Abnormal Psychology DSM 5 Flashcards
<p>Major Depression</p>
<p>2 wks.(to exclude normal lows)
5 symptoms -- must include depressed mood and loss of pleasure (universal symp)
- Core symptoms- mood-sadness, despair and emptiness, anhedonia, low self esteem, apathy-low motivation, excessive emotional sensitivity, neg. pessimistic thinking, irritability, Suicidal ideation
- appetite; loss, gain weight. decrease appetite
sleep early wake (reduce stage 3 and 4: slow wave sleep, decreased REM latency-earlier onset of REM and increase during of REM in early night)
Fatigue: expressed as tiredness, low energy; slowed speech and physical movement, pause before answering
Psychomotor retardation: quiet speech, stop talking completely unless directly questioned
agitation (become anxious) handwringing, pacing, inability to sit.
low self esteem, guilt
poor concentration (sometimes misdiagnosed with dementia
Suicidal thoughts
(other symptoms: crying spells, phobias, obsessions and compulsions, feeling hopeless, helpless worthless, Anx symp. panic attack: increase drinking, loss of reality-mood congruent (feelings of quilt-imagined he committed a sin) mood incongruent</p>
<p>Types of Depression</p>
<p>Reactive: Mild to moderate: occurs in response to an identified stressor, acute/intense, insidious - gradual (poor relationship) or past abuse.
Biological_ no stressor
medical or illness
female sex hormone- post partum
medication/drug
Bio genes: limbic
-sym appetite, fatigue, decrease sex, restlessness agitation, poor concentration</p>
<p>Depression
| Theories</p>
<p>Lewinsohn's behavioral theory of depression operant conditioning - low rate of response-contingent reinforcement for social and other behaviors. Reduced frequency of adjustive behavior-or behavior that maximizes reinforcement increases escape and avoidance behaviors in situations where positive reinforcement is attainable. thus failing to escape punishment
Seligman: Avoidance/escape of shock: those that couldn't became passive and accepted : learned helplessness: circumstances of reinforcement contingency-inescapable punishment Attribute it to internal, stable and global factors
Beck" negative cognitive biases-selective abstraction, over generalization negative self attributes. depressive cognitive triad: interpret their interactions with the environment as defeating, deprivation or disparagement. view self as deficient, inadequate or unworthy and attribute unpleasant experiences to physical, mental and moral defects in themselves and tend to reject themselves because of it. . View the future in a negative way and anticipate current sufferings will continue indefinitely</p>
<p>Etiology of Depression</p>
<p>genetic component: 1.5 to 3 times more common among first degree relatives; does not matter if one or two parents have the diagnosis.
link between depression and neurotic personality trait.
-catecholamine hypothesis- deficiency in norepinephrine.
-indolamine hypothesis: Low Serotonin (sensitive receptors)
-high cortisol (stress hormone) cause atrophy of neurons in hippocampus</p>
<p>Treatment for Depression</p>
<p>Tricyclics: "classic depression" veg body symptoms worsening symptoms on the AM, acute onset and short duration, symptoms of moderate severity.
-Amitriptyline/Elavil; Doxepin/ Adapin, Sinequan/Silenor ( most sedating; insomnia); Trimipramine/Surmontil; Nortriptyline, Pamelor/Aventyl; Desipramine/Norpramin; Protriptyline/Vivactil; Clomipramine/Anafranil (OCD too)
SSRI (moderate to severe) Fluoxetine/Prozac, Serafem (MDD, OCD, bulimia, PMDD, panic, BPD-depressive) Sertraline/ Zoloft (OCD, Panic, PTSD, PMDD, social anxiety); Paroxetime/ Paxil (GAD, OCD, PMDD, PTSD, pain &amp; Social anxiety)Fluvoxamine/Luvox (OCD, Social anxiety, MDD, Anx., PTSD, panic); Citalopram/Celexia (Depression; SE); Escitalopram/ Lexapro (MDD, GAD, SE: insomnia, Sex dys. nausea)
-Others: Trazodone/Desyrel, Nefazodone/Serzone, Vilazodone/Viibryd, Bupropion/Wellbutrin (smoking, soc. anx, )
(SNRI) Venlafaxine/Effexor (GAD, Soc. Anx., Panic)
Duloxetine/Cymbalta (MDD, GAD, chronic pain, fibromyalgia) Desvenlafaxine/Pristiq; Milnacipran/Remeron,
ECT: delusions/hallucinations; temp. anterograde, retrograde amnesia</p>
<p>Premenstrual Dysphoric Disorder</p>
<p>week before and symp improve few days after the onset of menses and absence or presence of minimal symptoms during the week post Menses. At least one symptom-affective lability, and one -loss of interest, concentration, appetite, insomnia/hypo. feeling out of control, or physical (breast swelling)</p>
<p>SSRI's</p>
<p>Fluoxetine (Prozac), Fluvoxamine, Paroxetine, Sertraline
Melancholic Dep, OCD, Bulimia, Panic, PTSD
Blocks reuptake of Serotonin
less cardiotoxic, safer overdose, less cognitive deficits, improve depressive symptoms,
Linked to suicide and violent act -Fluoxetine-
MAOI (monoamine oxidase inhibitors_ isocarboxazid, phenelzine and tranylcypromine)- results on serotonin syndrome-headache, nystagmus, tremor, dizziness, unsteady gait; irritability, confusion, delirium_ cardiac arrhythmia and coma/death</p>
<p>MAOI</p>
<p>isocarboxazid, phenelzine, tranylcypromine
non-endogenous and atypical depression that involves anx, reverse vegetative symptoms and interpersonal sensitivity.
