3) Hemorrhage & Shock Flashcards

1
Q

Internal Hemorrhage being forced between tissue layers (fascia):
Internal Hemorrhage:
Pregnant internal hemorrhage worry:
Internal blood loss may be self limiting:

A

= Hematoma (pocket of blood) usally from arterie)
= Blood accumulation in interstitial spaces, forming visible contusions
= multiple uterine arteries in umbilical cord
= Large body cavities (chest, ABDMN, pelvis) have little resistance & internal blood loss may be severe.

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2
Q

Vascular system consists of 3 types of vessels:
Arteries, veins, Capillary:
blood vessels that are called the “capacitance system” are the:
A blockage in the delivery of oxygenated blood to the cells:
Vessels that’re only 1 cell thick, allow efficient gas and metabolic substrate M-nt into & out of interstitial space are:

A

= Arteries, Veins, Capillaries
= “A Away”, “V Venga”, “Cap C venioles & arterioles for exchanging”
= Veins
= Ischemia
= Capillaries

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3
Q

If a decision is made to use a tourniquet to stop external hemorrhage, an important point to remember is that:

A

The accumulation of lactic acid, potassium, and anaerobic metabolites can occur with tourniquet use

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3
Q

The middle, muscular layer of the blood vessels that controls lumen size:

A

Tunica Media

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4
Q

The blood volume of an infant or young child is proportionally:
Population most at risk for trauma & trauma death:

A

= ~20%> than that of an adult
= Young adult male

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4
Q

Network of fibers that stick together, form strong protein mesh, & entrap RBCs, to form a stronger, more durable clot is:
Platelets are Important for:
Protein fibers that trap red blood cells as part of the clotting process:

A

= Fibrin
= clotting and blood vessel repair
= Fibrin

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5
Q

During Stage 2 hemorrhage, PT’s pulse pressure will:
PT lost ~20% of total blood vol/ anxious, restless, & cool clammy skin is In which stage of hemorrhage is he?

A

= Be noticeably narrower
= Stage 2

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6
Q

Patients with epistaxis should be placed:
Nose bleeding result from injury, disease, or environmental factors:

A

= In the recovery position
= Epistaxis

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7
Q

Stroke volume is predicated by:
Amount of blood ejected by heart in one cardiac contraction:
Pressure w/in ventricles at the end of diastole:
Blood vol/ moved by heart in 1 min:
Measurement of peripheral vascular resistance is the:
Resistance a heart contraction must overcome in order to eject blood:

A

= Preload, afterload, and cardiac contractility
= Stroke Volume
= Preload
= Cardiac output
= Mean arterial pressure
= Afterload

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8
Q

ETCO2 Lvs in head-injured intubated PT shouldn’t drop below:
When vent/ing a adult PT, each breath should have tidal volume of:

A

= 30 mmHg
= approximately _500 mL’s.

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8
Q

Body maintaining steady, norm/ internal environment:
A condition in which the blood’s ability to clot is impaired:
The body’s response to stop the loss of blood:
Clotting phase when smooth blood vessel M. contracts <lumen size:
1 common factor hindering clotting process is:
aggregation:

A

= Homeostasis
= Coagulopathy
= Clotting
= Vascular phase
= Medications such as aspirin
= process of coming & sticking together

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9
Q

Precapillary sphincters dilate & permit blood flow through capillaries during times of low O2 & CO2 build up in cells b/c:

A

= release of chemical Histamine

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10
Q

CUPS acronym, the “P” stands for:

A

the “P” stands for: Potentially unstable

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11
Q

An abnormal internal or external discharge of blood:
Venous bleeding usually looks:

A

= Hemorrhage
= Dark red and flowing

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12
Q

A state of inadequate tissue perfusion:
The ultimate killer of all trauma victims is:
A precipitous drop in blood pressure signals which stage of shock?
Cutaneous vasoconstriction in the shock victim contributes to:
In irreversible shock, if aggressive resuscitation restores pulse & BP:

A

= Shock
= Shock
= Decompensated
= Hypothermia
= Death still occurs

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13
Q

Tension pneumo & pulmonary emboli is which type of shock?
Pericardial tamponade presents w/:

