WEEK THREE Flashcards

1
Q

What do you need to assess and monitor in cardiac?

A
Preload
Contractility
After load
Stroke volume
Cardiac Output (CO)
Central Venous Pressure (CVP)
Mean arterial pressure (MAP)
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2
Q

What is Preload?

A

Pressure or stretch exerted on the walls of the ventricle by the volume of blood filling the ventricles at the end of diastole

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3
Q

What is Contractility?

A

The ability of a muscle to shorten when stimulated; in particular the force of myocardial contraction

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4
Q

What is Afterload?

A

The resistance to ventricular contraction; pressure the ventricles have to overcome to eject blood into the circulation

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5
Q

What is Stroke Volume?

A

Volume of blood pumped with each heart beat. 50-100mL/beat

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6
Q

What is Cardiac Output (CO)?

A

The amount of blood pumped by the heart each minute

SV X HR = CO

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7
Q

What is Central Venous Pressure (CVP)?

A

Preload of the right ventricle measured by the CVP

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8
Q

What is Mean Arterial Pressure (MAP)?

A

Average pressure within the aterial system throughout the cardiac cycle 70-90 mmHg

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9
Q

Cardiac Output features:

A

-CO regulated by homeostatic mechanisms
-Regulated in response to stress/disease
-Factors affecting CO
Preload
Afterload
Contractility
-Critically ill patients cannot compensate as well as healthy individuals
-May be unable to >HR to compensate

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10
Q

Preload reduction is related to?

A
  • Volume loss (eg haemorrhage)
  • Venous dilation – hyperthermia/medication
  • Tachycardias – AF/SVT
  • Raised intrathoracic pressure (NIV)
  • Raised intracardiac pressure – Tamponade
  • Body Position can affect preload due to affect on venous return
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11
Q

Increased Afterload is related to?

A
  • Increased ventricular radius
  • Increased aortic impedance
  • Raised intra cavity pressure
  • Negative intrathoracic pressure
  • Increased systemic vascular resistance
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12
Q

Contractility is decreased by?

A

Hypoxia
Ischemia
Drugs ie thiopentone, calcium channel blockers

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13
Q

Increasing Contractility will?

A

Increase myocardial oxygen demand

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14
Q

Assessment of Cardiac Output?

A

Establish Diagnosis, determine therapy and monitor response to therapy
Heart rate and rhythm:
-Continuous cardiac monitoring 4 or 5 lead
-12 lead ECG
Hemodynamic monitoring:
-Non invasive, (TPR&BP, oximetry, UO).
-Invasive intra arterial pressure monitoring
-Invasive CVP
-Pulmonary artery pressure (PAP)
-Cardiac output (CO) thermodilution (measures temperature change)

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15
Q

12 Lead ECG:

A
  • Essential in critically ill patients even if they have no cardiac symptoms
  • All patients who have a cardiac event must have an -ECG ASAP
  • Diagnosis can be made rapidly
  • Treatment can commence
  • Earlier treatment can lead to better prognosis
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16
Q

Pulmonary Artery Catheter?

A
  • Pulmonary artery pressure and wedge waveforms.
  • Filling pressure of the right ventricle (right ventricular preload) is measured through the pulmonary catheter port opening into the:
    1. Superior vena cava
    2. Right atrium
    3. Right ventricle
    4. Pulmonary artery
17
Q

Non invasive Assessment?

A
Visual observation of patient
Pulses
Pulses on monitors do not tell the whole story
Touch your patient
Check pulse for:
-Rate, rhythm, and strength
Pulses to check:
-Radial
-Carotid
-Femoral
18
Q

How to calculate rate:

A

A) Count the number of large boxes between 2 R waves

B) Divide 300 by the number of the large squares, this is your approximate rate.

19
Q

Mophine:

A

-Analgesia
-Opium – binds to receptors in brain, spinal cord and other tissues
-Primary effects on CNS and other smooth muscle
-Peak analgesia 30 – 60 minutes
-Contraindications:
Previous sensitivity, Opioid sensitivity, Bronchial asthma/COPD
-Reactions:
Sedation, Nausea, Vomiting, Constipation, Blurred vision, Local issue irritation (at site of injection)
-Dosage:
Adults – 5 - 20mg every 4 – 6 hourly max , Children – 0.1 to 0.2 mg/kg bodyweight Max 15mgs
-Overdose:
ABC, Oxygen, Naloxone 0.4 to 2 mg intravenously every 2 to 3 minutes as necessary, ?IM Naloxone to support respiratory function

20
Q

Metoclopromide :

A

-Anti emetic
-Accelerates gastric emptying, increases peristalsis
-Acts in 1-3 minutes IV, 10 – 15 mins IM
-Should not be used in patients with prolonged QT – can cause Torsades de Pointes
-Contraindications:
GI Haemorrhage, Obstruction, Perforation, Epilepsy, Extrapyramidal reaction, Oculogyric crisis – Treat with procyclidine
-Reactions:
Headache, Drowsiness, Fatigue
-Dosage:
10mg IMI TDS Slow (1-2 Mins), Children 1-10mg depending on Age