affective processing and poor associative learning, which promotes persistent antisocial behaviour.

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11
Q

What is the Paralimbic System Dysfunction Model of antisociality?

A

This model expands on the amygdala model, suggesting that abnormalities are distributed across multiple brain regions involved in emotional and social processing, possibly as a compensatory mechanism for intrinsic deficits.

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12
Q

What is the role of the amygdala?

A

The amygdala acts as a salience detector, receiving information from various brain regions and prioritizing stimuli based on their importance.

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13
Q

What are the Basal Lateral Amygdala (BLA) and CentroMedial Amygdala (CMA)?

A

The BLA is associated with emotional processing and connectivity to the neocortex, and the CMA has been implicated in sexual and social behaviour. The BLA is larger in more cognitively advanced mammals.

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14
Q

What are some risk factors for antisocial behaviour?

A

Risk factors include domestic violence, poverty, negative peer influence, and gender-related factors. Protective factors include caring parents, education, and support systems. The strongest predictor is having parents or close relatives with a criminal history.

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15
Q

What is the biopsychosocial model?

A

This model considers biological, psychological, and social factors in understanding behaviour and mental health.

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16
Q

What are the main differences in development between the Social Emotional System and the Cognitive Control System in adolescence?

A

The Social Emotional System (amygdala) fully develops around age 16-20, making adolescents highly engaged in emotions, peer relationships, and social perception. The Cognitive Control System (prefrontal cortex) is still in development during adolescence, causing an imbalance that can lead to risk-taking behaviour.

17
Q

What are some deficits that are related to persistent aggressive behavior?

A

Deficits in emotion regulation, empathy, reward sensitivity, and executive functioning.

18
Q

What are some critical aspects of empathy?

A

Empathy can be myopic (short-sighted), selective (biased towards people who look like us), fleeting, and destructive (potentially leading to torture).

19
Q

What are the levels of the Batushka metaphor for empathy?

A
  • Emotional contagion/automatic contagion
  • Personal distress
  • Concern
  • Mentalizing (cognitive empathy)
  • Multifaceted integration
20
Q

How does testosterone affect aggression?

A

Testosterone facilitates aggression by making individuals more emotionally reactive, dominant, and reducing fear and empathic responses. It can bias the output of the amygdala.

21
Q

How is empathy used in Antisocial Personality Disorder (ASPD)?

A

Individuals with ASPD may show affective empathy, but its use is often selective, manipulative, and not automatic.

22
Q

What is the relationship between the amygdala and orbitofrontal cortex (OFC) in individuals with psychopathy?

A

There is reduced anatomical and functional coupling between the amygdala and OFC in individuals with psychopathy, potentially affecting behaviour.

23
Q

What is the importance of social cognition?

A

It is a unique human ability to understand each other as conscious beings with internal mental states, and it is crucial for social behaviour.

24
Q

What are the shared characteristics of incarcerated individuals with psychopathy?

A

Interpersonal manipulation, erratic lifestyle, callous affect, and criminal tendencies.

25
Q

What is the Approach-Avoidance Task (AAT)?

A

The AAT measures automatic action tendencies (approach positive, avoid negative) by using stimuli such as happy and angry faces and measuring reaction times to pulling or pushing a joystick.

26
Q

How do individuals with psychopathy respond in the AAT?

A

They show decreased avoidance in response to angry faces, potentially because they ascribe a high status to themselves.

27
Q

How might Oxytocin be used as an intervention?

A

Oxytocin facilitates prosocial behavior, reduces anxiety, and can decrease avoidance to angry faces.

28
Q

What is performance monitoring and what does it show in individuals with psychopathy?

A

Performance monitoring is how we continuously monitor our performance for errors, and it is essential for adaptive behaviour. Individuals with psychopathy show decreased neural activity when monitoring other’s performance, which may be related to problems in social learning.

29
Q

What are the ERN and Pe?

A

ERN (Error-related Negativity) is a quick alert in our brain that happens rapidly after we make a mistake, reflecting a fast error-detection process. Pe (Error Positivity) is a subsequent brain response reflecting a closer look at the mistake and thinking about it (more controlled/affective processing).

30
Q

What is the Diathesis-Stress Model?

A

This model explains the development of mental disorders through the interaction of inherent vulnerabilities (diathesis) and external stressors. The presence of diathesis increases the risk of disorder when combined with significant stress.

31
Q

What are the key differences between correlates and risk factors?

A

Correlates are factors that are linked to a specific outcome but are not direct causes. Risk factors are factors that come before and heighten the likelihood of a particular outcome.