5) AV & Blocks Flashcards
Junctional Arrhythmias:
(PJC) Premature Junctional Contractions
Junctional Escape Complexes and Rhythm
Junctional Bradycardia
Accelerated Junctional Rhythm
Junctional Tachycardia
Ventricles have more muscles so P wave
“eat P wave” if AV fires same as ventricles
Digoxin) Typically for:
Dynamics
works bc
= CHF
= allows more Ca for better contraction
= confuses K/Na pumps
“AV node P waves” morphology:
= inverted before QRS, hidden w/in QRS, after QRS
w/ (PJC) Premature Junctional Contraction) 1Rules:
2CANNOT HAVE B/C:
3Compensatory pause
4Non-compensatory pause
1= rate by rhythm, usually slightly irregular, P waves are either inverted before, +after, or hidden w/in QRS
2=have upright P wave (up P= PAC)
3= keeps cadence
4= doesn’t keep cadence
AV pacing site defined by:
= P wave: 1 inverted before QRS, 2 hidden w/in QRS, 3 +/- after S
(AV pace impulses relation w/ P waves)Atriums fire 1st then ventricles:
Atriums & Ventricles fire at same time:
Ventricles fire 1st then atriums fire 2nd:
= inverted P wave before QRS
= P wave hidden w/in QRD
= P wave after QRS (before T wave)
- (Junctional rhythms) aka know by:
- Definer:
1= junctional escape: “pick up workload b/c something failed”
2= AV P waves & AV node rate 40-60BPM, Regular rhythm
- (Junctional rhythms) aka know by:
- Definer:
- S/S:
- Rules:
- Treatment:
1= junctional escape “pick up workload b/c something failed”
2= AV P waves & AV node rate 40-60BPM, Regular rhythm
3= Slow heart rate can decrease CO; angina
4= AV: Pace site, rate, & P-waves> regular rhythm, can have >PRI
5= O2 as needed, 15 Lead ECG, underlying cause (MI commonly), If signs poor perfusion, prepare for transcutaneous pacing (TCP)
(Junctional Bradycardia) 1. Remember:
2. Rules:
3. Etiology:
4. S/S
5. Treatment of Symptom Stable:
6. Treat of Symptom Unstable:
1= AV inherit firing rate 40-60 so <40BPM AV Brady
2= <40BPM, REG/ rhythm, AV P waves, QRS WNL but can be wide
3= +Vagal nerve tone, Patho/ slowing of SA node rate
4= Decreased HR: decreased CO, hypotension, angina, CNS S/S
5= “table” treat w/ Med admin/ of pos/ underlying cause (SBP >90)
6= (SBP<90 or AMS) “go straight 8 Cables!” PPM 60-80, Pace ASAP to increase pacing’s efficiency
(Atropine & Dopamine) 1. Med/ Admin/ for:
2. Atropine dosing:
3. Dopamine dosing:
(Symptomatic unstable) 4. S/S: go Cables! EX unconscious, RR<4,
5. Treatment:
Mili Amps MA (need to touch PT to feel pulse)
Pace ASAP to increase chance of pacing
1 = SBP greater than 90mmHg, “Stable to the table”
2= 1mg 3-5mins as needed (0.04mg/Kg (total 3mg)
3= “Real”2-5mcg, BC>5-10mcg/kg/min, Vaso-press> 10-20 mcg/kg/min
4= inadequate perfusion: hypoBP, AMS, etc)
5= “Straight 8 Cables!” PPM 60-80, (TCP)Transcutaneous Pacing ASAP
(Heart Blocks Raps) If the R is far from the P, then you have a:
Longer, longer, longer, drop, then you have a:
If some Ps don’t get through, then you have a:
If Ps and Qs don’t agree, then you have a:
= FIRST DEGREE!
= WENCKEBACH!
= MOBITZ II!
= THIRD DEGREE!
(Heart Blocks Raps) If the R is far from the P, then you have a:
FIRST DEGREE!
