ACLS Flashcards

1
Q

which rhythms are shockable

A

VF, pVT

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2
Q

which rhythms are not shockable

A

asystole, PEA

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3
Q

high quality CPR

A

rate 100-120 compressions/min, depth at least 2 inches, allow chest recoil, minimize interruptions

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4
Q

what are the ABCs

A

airway, breathing, circulation

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5
Q

which line is preferred

A

central line, meds reach the heart faster

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6
Q

intraosseous

A

used when IV access not available, treat as a central line

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7
Q

endotracheal

A

last line option for vascular access, absorption occurs in alveolar capillaries, doses often 2-2.5x higher than IV

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8
Q

administer _____ after each med administered

A

NS flush 10-20 mL

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9
Q

how does epinephrine work

A

increases coronary perfusion pressure

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10
Q

negative side effects of epinephrine

A

tachycardia, dysrhythmias, increased myocardial oxygen demand

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11
Q

IV/IO dose for epinephrine

A

1 mg q3-5 min

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12
Q

endotracheal dose for epinephrine

A

2-2.5 mg q 3-5 min

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13
Q

how does amiodarone work

A

antidysrhythmic properties through inhibition of sodium, potassium and calcium channels and alpha/beta adrenergic receptors

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14
Q

IV/IO dose for amiodarone administration

A

300 mg push once followed by 150 mg push

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15
Q

endotracheal dose for amiodarone

A

can’t be given via endotracheal tube.

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16
Q

pearl for amiodarone

A

only give IV push if patient is pulseless, IV push can cause hypotension and bradycardia in patients with a pulse

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17
Q

what is one alternative to amiodarone in ACLS algorithm

A

lidocaine

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18
Q

how does lidocaine work

A

antidysrhythmic properties through inhibition of Na channels

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19
Q

IV/IO dose for lidocaine

A

1-1.5 mg/kg once then 0.5-0.75 mg/kg if needed. may repeat for maximum total dose of 3 mg/kg

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20
Q

endotracheal dose for lidocaine

A

2-4 mg/kg once then 1-2 mg/kg if needed

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21
Q

what are the reversible causes

A

H’s and T’s

22
Q

H’s

A

hypovolemia, hypoxia, hydrogen ion (acidosis), hypo/hyperkalemia, hypothermia

23
Q

T’s

A

tension pneumothorax, tamponade (cardiac), toxins, thrombosis (pulmonary), thrombosis (cardiac)

24
Q

magnesium indication

A

cardiac arrest due to torsades or hypomagnesemia

25
Q

magnesium mechanism

A

inhibits calcium channels, suppressing early abnormal depolarizations responsible for arrhythmias

26
Q

magnesium dose

A

1-2 g diluted in 10 mL D5W or NS given over 5-20 minutes. rapid administration can cause hypotension in patients with a pulse

27
Q

sodium bicarbonate indication

A

acidosis or hyperkalemia

28
Q

mechanism of sodium bicarbonate

A

neutralizes acidosis; pushes K into cells via H/K exchange

29
Q

dose of sodium bicarbonate for acidosis

A

1 mEq/kg/dose IV/IO; repeat doses as needed guided by arterial blood gas

30
Q

dose of sodium bicarbonate for hyperkalemia

A

50 mEq once IVP/IO

31
Q

naloxone indication

A

suspected opioid overdose

32
Q

naloxone mechanism

A

opioid receptor antagonist

33
Q

naloxone dose IV/IM/SQ

A

0.4-2 mg

34
Q

naloxone dose intranasal

A

4-8 mg

35
Q

naloxone dose endotracheal tube

A

0.8-4 mg

36
Q

when should you give atropine for adult bradycardia

A

persistent bradyarrhythmia causing hypotension, acutely altered mental status, signs of shock, ischemic chest discomfort, acute heart failure

37
Q

what meds to give for adult bradycardia if atropine is ineffective

A

dopamine or epinephrine

38
Q

atropine mechanism

A

inhibits muscarinic acetylcholine receptors, increasing automaticity of SA & AV nodal cells

39
Q

atropine dose

A

0.5-1 mg q3-5 min, max total dose 3 mg

40
Q

dopamine mechanism

A

dopaminergic & beta-1 adrenergic receptor agonist

41
Q

dopamine dose

A

5-20 mcg/kg/min; titrate by 5 mcg/kg/min every 2 minutes

42
Q

epinephrine mechanism of action

A

beta1 receptor activation results in increased inotropy and chronotropy

43
Q

epinephrine dose

A

initial dose 2-10 mcg/min, max 40 mcg/min

44
Q

treatments for adult tachycardia

A

atropine (nonpharm: vagal maneuvers)

45
Q

what to do if persistent tachycardia causing: hypotension, acutely altered mental status, signs of shock, ischemic chest discomfort, acute heart failure

A

synchronized cardioversion, consider sedation; if regular narrow complex, consider adenosine

46
Q

if persistent tachycardia with no symptoms and wide QRS > 0.12 s

A

IV access & 12 lead ECG, consider adenosine only if regular & monomorphic, consider antiarrhythmic infusion

47
Q

if persistent tachycardia with no symptoms and no wide QRS

A

IV access & 12 lead ECG, vagal maneuvers, adenosine if regular, CCB or BB

48
Q

how do vagal maneuvers work

A

intended to stimulate vagal nerve, resulting in acetylcholine release and slowing of conduction through AV node

49
Q

examples of vagal maneuvers

A

bear down, blow through a syringe, immersion of face in ice water

50
Q

adenosine mechanism

A

slows conduction through AV node, 6 mg rapid IVP, can follow with 12 mg IVP if needed… MUST BE RAPID PUSH

51
Q

what to counsel patient before adenosine

A

may feel impending doom or feeling of dropping on a rollercoaster

52
Q

which line is preferred for adenosine

A

central line. if peripheral, raise extremity