MAOI inhibits enzyme monoamine oxidase-deactivates dopamine, norep, serotonin.
SE: Hypertensive Crisis-when in conjunction with barbiturates, amphetamines antihistamines, or with foods containing tyramine (cheeses, meats, beer, red wine, avocados, bananas,</p>
<p>Treatment for ADHD</p>
<p>neurofeedback (EEG biofeedback) is effective for inattention and impulsivity and ok for hyperactivity and effects are similar to stimulants ( Methylphenidate (Ritalin) CNS
Behavioral interventions
Teacher training and classroom management: positive reinforcement, time out, ect. Parent Training in Behavioral Management
Medication treatment alone and combined community and intensive behavioral treatment produced similar reduction in core symptoms.</p>
<p>ADHD</p>
<p>6 mos. before 12 yrs. in two settings 6 to 6 subtypes
Cognitive Control theory _ Top Down Theory -Delay aversion (dopamine: BX. reinforcement-delayed aversion) - inhibition, self-awareness, working memory, self directed attention.
impaired reward learning, difficulties adaptively processing rewards, and heightened delay discounting motivational control-executive dysfunction- cog to em/</p>
<p>ADHD Differential D/O</p>
<p>An att. Hyperactivity, and impulsivity can be found in Anx., Stress, and in Mood D/o
Learning D/o- poor concentration
Substances Adult/adolescence- impair exe. func. mild cognitive impairment and increases with age.
Health Conditions: Inatt. and impulsive-seizures-autoimmune, thyroid, concussion
Chronic sleeplessness
Co-occur- 44% with Disruptive BX
ODD (against authority, interpersonal sensitivity and EM. reactivity)
Learning D/O
Anx, and Dep. (Dep. earlier onset)
Tic_core inhibitory
Autism: imp. communication, social reciprocity and stereotype. / repetitive Bx.
Personality_ Borderline. Antisocial</p>
<p>Tics</p>
<p>Tourette's D/O multiple vocal and motor tics occur frequently throughout the day
Persistent (chronic) motor or vocal tic: patient has either motor or vocal tics but not both
Provisional tic D/O tics occur for no longer that 1 year
--Appear early around 2 yrs. mean onset 5 to 7 yrs.
vocal begin sometime later- barks, coughs, throat clearing, sniffs, and single syllables-can be suppressed more so in adulthood, disappear during sleep -they are persistent but can disappear entirely for weeks - frequency increases when one is sick, tired, or stressed.10% boys and 15% girls most motor tics disappear in adulthood, -poor prognosis_ comorbid MH, chronic physical illness lack of support at home and drug use. - strong familial and concordance rate over 50% in mono twins and 10% in Dis. twins- Fam. Hx for OCD
Typically begins by age 6- most severe by ages 10 to 12, AFTER IMPROVE 75% 25% WILL CONTNIUE TO HAVE TICS that ARE mod. or worse-comorbid OCD and ADHD
TD-Both at least 1 vocal and 2 motor-
Persistent Motor or Vocal
--longer than a yr.
Before 18 yrs.
-elevated Dop. antipsychotic drugs - haloperidol and pimozide
SSRI left OCD hyperactivity and inattention treated by clonidine and desipramine</p>
<p>Communication D/O</p>
<p>Childhood Onset Fluency D/O Stuttering-begins in early childhood, distress, diff. speech motor deficits, neurological cond. Ex. stroke, or other mental D/O</p>
<p>Gender differences between rates of Major Depressive D/O</p>
<p>does not become evident until puberty. Prior to that time, the rates between boys and girls are equal.