A

= Obstructive
= Becks triad JVD, tachycardia, hypotension, & Muffled heart tones

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14
Q

1st step used to control external bleeding is:
med w/ beneficial role in management of trauma/hemorrhagic shock:
A weak pulse:

A

= Direct pressure
= Tranexamic acid TXA
= Thready

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15
Q

If you suspect a pelvic fracture in your patient, you should:

A

Not compress or otherwise manipulate the pelvis

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16
Q

The molecule in the blood to which oxygen attaches is:
Able to live without oxygen:
Protein that spurs production of RBC in bone marrow:

A

= Hemoglobin
= Anaerobic
= Erythropoietin

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17
Q

Cardiogenic shock:

A

Has a high mortality rate

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17
Q

Septic shock

A

Toxins release affecting vascular system’s control of blood vessels

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18
Q

Recent evidence suggests that Mechanism of Injury:

A

may not be as good an injury severity predictor as once thought.

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19
Q

if one lung needs to be darted:

A

= dart the other lung

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19
Q

Sympathetic NS is the division of the autonomic nervous system that:

A

+ tropic effects, GI constriction, vasoconstriction, mydriasis

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19
Q

Orthostatic hypotension

A

PT’s BP drops 20 HR+20 when moved from supine to a seated position

20
Q

Starling’s law of the heart process:

A

= increase CO occurs in proportion to diastolic stretch of heart M. fibers

21
Q

Perfusion is:
Internal cellular gas exchange:
External gas exchange:

A

= Adequate supply of oxy/gen/ed blood & nutrients to all vital organs
= blood/ cells
= pulmonary gas exchange

21
Q

Baroreceptors:
Baroreceptors:

A

= receptors that monitor blood pressure
= Great vessels recept/ Gives feedback to brain > Sympathetic NS Activation, AArch & carotid arteries> feedback to medulla >SNS

22
Q

Multi organ dysfunction:

A

organs dying b/c lacking perfusion (ICU dying shock)

23
Q

Beta blockers effect w/ blood
Ca-blockers effect w/ blood

A

= block norepi & epi
= cant shunt/vaso-constrict

24
Q

(Blood Vessel Control) Blood flow through capillary beds
which vessel vaso-constricts best & which barely can constrict:
blood sits in which vessel systems:

A

= reg/ed by capillary sphincters: Under control of ASNS
= Cap constrict most, Veins >greatly & arteries barely constrict
= Venous system> 2/3s & ~13% in arterial system

24
Q

Hypoadrenalism

A
  • hard response to stress
25
Q

Ventilations increasing indicates
Pale cool diaphoretic skin indicates

A

= aerobic to anaerobic drive
= shock

26
Q

Shock’s 5 categories

A

Hypovolemic, Cardiogenic, Distributive, Obstructive, resp/metabolic

26
Q

(Shock categories) Hypovolemic:
Cardiogenic:
Distributive:
Obstructive:
resp/metabolic:

A

= fluid loss non/hemorrhagic (most common)
= heart can no longer pump
= anaphylaxis, sepsis, Neurogenic
= obstructsion to blood flow/hemostasis
= DKA- bohr haldane (compartment)

27
Q

3 parts of circulatory system:
maintain homeostasis via:
Pos feedback:

A

= “pump fluid pipes” heart, blood, & blood vessels
= negative feedback loop
= childbirth & blood clots

28
Q

Base of the heart @
Apex of the heart @:
Apex of heart attachs to
Heart’s 4 chambers:

A

= ~2nd rib
= just above the diaphragm
= diaphragm
= Atriums 2 upper chambers & ventricles 2 lower chambers

29
Q

DIC intra cholopathy (septic shock)

A
30
Q

Ventricles separated by:
Atriums separated by:
bi/tricuspid valves separate:

A

= intraventricular septum
= interatrial septum
= upper & lower chambers

31
Q

Pericardial steatosis:
Pericarditis PT’s will have more pain with:

