02.18b Surgical Management for Valvular Heart Disease Flashcards

1
Q

Etiology of MS

A

Rheumatic fever

RHD

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2
Q

Pathophysiology of MS

A

Leaflet thickening or calcification
Commissural fusion
Chordal fusion and shortening

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3
Q
Exertional dyspnea
Decreased exercise capacity
Orthopnea/paroxysmal nocturnal dyspnea
Hemophysis and pulmonary edema
Pulmonary hypertension
Afib
A

MS

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4
Q

Auscultatory findings in MS

A

Increased first heart
Opening snap
Apical diastolic rumble

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5
Q

Primary cause of mortality for MS

A

Progressive pulmonary and systemic congestion

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6
Q

CXR findings for MS

A

Straight left border (left atrial enlargement, pulmonary artery enlargement)
Double contouring

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7
Q

For ruling out left atrial appendage thrombus and Mr severity for percutaneous mitral balloon valvotomy candidates

A

Transesophageal echocardiography

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8
Q

First choice of treatment for MS

A

Percutaneous mitral balloon valvotomy

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9
Q

lf not amenable for valvotomy

A

Commissurotomy

Mitral valve repair or replacement

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10
Q

Etiology of MR

A
Mitral valve prolapse
RHD
CAD
Infective endocarditis
Certain drugs
Collagen diseases, carcinoid diseases
Trauma
Previous chest radiation
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11
Q

Apical holosystolic murmur and forceful apical impulse

Transmitted to the left axilla or left sternal border

A

MR

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12
Q

Insufficiency occurs secondary to annular dilatation or leaflet perforation with normal leaflet motion

A

Type I

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13
Q

Thickened leaflet prolapsed, or ruptured or elongated chordae tendinae with increased leaflet motion
Myxomatous conditions, MVP

A

Type II

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14
Q

Restricted leaflet motion

RHD, chronic ischemic MR

A

Type III

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15
Q

If you have MR, what to find in ECG

A

LA and LV enlargement

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16
Q

Evaluate LV size and function, RV and LA size, PA, MR severity
To evaluate MR mechanism

A

Transthoracic echocardiograph

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17
Q

Establish anatomic basis of MR to assess feasibility of repair, and guide the repair

A

TEE

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18
Q

Indications of surgical intervention for patients with MR

A

Any symptomatic patient with MR
Asymptomatic severe MR with LV systolic dysfunctions
*Recent onset of Afib, pulmonary HPN, abnormal response to stress testing

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19
Q

Treatment of choice for MR

A

Mitral valve repair

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20
Q

Etiologies of AS

A

Calcification
Congenital
Rheumatic

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21
Q

Pathophysiology of AS

A

Pressure overload

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22
Q
Exertional dyspnea
Angina
Syncope
Decreased exercise capacity
Heart failure
A

AS

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23
Q

Harsh, crescendo-decrescendo
Systolic murmur at right 2nd ICS
Radiating to carotid arteries

A

AS

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24
Q

ECG findings in AS

A

LV hypertrophy

Conduction abnormalities, Afib

25
Exercise testing of AS
Dobutamine stress echocardiography
26
Procedure for AS with CAD risks, symptomatic but inconclusive results in non invasive tests regarding AS severity
Cardiac catheterization
27
Management of AS
Medical therapy Aortic balloon valvotomy Surgery
28
Etiology of AR
``` Aortic root disease Congenital Calcific degeneration Rheumatic disease Infective endocarditis Myxomatous degeneration ```
29
What will happen if the heart keeps pumping blood to the aorta and blood keeps coming back
Left ventricular dilatation
30
AS is ______ | AR is ______
Pressure overload | Volume overload
31
High pitched decrescendo diastolic murmur in 3rd ICS
AR
32
Bounding pulses quickly collapsing pulses
Corrigan's sign
33
Pistol shot sounds by auscultation
Traube sign
34
ECS findings in AR
Left axis deviation | Intraventricular conduction defects
35
Assess the severity, cause of AR, degree of pulmonary HPN
TTE
36
Considered surgical emergency in AR
With aortic dissections | Infectious endocarditis with severe AR in failure
37
Has traditionally been performed through a median sternotomy incision
Aortic valve replacement
38
Types of surgery
Aortic valve replacement | Aortic valve repair
39
Etiology of TS
Organic (rheumatic fever, endocarditis, rarely trauma)
40
Etiology of TR
Functional (mitral valve disease, pulmonary hpn, RV failure)
41
Pathophysiology of TR/TS
Elevated RA pressure | Right heart failure
42
Signs of severe TS
``` Jugular vein distension Hepatomegaly Splenomegaly Ascites Lower extremity edema ```
43
Diagnosis of TS
``` CXR TTE (size, structure, motion) ```
44
Management of TS
Tricuspid valve repair | Tricuspid valve replacement
45
Pathology of multivalve disease
Rheumatic heart disease Calcific disease Marfan syndrome Secondary to another valve pathology
46
Advantages of mechanical aortic prosthesis
Highly durable | Minimized risk for reoperation
47
Disadvantages of mechanical aortic prosthesis
Require permanent anticoagulation (warfarin) Risk of hemorrhagic complications Lifestyle changes
48
Types of valve of mechanical aortic prosthesis
Ball-cage valve (Starr-Edwards) Single titling disc (Medtronic-Hall, Ominicarbon) Bileaflet prosthesis
49
Advantagaes of biologic valves
Less thrombogenic Less anticoagulant-related complications Good for older population
50
Disadvantages of biologic valves
More prone to structural failure | Higher chance of reoperation
51
Types of biologic valves
Stented and non-stended heterografts Homografts Autografts (pulmonic valve auto-transplantation, Ross operation)
52
Recommendation for warfarin INR 2-3
Increase INR to 2.5-3.5
53
Recommendation for warfarin INR 2.5-3.5
Increase INR to 3.5-4.5
54
Recommendation for no aspirin
Starts ASA 75-100mg
55
Recommendation for warfarin plus ASA
May increase ASA to 325mg
56
Recommendation for ASA alone
Increae ASA to 325mg or add Clopidogrel 75 mg OD and/or warfarin
57
What level of INR is at higher risk for hemorrhage
> 5
58
What to do when INR is 5-10 and not bleeding
Withhold warfarin, serial INR Vit K1 (phytonadione) Restart warfarin
59
What to do in emergency cases of bleeding
Transfuse fresh frozen plasma | Low dose Vit K