Adults rates females are 1.5 to 3.0 times the rate for men
Unipolar depression: .5 or 50% in Mono and .2 or 20% in diz. both or one parent with the D/O does not matter.</p>
<p>Panic Attack</p>
<p>recurrent unexpected panic attack with at least one being followed by concern about having another or consequences and change in adaptive behavior.
prepubertal children may experience physical symptoms of panic (hyperventilation) they rarely receive the diagnosis. belief that children do not make catastrophic interpretations of their body symptoms.</p>
<p>Differentiate Panic and agoraphobia</p>
<p>-All involve avoidance
- how many panic attacks and what type (cued, uncued, situationally predisposed) uncured attack suggest panic. cued attacks suggest specific phobia or social anx. (but can be intermixed)
- in how many situations did they occur? Limited situations suggests specific phobias or social anxiety; attacks that occur in a variety of situations suggest panic D/O and agoraphobia
- Does the Pt. awaken at night with panic attacks? This is more typic of panic d/o
- what is the focus of the fear? having a subsequent attack then it's panic D/O, unless it occurs only in one situations suggest as riding a horse then specific phobia situational type.
- Does the pt. constantly worry about having a panic attack even when in no danger of facing a feared situation- this would suggest panic and agoraphobia,</p>
<p>Most effective intervention for GAD</p>
<p>CBT
SSRI or SNRI ( Venlafaxine (Effexor) Duloxetine (Cymbalta) if not benzo an anxiolytic ( Diazepam, alprazolam, oxazepam, triazolam, chlordiazepoxide, lorazepam) stimulate the inhibitory action of GABA= enhance GABA
-</p>
<p>Rates of OCD for males and females</p>
<p>Common for males and females, but onset earlier in males -OCD is more prevalent in males</p>
<p>How does medication and behavioral therapies treat OCD?</p>
<p>SSRI block the reuptake of Serotonin that is believed to be low in OCD PT.
reduces activity in the caudate nucleus that involves converting sensory input into cognitions and actions, and is overactive in people with OCD- other areas include, orbitofrontal cortex, and cingulate cortex which mediates emotional reactions.</p>
<p>How does medication and behavioral therapies treat OCD?</p>
<p>SSRI or Tricyclic (Clomipramine) block the reuptake of Serotonin that is believed to be low in OCD PT.
reduces activity in the caudate nucleus that involves converting sensory input into cognitions and actions, and is overactive in people with OCD- other areas include, orbitofrontal cortex, and cingulate cortex which mediates emotional reactions.
Exposure with Response Prevention</p>
<p>Logan was an 12 yr old boy who was reffered for long standing anxiety about losing his parents and recent fear about getting sick. Recently Logan watched a TV show about deadly diseases. He had 3 panic attacks in the past, has frequent headaches and stomachaches. Insists he is not scared about having another panic attack. His physical complaints are caused by fears of being ill and is petrified about being left sick and alone. Logan had intense separation difficulties starting in kindergarten. His most persistent fear centered around the safety of his parents. He was fine when they were both at work but in transit or somewhere else, he was afraid they would get in a car accident. What is the diagnosis?</p>
<p>Separation Anx with panic
Fear since young child. requires 3 of 8 symptoms.
-long standing excessive and disturbing fears of anticipated separation, to harm to his parents or event that could lead to separations, and of being left alone. physicals complaints that are traced to fear of dying or separation.</p>
<p>This brief treatment that incorporates psychoeducation, relaxation training, cognitive restructuring, and interoceptive exposure (exposure to physical symptoms) treats what disorder</p>
<p>Panic disorder-
Named Panic Control Therapy.
Also treatment includes, TCA, SSRI, SNRRI and benzo. high relapse with discontinued use (70%)</p>
<p>Agoraphobia requires marked fear or anxiety about at least two of the following.. because escape might be difficult and help unavailable in case he/she develops panic like symptoms, incapacitating, or embarrassing symptoms.</p>
<p>using public transportation, being in open spaces, standing in line or being part of a crowd, and being outside the home alone.
Requires presence of a companion
persistent 6 mo.</p>
<p>How do you differentiate Specfic Phobia situational type and social anxiety from Agoraphobia?</p>
<p>Specific Phobia involves fear or anxiety about a single situation that is characteristic of Agoraphobia and is related to something other than concern of experiencing panic like symptoms, incapacitating and embarrassing symp.