A

= extract blood from pericardial sac fixing tamponade
= laying down & coughing

31
Q

Stroke volume:
Cardiac Output (CO): Amount of blood moved in 1min
CO = HR X SV > 5-6 L of blood moved in 1 minute
3 factors that affect cardiac output: preload, afterload, contractility
Dynamic CO:

A

= blood amount ejected in 1 contraction> varies 60-100mL 70average
= Amount of blood moved in 1min
= HR X SV -> 5-6 L of blood moved in 1 min
= preload, afterload, contractility
= “1 up & other down”

32
Q

Frank Starling law/mechanism:

Vascular Resistance:
SVR vs. PVR:
SVR (Systemic Vascular Resistance):
PVR (Pulmonary Vascular Resistance):

A

= more myocardium is stretched the more force/greater the next contraction will be (increase in preload increases contraction)
= Resistance to overcome to push blood through circulatory system
= Body’s circulation resistance & Lung’s circulation resistance
= resistance blood faces in systemic circulation (arteries size)
= resistance blood faces in pulmonic circulation EX CHF, R ventricle

33
Q

Preload:
Afterload:

Mean Arterial Pressure (MAP):

A

= pressure/vol/ w/in ventricles @ end of diastole “End-diastole vol/“
= resistance against which the heart must pump against (increase afterload= increase ventricular workload)
= average pressure in arteries during a single cardiac cycle = pressure that drives blood to tissues (ensuring organ perfusion)

34
Q

cardiovascular system arrangement of connected tubes:
Tunicas:
Capillaries made by
Capillaries sphincters:

A

= Arteries, Arterioles, Capillaries, Venules, Veins
= Intima, Media, Externa
= venules & arterioles connecting
= “muscles” determine shunt/flow

35
Q

Pulse pressure:
Pulse pressure signifies:
Pulse pressure <25 mmHg may be seen w/

A

= dif/ between DBP & SBP pressures (PP= SBP-DBP)
= amount of force the heart generates with each contraction
= PTs w/ sings of shock

36
Q

External Hemorrhage:
Transmural Pressure:
Intramural Pressure:
How to treat both:

A

= Transmural Pressure > Intramural Pressure
= Pressure inside of blood vessels
= pressure outside of blood vessels
= apply more pressure outside than inside to tamponade (direct P)

36
Q

Hemostasis:
Hemostasis order:
Factors that effect hemostasis:

A

= Body’s response to a local hemorrhage w/ 3 phases
= Vascular, Platelet, Coagulation “flow, stick, build”
= Wound type(can pull w/in>cant constrict), M-nt, Temp, Meds( most blood thinners stop/prevent fibrin) Warfrin/Coumadin), IV Fluids

36
Q

Body’s response to a local hemorrhage:
Vascular Phase:

A

= hemostasis 3 steps: Vascular, Platelet, & Coagulation phase
= Blood vessel is damaged & begins to lose blood , Smooth muscle w/in walls contracts, thus vessel to withdraw into the wound, thicken vascular wall, & reduce lumen.

36
Q

Blood:
Plasma:
Leukocytes:
Erythrocytes:

A

= Mixture of water, cells, proteins, & suspended elements.
= makes up 55% of the blood volume
= WBC & platelets make up the “Buffy Coat”
= RBC make up 45%

37
Q

Fluid accounts for ~% of the body’s weight, Only ~% of the fluid is contained in our vascular system

A

= 60% of the body’s weight, Only about 7% of the fluid is contained in our vascular system

37
Q

tools for external hemorrhage:

Trauma Clamps:
Tourniquets:

Tourniquet use adverse effects:
Hyperkalemia & Sign:

A

= Hemostatic Agents/Dressings, (combat gauze), trauma clamp, Tourniquets,
= IT clamps “teeth chomper”
= Commercial best at least 1in diameter/width (Willis=turning stick), Improvised (ad hawk), Air inflatable (like BP cuff), Junctional (sam splint=pelvic)
= Due to distal hypo-perfusion, toxins build up>Hyperkalemia:
= tall sharp T waves & treat w/ sodium bicarb bc Acidosis

