week 9: postpartum complications Flashcards

1
Q

what is the difference between mood changes (postpartum blues) and mood disorders

A

Baby Blues
- within 3-5 days postpartum
- common and temporary emotional state that many women experience in first few days to wks after childbirth.
- mood swings, fatigue, anxiety, weepiness, impaired concentration, diff sleeping or eating
- typically resolves in 2 weeks
- due to hormonal changes
Postpartum Mood Disorders
- affect daily functioning
- more severe, long lasting
- typically emerges within 4-6 wks after childbirth
- persistent sadness, severe mood swings, loss of interest, fatigue, changes in appetite or sleep patterns, feelings of worthlessness or guilt, difficulty bonding

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2
Q

potential risk factors for perinatal mood disorders

A
  • relocation
  • problems w infertility, and anxiety
  • spontaneous abortion
  • change in birth plan
  • NICU admission can be stressful
  • low milk supply
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3
Q

give examples of cognitive domain

A
  • what is diff btwn postpartum depression, baby blues
  • resources for lactation support and possible referrals
  • teaching about baby cues
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4
Q

give examples of affective domain

A
  • support groups
  • mom baby class
  • talking about importance of emotional expression
  • birth story share circle
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5
Q

give examples of behavioural domain

A
  • establishing a routine
  • responding to cues
  • time management
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6
Q

what is the edinburg postnatal depression screen

A

10 statements about individual reflex about each statement in the last week, they choose the responsiveness that looks like what they felt the past week. individuals should be screening themselves.

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7
Q

perinatal depression + treatment options

A
  • characterized by low mood and lack of interest in activities, can be mild to severe
  • intense, pervasive sadness and labile mood swings that last longer than 2 wks

treatment options:
- psychotherapy, CBT, psychodynamic therapy
- antidepressants, anti anxiety meds, and electroconvulsive therapy

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8
Q

health teaching practices for baby blues and postpartum depression

A
  • what are somes signs of baby blues? when should the symptoms go away?
  • what are symptoms of postpartum depression and when can they begin?
  • what are some ways to support wellbeing in the postpartum period?
  • what are warning signs that clients and families should be aware of that require urgent immediate care?
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9
Q

risk factors + s/s for perinatal mood disorders in partners

A

risk factors:
- previous history
- partner w mood disorder
- work or financial stress
- poor social and relationship
- difference w parenting expectations vs reality
s/s:
- fatigue, frustration, anger, irritability

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10
Q

postpartum psychosis

A
  • most severe
  • rare 0.1% of postpartum patients
  • onset tends to show within 2 weeks postpartum, small number develop symptoms later
  • rapid onset of unusual behaviour, hallucinations, paranoia, disorientation, high levels of impulsivity, increase risk for suicide
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11
Q

postpartum psychosis care

A
  • inpatient psychiatric care, antipsychotics, mood stabilizers, benzodiazepines, electroshock therapy
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12
Q

what is a PPH defined as for v birth and c section

A

loss of > 500mL of blood after v birth
loss of >1000 mL after c section

  • any blood loss that has the potential to cause hemodynamic instability
  • blood loss is difficult to estimate and is frequently underestimated
  • amount of blood loss required to cause hemodynamic instability depends on pre-existing condition of the client
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13
Q

described early, acute, primary PPH

A

occurs within 24 hrs of birth

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14
Q

describe late or secondary PPH

A

occurs 24 hrs but less than 6 wks after birth

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15
Q

PPH prevention

A

active management in 3rd stage of labour
- oxytocin after delivery of anterior shoulder
- gentle cord traction (no pulling)
- immediate fundal assessment after birth

if 3rd stage of labour takes longer than 30 min, risk of PPH increase 6-fold

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16
Q

4 T’s of PPH

A

tone - uterine activity
trauma - retained placenta
tissue - lacerations
thrombin - coagulation

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17
Q

describe tone for PPH + risk factors

A

Most common cause of PPH
Lack of uterine tone*
- Soft, spongy, boggy - slow and steady loss of blood
- Higher amount of blood loss than wed expect
Myometrium: smooth muscle