Social Anxiety i is anxiety related to being scrutinized by others and increases in the presence of others particularly family or friend while Agoraphobia decreases when accompanied by a family or friend.</p>
<p>What are the clinically significant symptoms of PTSD that one experiences for at least 1 mo ?</p>
<p>intrusion, avoidance, negative alterations of cognition and mood and alterations in arousal and reactivity.</p>
<p>Specific phobias are characterized by intense fear or anxiety about a specific object or situation that persists (6 m. or longer) what are the subtypes?</p>
<p>animal, natural environment (heights, storms), blood-injection-injury, situational and other (loud noises, costumed characters)</p>
<p>What are the causes of Specific Phobias and other anxiety disorder?</p>
<p>Biological factors- abnormal levels of serotonin, norepinephrine and GABA
cognitive factors
Classical Conditioning. Mower's two factor theory: avoidance conditioning (classical and operant conditioning) learn to fear a neutral stimulus (conditioned) stimulus (loud noise and bunny) because it is paired with an intrinsically fear-arousing stimulus and avoidance is negatively reinforcing because it eliminates anxiety</p>
<p>What differentiates PTSD and Acute Stress Disorder?</p>
<p>PTSD requires the presence of symptoms from each of 5 symptoms clusters: intrusion symptoms(re-experiencing, dissociation ect.) avoidance symptoms, negative alterations in cognition and mood and arousal.
ASD can have at least 9 of 14 symptoms meaning that one person could have all 4 intrusion symptoms while another might have one.
Duration PTSD persists at least 1 mo after the external event and ASD lasting no more that 1 mo.</p>
<p>Adriana was a 4.5 yr old girl referred for mental health evaluation of dangerous behaviors particularly poor boundaries, impulsivity, and "too quick to trust strangers." Adriana was adopted from an Eastern Europe orphanage at age 29 mo. Medical records at that time were normal. After adoption, Adriana would seek out her mother for comfort when distressed, however, she did not distinguish between strangers and family. In the grocery store she would hug who ever was in line next to them. Once in a mall she tried to leave with another family. She had trouble taking turns and sitting in a circle. She interrupted, intruded in classmate's space and occasionally hit others. she had trouble self soothing and could generally calm down when held by her teacher or mother. What is the D/O?</p>
<p>Disinhibited social engagement disorder
Preschool aged child with dangerous behaviors that related primarily to excess physical familiarity with strangers= risk for predation
trouble regulating her proximity to other people- going too far away from her mother and getting too close to stranger.
2 symptoms are required out of 4 core symptoms
-reduced or absent reticence in approaching and interacting with unfamiliar adults: overly familiar behavior; diminished or absent checking back with an adult caregiver after venturing away. and willingness to go off with an unfamiliar adult with minimal or no hesitation.
developmental age of at least 9 mo
Child with this disorder can approach caregivers when stressed or hurt.
Comorbid ADHD is common but DSED is specific to relationships.</p>
<p>Traumatic event Bethany and Charles witnessed a shooting at a movie theatre. two days later both Bethany and Charles considered themselves nervous and on edge. They jumped at the slightest noise and kept watching TV for the latest information on the shooting but every time they saw footage, they experience panic attack, brake out in sweats, unable to calm down and couldn't stop thinking about the traumatic event. they both had nightmares and had intrusive thoughts of the shootings
2 wks later. Bethany reclaimed her peritraumatic thoughts, feelings and behaviors. Although reminders of the event resulted in brief panic or physiological reactions these did not dominate her life. Charles did not recover, but felt emotionally constricted and unable to experience positive emotions, he jumped at the slightest sound and was unable to focus, poor sleep with nightmares. He avoided reminders and recalled the sound of shootings, He felt disconnected from his surroundings and self.</p>
<p>Betheny- no diagnosis
Charles - Acute Stress disorder
Normal response to traumatic event- transient reactions will resolve within 2 to 3 days.
Normal response usually presents with emotional reaction- shock, fear, grief, resentment. guilt. shame. helplessness, hopelessness and numbing
Cognitive reactions: confusion, disorientation, dissociation, indecisiveness, difficulty concentrating, memory loss, self blame and unwanted memories
physical reactions-tension, fatigue, insomnia, startle, racing pulse, nausea. loss of appetite.
Interpersonal reaction
Acute is more intense and occur during the month after the event. - minimum of 9 of 14 symptoms spread across 5 categories.