(Heart Blocks Raps) Longer, longer, longer, drop, then you have a
= WENCKEBACH!
(Heart Blocks Raps) If some Ps don’t get through, then you have a:
= MOBITZ II!
(Heart Blocks Raps) If Ps and Qs don’t agree, then you have a:
= THIRD DEGREE!
Systematic approach
- rate, 2. rhythm, 3. P waves, 4. PRI, 5. QRS
Re-entry loops
= stuck in nascar loop in a chambers pathway causing SVT / no P waves
WPW) Orthodromic loop;
Antidromic loop:
Treatmeats:
= Clockwise reentry w/ narrow complex
= Counterclockwise reentry w/ wide QRS
= procainamide 1a Na blocker, (if no procain) sedate & cardiovert) cardioversion
- (Accelerated Junctional) Know by:
- Definer:
- Symptomology:
- Treatment:
1= “Baby Tachy” faster than 60 not faster than 100
2= 61-100BPM, (from SNS & AV firing), Regular rhythm, AV P waves
3= usually does not cause a PT to have symptoms
4= Be a investigator ,History/Physical ,O2 as needed , 15 Lead ECG
- (Accelerated Junctional) Know by:
- Definer:
1= “Baby Tachy” faster than 60 not faster than 100
2= 61-100BPM, (from SNS & AV firing), Regular rhythm, AV P waves
- (Junctional Tachycardia) Know by:
- Definer:
- Etiology:
- Rules:
- Symptomatology:
- Treatment:
1= “Tachy is Tachy”
2= >100BPM, AV P waves
3= +SNS response w/ AV site & Result of AV ischemia (rarely>150)
4= >100, AV P waves & Pacing, N. QRS, ~reg/rhythm, if PRI ~<.12secs
5= usually PT doesn’t has symptoms
6= invest/, Hx, O2 PRN, 15 Lead, monitor for other arrhythmias
- (Junctional Tachycardia) Know by:
- Definer:
1= “Tachy is Tachy”
2= >100BPM, AV P waves
Atrioventricular (AV) block:
Electrical impulse is slowed or blocked as it passes through AV node
AV block possible causes:
MI, (inferior RCA) AV ischemia and/or necrosis, Degenerative disease of conductive system, Drug toxicity (particularly digitalis)
Heart Blocks 4 Different Types) A.
B.
C.
D.
1st-Degree AV Block> add to rhythm “w/”
2nd-Degree Type I AV block (Mobitz I, or Wenckebach) rhythm
2nd-Degree Type 2 AV block (Mobitz 2 or intranodal)”2:1 block” rhythm
3rd-Degree AV Block aka “complete heart block” rhythm
2nd-Degree Type I AV block) names
Mobitz 1 or Wenckebach
Wenckebach) Sir name
2nd-Degree Type I AV block
2nd-Degree Type 2 AV block) names:
Mobitz 2 or intranodal AKA “2:1 block” rhythm
Intranodal/Mobitz 2) Sir name
2nd-Degree Type 2 AV block
- (1st Degree AV Block) know:
- Definer:
- Rap:
- Rules:
- Etiology:
- Cause:
- Symptoms:
- Treat:
1= “add to any rhythm” “gandolf slowly opening door(PRI)”
2= PRI: >than 0.20 seconds for every PRI & P-P cadence
3= “If the R is far from the P, then you have a FIRST DEGREE!”
4= BPM & rhythm is underlying rhythm, P Waves: Norm/ shape, PRI >0.20secs Pace-Site: SA or atria, QRS: Usually normal
5= Delay in AV node rather than actual block (increases PRI), Not a rhythm but a condition w/in another rhythm
6= Ischemia @ AV junction, MI/@AV, getting old, arteriosclerosis
7= PT usually don’t have symptoms
8= investigate, History/Physical, O2 as needed, 15 Lead ECG
- (1st Degree AV Block) know:
- Definer:
1= “add to any rhythm” “gandolf slowly opening door(PRI)”
2= PRI: >than 0.20 seconds for every PRI & P-P cadence
- (2nd Degree Type I) AKA & Know:
- Definer:
- Rap:
- Rules:
- Symptomology:
- Treatment:
1= “Morbitz 1”/“Wenckebach” rhythm & “AV turning off to fully down”
2= progressive longing PRI till drops beat then resets/starts over
3= “Longer, longer, longer, drop, then you have a WENCKEBACH!”