Contractions -> placental arteries - allows blood vessels to constrict

If too much amniotic fluid, carrying multiples: uterus has been overdistended so takes more work to contract

Why someone who had oxytocin during labour is at more risk for PPH: could be hyperstim of uterus, oversaturation of synthetic oxytocin so body becomes desensitized to it

Mag sulfate: prevention of eclampsia, and preterm labour for neuroprotection, tocolytics (diff tocolytics - mag sulfate to relax the uterus) smooth muscle relaxant make sit more difficult to uterus to relax post birth

Inflammation makes it harder for muscle to contract in chorioamnionitis

uterine atony
- marked hypotonia of uterus
- leading cause of early PPH (around 70%)

risk factors
- overdistended uterus (macrosomia, multiples, polyhydramnios)
- high parity
- prolonged labour, oxytocin induced labour
- MgSO4 administration
- chorioamnionitis

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18
Q

describe trauma for PPH + risk factors

A
  • lacerations of birth canal
  • uterine rupture
  • uterine inversion
  • hematomas

risk factors
- operative birth
- precipitous birth

note: hemorrhage related to lacerations should be suspected if bleeding continues despite a firm, contracted uterus

rupture of uterus, uterine inversion (flips inside and out and prolapses)
Placenta is embedded in uterus, and cord pulls too hard, uterine flips (medical emergency)

Firm fundus - trauma not tone

19
Q

describe tissue for PPH + risk factors

A
  • retained placental fragments
  • placenta previa, placenta accreta, increta, percreta
  • higher risk of infection
  • manual removal by OB provider
  • D&C may be required

Retained products: risk of hemorrhage, but more long term risk is infection

Is there abnormal implementation of placenta

Placenta acreta: (goes deeper into uterus layers, invades, and less likely to fully detach)
Body thinks placenta is there, consistently perfuses blood there = bleeding
Recommended c section due to risk of not getting rid of placenta

Dilatino and evacuation if pt is no longer
Misprostolol if they suspect it’s a blood clot and no parts of birth

20
Q

describe thrombin for PPH + risk factors

A
  • idiopathic thrombocytopenic purpura (ITP)
  • von willebrand’s disease
  • disseminated intravascular coagulation (DIC)

When pt is continuing to bleed, bc they have coagulation issues
History of bleeding - may not be known
Issues with ITP, can also be an autoimmune disorder
Hard time having sufficient platelets
Followed by hemotology during pregnancy, and some medications that help with coagulation (transfusinos of platelets can be helpful too)

DIC - imbalance in platelet coagulation, start to bleed somewhere, they send clotting factors there, but then there is a rapid internal hemmorhage that turns into widespread internal bleeding, s/s oozing of blood, BASICALLY BLEEDING EVERYTHING THIS IS SCARY ASF
Low platelets and fibrogen
Risk factors: preeclampsia (HELLP syndrome)

21
Q

signs of hemorrhagic shock

A
  • tachypnea and shallow respirations
  • tachycardia, weak and irreg HR
  • hypotension (late sign)
  • cool, pale, clammy skin
  • urinary output decreasing
  • LOC become less alert and lethargic
  • anxious

can occur rapidly BUT classic signs of shock may not appear until the postpartum client has lost 30- 40% of their blood volume

To consider: bc we know blood volume in preg increases by 40-50%, to manage blood loss during birth. Now hypotensive, tachycardic, you know they have lost 20% of their blood volume, so hypotension is a late sign of PPH

22
Q

describe oxytocin

A

med for PPH
contracts uterus, decreases bleeding
no contraindication for PPH
monitor bleeding and tone