When evaluated individually symptoms may look like, Panic, anxiety, depression, dissociation, and intrusive, obsessional thoughts,</p>
<p>What disorder involves the inability to remember autobiographical information that is not caused by normal forgetfulness and is often related to a trauma?</p>
<p>Dissociative Amnesia
The core symptom of Dissociative Amnesia involves memory loss for periods that extend well beyond the actual trauma. Ex. Sexual abuse with a 6 year memory deficit.</p>
<p>What are the four forms or dissociative amnesia</p>
<p>Di</p>
<p>If you were to do an assessment on an individual with Alzheimer's during the 4th or 5th year of the disorder, you will most likely find?
What symptoms would you most likely see in the first to second year?</p>
<p>-Stage 2 (2 to 10 years) Severe impairments in recent and remote memory. Fluent aphasia indifference or irritability (flat labile mood, restlessness and agitation, acalculia, ideomotor apraxia, (inability to translate a idea to movement, delusions)
- Stage 1 ( anterograde: declarative) Deficits in new learning with remote memory mildly to moderately impaired, anomia, sadness, deficits in visuospatial (wandering) indifference, irritability.</p>
<p>An individual experiencing symptoms of severely deteriorated intellectual functioning, apathy, limb rigidity, and urinary and fecal incontinences has what disorder and what area's of the brain are being affected?</p>
<p>Alzheimer's stage 3
severe neuron loss and the presence of an amyloid-predominant neuritic plaques and tau-predominant neurofibrillary tangles esp. in the--
=medial temporal structures (amygdala, hippocampus, entorhinal cortex) responsible for memory, navigation and the perception of time/ main interface between the hippocampus and neocortex; which system plays an important role in declarative memories particularly spatial including memory formation, consolidation and memory optimization in sleep hippocampus)
--abnormal levels of Ach which is involved in memories. ---</p>
<p>Is there a genetic component to Alzheimer's and what abnormalities indicate early onset and late onset?</p>
<p>Yes. abnormalities in the chromosome 1,14,21 which is linked to familial type early onset and
abnormalities on the ApogeneE4 on chromosome 19 indicating later onset</p>
<p>What is the risk factors to Alzheimer's ?</p>
<p>lower level of education, adult onset (type 2) diabetes, depression, traumatic brain injury, down syndrome</p>
<p>When an individual demonstrates high levels of interoceptive avoidance and fear of interoceptive cues what are they struggling with?</p>
<p>Panic Disorder
| interception (sense of the internal state of the body</p>
<p>Mower's two stage theory of fear acquisition and maintenance notes that?</p>
<p>Fears are originally acquired through classical conditioning and maintained through operant.
Why? The first part of a two-stage theory for acquiring and maintaining fear and avoidance behavior posits that an otherwise neutral event acquires the capacity to provoke fear because of its pairing with an aversive experience, much as a dog phobia might develop from being bitten (Dollard &amp; Miller, 1950; Mowrer, 1960).
In the second stage of the two-factor model, any actions (escape or avoidance behavior, compulsions) that relieve obsessive anxiety/ discomfort are negatively reinforced because they result in reduction of discomfort. Their demonstrated ability to terminate unpleasant experiences renders them very likely to be repeated in future situations</p>
<p>What is the difference between illness anxiety and somatic symptom disorder?
If they engage in deceptive behavior by presenting themselves as sick even in the absence of an external reward are struggling with what disorder?
What is happening if they are doing it for a reward?
What is the best treatment?</p>
<p>illness anxiety is a preoccupation with having a serious illness (family has cancer fear that they have cancer) when you don't (6 mo health checks that suggest no illness; symptoms need to persist for at least 6 m.) and having high anxiety about one's health and perform excessive health related behaviors (care seeking or care-avoidant type). (minimal if any concern over somatic symptoms)
Somatic symptom: presence of a somatic symptom
Factitious disorder
Malingering
Supportive therapy and therapeutic relationship.</p>
<p>Major Depression has been linked to what sleep disturbances</p>
<p>reduced stage 3 and stage 4 sleep (slow wave sleep/delta)
decreased slow wave (non-REM) sleep, decreased REM latency (define: the state of existing but not yet fully developed) ( earlier onset of REM/ 30 to 60 mins after falling asleep instead of 90 mins) and decreased sleep continuity, decreased REM sleep early in the night.</p>
<p>A person does not like their neighbor, does not experience strong emotions, disinterested in marriage and was a loner in school who was teased by his peer. What is his diagnosis</p>
<p>schizoid personality disorder: a lack of desire for interpersonal relationships and restricted range of emotional expression in social settings, doesn't desire or enjoy close relationships. chooses solidarity, little interest in sexual relationships lack close friends except close relatives, indifferent to praise or criticism, exhibits emotional coldness.</p>
<p>Research on the treatment of pediatric acute lymphoblastic leukemia with cranial radiation or chemotherapy have found that</p>
<p>either treatment alone is associated to decrease intellectual ability and academic achievement</p>
<p>PET scam shows that an individual has increased activity in the orbitofrontal cortex, cingulate cortex, and caudate nucleus has what disorder</p>
<p>OCD
caudate nucleus is part of the basal ganglia which is involved in movement, and the orbitofrontal cortex and cingulate cortex are involved in emotional reactions. drug and behavioral treatments lower activity in these areas,</p>
<p>What is the best interventions for cigarette smoking?</p>
<p>nicotine replacement therapy and support from a clinician and skills training that focuses on avoiding and dealing with relapse.</p>
<p>What are the key features to Bulimia Nervosa?</p>
<p>Binge eating and recurrent inappropriate compensatory behaviors (purging, diuretic use, excessive exercise that occur 1 time a wk) for at least 3M</p>
<p>What is the difference between anorexia and bulimia? Over half individuals with Anorexia and Bulimia with the onset usually preceding the diagnosis</p>
<p>Anorexia- restriction of energy that leads to low body weight; intense fear of getting fat, disturbance in how one experiences body weight or lack of recognition of the seriousness of weight loss.