38
Q

body’s 1st line comp response:

A

= Blood loss > decreased arterial pressure, altered blood gases > baro&chemoreceptor reflex >cardiac stim/ systemic vasoconstriction, flow&Vol/ retribution

39
Q

2nd line comp response:

A

Blood loss > RAAS, Catecholamine & Vasopressin released> vasoconstriction, increased Vol, cardiac stim

40
Q

Platelet Phase of hemostasis:

A

2nd phase, Platelets aggregate, or collect and adhere. Slows hemorrhage from capillaries and small vessels- splint fractures to decrease of clots being broken down & bleeding again

41
Q

Coagulation Phase of hemostasis:

A

3rd phase Clotting factors activated and released into bloodstream through a very complex cascade of events Triggers series of chemical reactions; formation of strong protein fibers (fibrin)

42
Q

(Class I Hemorrhage) 1 injuries:
2Compensation for blood:
3Blood Loss:
4Pulse:
5Vent rate, BP & Pulse Pressure:
6Cap-Refill:
7Urine Output (mL/hr):
8Mental Status:

A

1= Broken humorous 750mL
2= Healthy PTs can easily compensate for such blood volume loss
3= Blood Loss: < 15% (<750 mL’s)
4= Pulse: Slightly Tachy
5= all Normal
6= Cap-Refill <2secs
7= 30mL/Hr or more
8= Slightly Anxious

43
Q

(Class II Hemorrhage) 1 injuries:
2Compensation for blood:
3Blood Loss: 15 - 30%
4Pulse:
5Blood Pressure:
6Pulse Pressure:
7Capillary Refill:
8Ventilation Rate:
9Urine Output (mL/hr):
10Mental Status:

A

1= 1/2 Humorous fractures, a femur fracture, 1 full Hemopneumo
2= 1st line comp/ no longer maintain perfusion & 2ndary employed
3= 15 - 30% 750mLs-1.5L
4= > 100BPM
5= Normal
6= Starts to narrow
7= 2-3 secs
8= 20-30RR
9= (mL/hr) 20-30
10= Mildly Anxious

44
Q

(Class IV Hemorrhage) 1 injuries:
2= Compensation to blood:
3= blood loss:
4= Pulse:
5= Blood Pressure:
6= Pulse Pressure:
7= Capillary Refill:
8= Ventilation Rate:
9= Urine Output (mL/hr):
10= Mental Status:

A

1= GSWs, multiple major fractures, Pelvis
2= Irreversible Shock!
3= > 40% (>2000mLs) of total blood
4= > 140 & barely palpable in central arteries
5= Very low
6= Narrows more
7= > 5 seconds
8= > 40 or agonal
9= Negligible
10= Lethargic or Unconscious

44
Q

(Class III Hemorrhage)1 injuries/fractures:
2 Compensation to blood:

3 Blood Loss:
4 Pulse:
5 Blood Pressure:
6 Pulse Pressure:
7 Capillary Refill:
8 Ventilation Rate:
9 Urine Output (mL/hr):
10 Mental Status:

A

1= 2 Humorous, 1-2femur, 1 full Hemopneumo
2= Both 1&2nd comp/ responses failing to maintain perfusion & entering/in Decompensated Shock! (SBP <90)
3= Blood Loss: 30 - 40%, (1500 - 2000 mL’s)
4= >120
5= Starts to decrease
6= Narrows more
7= 3-4secs
8= 30-40
9= 5-10mL/hr
10= Anxious/Confused

45
Q

(Non& Hemorrhagic treatment) If hemorrhage can be controlled:
If hemorrhage cannot be controlled:

Med for Sig/hemorrhage, in/external (after external controlled)

A

= IV/IO therapy (don’t delay transport) can administer 20 mL/kg bolus.
=administer just enough IV fluid to obtain a radial pulse (permissive hypotension therapy!)NO MORE SBP 80-90 (IV fluids Warm)
= Tranexamic Acid (TXA) Adult 1G/10 mins (mix in 50 mL of NS) follwed w/ 1G/8Hrs (500 mL bag) & Pediatric Not recommended