22
Q

management of PPH

A
  • early recognition is critical
  • 1st is evaluation of contractility of uterus
    if boggy, firm massage of fundus
    expression of clots in the uterus
    fundus firm and bleeding continues, assess for the source of bleeding (trauma, thrombin) and treated
  • elim bladder distention
  • admin of meds
  • rapid admin of IV fluids
  • blood transfusion
  • o2 admin
23
Q
A
24
Q

describe misoprostol

A

contracts uterus
contraindicated if have allergy to prostaglandins
monitor bleeding and tone

25
Q

describe carboprost thromethamine (hemabate)

A

contracts uterus
avoid w severe asthma or hypertension
monitor bleeding and tone

26
Q

describe methylergonovine; ergonovine

A

contracts uterus
contraindicated in HTN, PET, cardiac disease
check BP before giving, do not give if greater than 140/90

27
Q

describe tranexamic acid

A

blood clotting and to stop prolonged bleeding
contraindicated if hx of blood clots or taking any anticoagulants
given in PPH for someone w a bleeding disorder

28
Q

management of PPH

A
  • bimanual compression by OB
  • manual exploratin of uterus for retained placenta
  • uterine tamponade (packing or balloon)
  • ligation of arteries
  • uterine compression suturing
  • hysterectomy
29
Q

postpartum infections

A

puerperal infection: anu infection of genital canal within 28 days after abortion or birth
- most common infecting agents are numerous streptococcal and anaerobic organisms
- endometritis
- wound infection
- UTI
- mastitis

30
Q

intrapartum risk factors for infection

A
  • episiotomy or lacerations
  • c-section
  • prolonged ROM
  • chorioamnionitis
  • prolonged labour
  • frequent bladder catheterization
  • internal FHR monitor or IUPC
  • multiple vag exams after ROM
  • epidural
  • retained placental fragments
  • PPH
31
Q
A
31
Q
A
31
Q

clinical manifestations of puerperal infection

A
  • fever
  • foul smelling lochia
  • lethargic
  • abdo pain
  • subinvolution of uterus
32
Q

venous thromboembolic (VTE) disorders

A
  • blood clot(s) inside a blood vessel due to venous stasis and hypercoagulation
  • preg clients have 4-5x increased risk of VTE
  • occurs during preg or postpartum (up to 3 wks)
  • superficial venous thrombosis
  • DVT
  • PE (more common in postpartum vs antenatal period)
33
Q

VTE risk factors

A
  • c-section double the risk for VTE
  • operative vaginal delivery
  • history of VTE, PE, or varicosities
  • obesity
  • maternal age greater than 35 yrs
  • multiparity
  • smoking
34
Q

clinical manifestations of venous thrombosis

A
  • pain and tenderness in lower extremities
  • warmth, redness, enlargement, and hardened vein over the site of thrombosis
35
Q

clinical manifestations of PE

A
  • dyspnea and tachypnea
  • tachycardia
  • chest pain
  • cough and hemoptysis
  • elevated temp
36
Q

POST BIRTH acronym health teaching

A

P - pain in chest
O - obstructed breathing or SOB
S - seizures
T - thoughts of hurting yourself or baby

B - bleeding (heavy, large clots)
I - incision that is not healing
R - red or swollen leg, painful or warm to touch
T - temp of 38 or higher
H - headaches

37
Q

definition of structural disorders following childbirth and risk factors

A

structural disorders of the uterus and vagina related to pelvic relaxation and urinary incontinence

risk factors:
- fetopelvic disproportion
- prolonged labour
- precipitous labour
- precipitous birth

38
Q

describe uterine displacement

A
  • posterior displacement
  • retroversion (tilted posteriorly and cervix rotates anteriorly)
  • retroflexion and anteflexion
39
Q

uterine prolapse

A
  • pelvic stress and strain
  • degrees range from mild to complete
  • s/s related to pelvic relaxation most often appear during perimenopausal period
  • treatment: estrogen therapy, pessaries, surgery
40
Q

cystocele

A

protrusion of bladder downward into vagina when support structures in vesicovaginal septum are injured

41
Q

rectocele

A

is herniation of anterior rectal wall thru relaxed or ruptured vaginal fascia and rectovaginal septum