Bulimia is a binge with a lack of control and compensatory behaviors to prevent weight gain. self evaluation that is influenced by body shape and weight
-Anxiety ( Social phobia OCD)</p>
<p>Research on Phototherapy and Seasonal Affective disorder shows?</p>
<p>That is an effective treatment for this disorder and is as effect as antidepressants for reducing symptoms.</p>
<p>Conversion Disorder</p>
<p>A diagnosis of Conversion Disorder requires the presence of symptoms that involve a disturbance in voluntary motor or sensory functioning and suggest a serious neurological or other medical condition with evidence that the symptoms are incompatible with recognized neurological and medical conditions.</p>
<p>The psychoanalyst Adolph Stern provided the first organized clinical description of the borderline patient. Of the ten basic characteristics Stern delineated, which of the following did he consider to be the most primary?
What other theories?</p>
<p>-Stern considered the difficulties experienced by the borderline patient to be secondary to narcissism and viewed narcissism as arising from a serious disturbance in the early mother-child relationship. (Note that other psychoanalytically-oriented theorists have identified other factors as being primary: Kernberg, for example, emphasizes the role of excessive aggression.)
- Object Relations: Mahler: fixation in the reapproachement phase of separation-individuation--need for separation with overwhelming fear of abandonment
- Kerberg: adverse, unpredictable caregiver-child interactions that alternate between rejection and smothering, that produce an insecure ego, that relies on primitives defenses-splitting-all good and all bad
- Linehan's biosocial model: emotional dysregulation as the core. -excessive emotional vulnerability, inability to modulate strong emotions, end exposure to invalidating environment.</p>
<p>Borderline Disorder has a pattern of</p>
<p>A pervasive pattern of instability of interpersonal relationships, self-image, and affects, and marked impulsivity, beginning by early adulthood and present in a variety of contexts, as indicated by five (or more) of the following:
Frantic efforts to avoid real or imagined abandonment. (Note: Do not include suicidal or self-mutilating behavior covered in Criterion 5.)
A pattern of unstable and intense interpersonal relationships characterized by alternating between extremes of idealization and devaluation.
Identity disturbance: markedly and persistently unstable self-image or sense of self. (here may be an identity disturbance characterized by markedly and persistently unstable self-image or sense of self (Criterion 3). There are sudden and dramatic shifts in self-image, characterized by shifting goals, values, and vocational aspirations. There may be sudden changes in opinions and plans about career, sexual identity, values, and types of friends. These individuals may suddenly change from the role of a needy supplicant for help to that of a righteous avenger of past mistreatment.)
Impulsivity in at least two areas that are potentially self-damaging (e.g., spending, sex, substance abuse, reckless driving, binge eating). (Note: Do not include suicidal or self-mutilating behavior covered in Criterion 5.)
Recurrent suicidal behavior, gestures, or threats, or self-mutilating behavior.
Affective instability due to a marked reactivity of mood (e.g., intense episodic dysphoria, irritability, or anxiety usually lasting a few hours and only rarely more than a few days). (Individuals with borderline personality disorder may display affective instability that is due to a marked reactivity of mood (e.g., intense episodic dysphoria, irritability, or anxiety usually lasting a few hours and only rarely more than a few days)
Chronic feelings of emptiness. (boredom)
Inappropriate, intense anger or difficulty controlling anger (e.g., frequent displays of temper, constant anger, recurrent physical fights).