4= Rate: Variable, QRS rate will be slower than atrial rate Rhythm: Irregular P Waves: Normal but some P waves don’t have a QRS PRI: longing ‘til a QRS dropped Pace-Site: SA node or atria QRS: ~normal
5= Can compromise cardiac output, Syncope, angina, Commonly MI
6= O2 as needed, 15 Lead ECG, If signs of poor perfusion, prep for transcutaneous pacing only if brady.
- (2nd Degree Type I) AKA & Know:
- Definer:
1= “Morbitz 1”/“Wenckebach” rhythm & “AV turning off to fully down”
2= progressive longing PRI till drops beat then resets/starts over
- (2nd Degree Type II) AKA & know:
- Definer:
- Rap:
- Rules:
- Etiology:
- Symptomology:
- Treatment:
1= “Mobitz 2/Intranodal” & “random extra Ps”
2= some P’s w/o QRS & same PRI/No longing before drop beat
3= “If some Ps don’t get through, then you have a MOBITZ II!”
4= Rate P’s unaffected; QRS rate usually brady Ir/Reg/ Rhythm, P’s WNL but some w/o QRS, PRI Constant for conducted beats, Pace-Site SA node or atria, QRS WNL or wide
5= Intermittent block, Ps not conducted to ventricles via AV (Associate w/ acute MI & septal necrosis)“2-1 block” = 2 P’s before QRS
6= May comp/ CO, syncope, angina; May dev/ into complete AV-block
7= PT condition based: If signs of poor perfusion prep for trans/pacing
- (2nd Degree Type II) AKA & know:
- Definer:
1= “Mobitz 2/Intranodal” & “random extra Ps”
2= some P’s w/o QRS & same PRI/No longing before drop beat
- (3rd Degree AV Block) AKA & know
- Definer:
- Rap:
- Rules:
- Etiology:
- Symptomology:
- Treatment:
1= “Complete AV-Block/dissociation” (always TCPP on) “gandalf died”
2= No relations w/ Ps & QRSs & no same PRI (top & bottom dif)
3= “If Ps and Qs don’t agree, then you have a THIRD DEGREE!”
4= P’s unaffected; QRS rate usually brady Rhythm: Ps & QRSs WNL but don’t coincide w/ other Ps: Norm w/ no relation w/ QRS Pace-Site: SA node or atria for P’s; AV node or Ventricle for QRS, QRS WNL or wide
5= NO conduction w/in atria & ventricles, Complete electrical block @/ below AV node Acute MI, Digoxin toxicity, Degen/ of conductive system
6=May severely compromise CO
7= If signs of poor perfusion, prep for immediate TCP
- (3rd Degree AV Block) AKA & know
- Definer:
1= “Complete AV-Block/dissociation” (always TCPP on) “gandalf died”
2= No relations w/ Ps & QRSs & no same PRI (top & bottom dif)
only condition A-Fib has cadence:
Afib w/ 3rd degree In rhythm “Gandalf dead so Atriums & Ventricles doing own thing
(Adenosine) class:
Dynamics:
= Misc antiarrhythmic binds to adenosine A1 receptors causes efflux of K & inhibits Ca influx (in autoarhythmic cells)
= Causes hyperpolarization of autorhythmic cells (SA/AV node)
Slows AV conduction w/ very short half-life
(Adenosine) indications:
contraindications
= 1st for stable narrow complex SVT, Regular & monomorphic wide-complex Tcardia thought from a reentry SVT (SVT w/ BBB)
= Torsades de pointes, Poison/drug-Tcardia, 2nd or 3rd AVB, WPW,DOESNT CONVERT A-FIB/FLUTTER