Transient, stress-related paranoid ideation or severe dissociative symptoms.
-onset: 19 through 34 most severe in young adult. impulsive symptoms resolve in 40 (75%) affective most chronic and cognitive and interpersonal were intermediately resolved.</p>
<p>General Personality Disorder</p>
<p>An enduring pattern of inner experience and behavior that deviates markedly from the expectations of the individual’s culture. This pattern is manifested in two (or more) of the following areas:
Cognition (i.e., ways of perceiving and interpreting self, other people, and events).
Affectivity (i.e., the range, intensity, lability, and appropriateness of emotional response).
Interpersonal functioning.
Impulse control.
The enduring pattern is inflexible and pervasive across a broad range of personal and social situations.
The enduring pattern leads to clinically significant distress or impairment in social, occupational, or other important areas of functioning.
The pattern is stable and of long duration, and its onset can be traced back at least to adolescence or early adulthood.
The enduring pattern is not better explained as a manifestation or consequence of another mental disorder.
The enduring pattern is not attributable to the physiological effects of a substance (e.g., a drug of abuse, a medication) or another medical condition (e.g., head trauma).</p>
<p>Recent research has shown that single-session Psychological Debriefing (PD):
What is the treatment of choice? what does it incorporate?
What is usually prescribed?</p>
<p>review of the research that there is sufficient empirical evidence to indicate that PD should not be provided to individuals immediately after a trauma. The studies they reviewed showed that one-session PD is not effective and, in some cases, actually increases the likelihood of PTSD symptoms: Cognitive Incident stress debriefing: within 72 hours, whether or not they show symptoms and is long-bad.
- Comprehensive Cognitive behavioral intervention- incorporates exposure, cognitive restructuring, and anxiety management
- SSRI to reduce depressive symptoms and anxiety</p>
<p>For people with Specific Phobia, blood-injection-injury type</p>
<p>For people with the blood-injection-injury type, feared stimuli produce an initial increase in heart rate and blood pressure, which is immediately followed by a drop in both and fainting. In contrast, people with other types of Specific Phobia experience only an increase in heart rate and blood pressure. Because of the physiological response associated with the blood-injection-injury type, treatment involves tensing (rather than relaxing) muscles in the presence of feared stimuli. - relaxation techniques are contraindicated</p>
<p>What is the difference between an illusion and an hallucination?</p>
<p>An illusion is a misperception of reality (e.g., misperceiving a coffee mug as a rodent).
Answer B is incorrect: A delusion is a false belief about reality that is firmly held regardless of evidence to the contrary.
Answer C is incorrect: While an illusion is elicited by an actual stimulus, an hallucination is a sensory perception in the absence of an external stimulus</p>
<p>When do you ECT to treat Depression?
What is the primary undesirable effect of ECT?</p>
<p>-when severe enough and involves delusions, and suicidal ideation or have not responded to antidepressants. -
Temporary anterograde and retrograde amnesia, confusion, and disorientation - this can be reduced by administering ECT unilaterally to the right (nondominant) hemisphere - less retrograde amnesia but also less anterograde amnesia for verbal and nonverbal tasks.</p>
<p>A DSM-5 diagnosis of Schizophrenia requires the presence of two or more active-phase symptoms during a one-month period with at least one symptom being \_\_\_\_\_\_\_\_\_\_\_\_ plus continuous signs of disturbance for least six months.</p>
<p>Hallucinations, delusions, and disorganized speech
(Schizophrenia spectrum and other psychotic disorders include schizophrenia, other psychotic disorders, and schizotypal (personality) disorder. They are defined by abnormalities in one or more of the following five domains: delusions, hallucinations, disorganized thinking (speech), grossly disorganized or abnormal motor behavior (including catatonia), and negative symptoms. Avolition- reduced drive to pursue goal-directed behavio)
Two (or more) of the following, each present for a significant portion of time during a 1-month period (or less if successfully treated). At least one of these must be (1), (2), or (3):
Delusions.
Hallucinations.
Disorganized speech (e.g., frequent derailment or incoherence).
Grossly disorganized or catatonic behavior.
Negative symptoms (i.e., diminished emotional expression or avolition).
For a significant portion of the time since the onset of the disturbance, level of functioning in one or more major areas, such as work, interpersonal relations, or self-care, is markedly below the level achieved prior to the onset (or when the onset is in childhood or adolescence, there is failure to achieve expected level of interpersonal, academic, or occupational functioning).
Continuous signs of the disturbance persist for at least 6 months. This 6-month period must include at least 1 month of symptoms (or less if successfully treated) that meet Criterion A (i.e., active-phase symptoms) and may include periods of prodromal or residual symptoms. During these prodromal or residual periods, the signs of the disturbance may be manifested by only negative symptoms or by two or more symptoms listed in Criterion A present in an attenuated form (e.g., odd beliefs, unusual perceptual experiences).
Schizoaffective disorder and depressive or bipolar disorder with psychotic features have been ruled out because either 1) no major depressive or manic episodes have occurred concurrently with the active-phase symptoms, or 2) if mood episodes have occurred during active-phase symptoms, they have been present for a minority of the total duration of the active and residual periods of the illness.
The disturbance is not attributable to the physiological effects of a substance (e.g., a drug of abuse, a medication) or another medical condition.
If there is a history of autism spectrum disorder or a communication disorder of childhood onset, the additional diagnosis of schizophrenia is made only if prominent delusions or hallucinations, in addition to the other required symptoms of schizophrenia, are also present for at least 1 month (or less if successfully treated).</p>
<p>What ADHD symptoms decrease in adolescents.</p>
<p>Hyperactivity typically decreases in adolescence, while impulsivity (especially verbal and emotional impulsivity) and inattention continue. In addition, during adolescence, ADHD is often masked by other problems such as oppositionality, learning difficulties, low self-esteem, and deficits in social skills.
-- Faraone and colleagues concluded that the rate of persistence of ADHD into adulthood depends on how persistence is defined. The results of their meta-analysis indicated that about 15% of children with ADHD continue to meet the full diagnostic criteria for the disorder in early adulthood and that between 40 and 60% meet the criteria for ADHD in partial remission.</p>
<p>What are the associated features of Schizophrenia?
What is the prevelance rate? and is it higher in females or males?</p>
<p>Individuals with schizophrenia may display inappropriate affect (e.g., laughing in the absence of an appropriate stimulus); a dysphoric mood that can take the form of depression, anxiety, or anger; a disturbed sleep pattern (e.g., daytime sleeping and nighttime activity); and a lack of interest in eating or food refusal. -Depersonalization, derealization, and somatic concerns may occur and sometimes reach delusional proportions. -Anxiety and phobias are common(Tandon et al. 2009). -Cognitive deficits in schizophrenia are common and are strongly linked to vocational and functional impairments. These deficits can include decrements in declarative memory, working memory, language function, and other executive functions, as well as slower processing speed(Mesholam-Gately et al. 2009). -Abnormalities in sensory processing and inhibitory capacity, as well as reductions in attention, are also found. -Some individuals with schizophrenia show social cognition deficits, including deficits in the ability to infer the intentions of other people (theory of mind)(Bora et al. 2009), and may attend to and then interpret irrelevant events or stimuli as meaningful, perhaps leading to the generation of explanatory delusions. These impairments frequently persist during symptomatic remission.
Some individuals with psychosis may lack insight or awareness of their disorder (i.e., anosognosia). This lack of “insight” includes unawareness of symptoms of schizophrenia and may be present throughout the entire course of the illness
-The lifetime prevalence of schizophrenia appears to be approximately 0.3%–0.7%.
-The sex ratio differs across samples and populations: for example, an emphasis on negative symptoms and longer duration of disorder (associated with poorer outcome) shows higher incidence in males whereas definitions allowing for the inclusion of more mood symptoms and brief presentations (associated with better outcome) show equivalent risks for both sexes</p>
<p>There is a higher reported rate of African Americans to Caucasians but this is due to a misdiagnosis due to the fact that African Americans are more likely to experience \_\_\_\_\_\_\_\_\_\_\_\_\_\_\_?
Also patients in developing countries are more likely to experience \_\_\_\_\_\_,\_\_\_\_\_\_, \_\_\_\_\_\_\_\_</p>
<p>Hallucinations and delusions as symptoms of depression and other disorders.
acute onset, shorter clinical course, complete remission.</p>
<p>Good prognosis for Schizophrenia
What brain abnormalities?</p>
<p>good premorbid adjustment, acute and late onset, female, presence of precipitating event, brief active phase, insight, family history of mood, no family history of schiz.
Psychotic symptoms tend to diminish over the life course, perhaps in association with normal age-related declines in dopamine activity (dopamine hypothesis: elevated levels of dopamine)
Negative symptoms are more closely related to a poor prognosis than are positive symptoms and tend to be the most persistent. Furthermore, cognitive deficits associated with the illness may not improve over the course of the illness possibility due to hypofrontal land lower than normal activities in the prefrontal cortex
Enlarged Ventricles is the most common</p>