Week 8 Flashcards

1
Q

Tissue layers of blood vessel

A

A= endothelium
B= tunica intima: endothelium and internal elastic membrane
C= tunica media: muscle and external elastic membrane
D= tunica adventitia

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2
Q

The descending aorta

A

Commences at end of aortic arch continues down abdomen
Formally ends by bifurcating into left and right common iliac arteries
Thoracic part- commences at sternal angle of Louis (T4-5), ends at aortic hiatus T12, supplies arterial blood to muscles of chest wall and spinal cord
Abdominal part- commences at T12, ends at L4 bifurcating into left and right common iliac arteries,supracistal plane of pelvis, vertebral level umbilicus, T10 dermatome Renal arteries branch off at L1/L2, has two phases: suprarenal segment and infrarenal segment

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3
Q

Five major arteries arise from abdominal aorta

A

Coeliac artery: L1
Superior mesenteric artery: L1
Renal arteries: L1/L2
Inferior mesenteric Artery: L3
Left common iliac artery: L4/5
Right common iliac artery: L4/5
Arteries supplying spinal cord, all vertebral levels L1

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4
Q

Arterial tree of lower limb

A

Common iliac artery: L3/L4, umbilicus, supracristal plane
External iliac artery: L5-S1
-internal iliac artery
Femoral artery : inguinal ligament
-profunda femoris runs through adductor canal
Popliteal artery : adductor canal
Tibial arteries: adductor hiatus
-anterior, posterior : lower border of popliteus
Dorsalis pedis, post tibial artery

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5
Q

Femoral artery

A

Begins at lower border of inguinal ligament
Passes inguinal ligament at mid point
Ends at apex of femoral triangle
Ends by entering adductor canal
The apex of femoral triangle is an opening in adductor Magnus - adductor canal/ subsartorial canal, hunters canal
Then becomes popliteal artery

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6
Q

Popliteal artery

A

Starts at the opening adductor canal and ends at adductor hiatus
Then enters diamond shaped anatomical space at back of thigh- Popliteal fossa
In popliteal fossa it gives off a series of geniculate arteries to supply the knee joint
Popliteal artery ends at lower border of popliteus muscle bifurcating into anterior and posterior tibial artery

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7
Q

Anterior tibial artery

A

Pulses often palpated at its level known as dorsalis pedis
Points of best palpitation are: above the navicular bone, medial to the tendon of extensor hallucis longus

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8
Q

Posterior tibial artery

A

Larger of the terminal branches of the popliteal artery
Descends deep to soleus then becomes superficial in lower third of the leg
Passes behind the medial malleolus between tendons of flexor digitorum longus and flexor hallucis longus
Below the ankle it divides into medial and lateral plantar arteries which constitute principal blood supply to the foot

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9
Q

Venous tree of the body

A

Commences at levels of post capillary venules
Ends when venae cava access the right atrium of the heart

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10
Q

Normal drainage venous blood

A

Longitudinal conduction is that venous blood is conducted from post capillary venules- venules-small veins- larger veins- right atrium
Low pressure system
Has evolved to overcomes an element of the circulatory system where there’s no pump or driving force, tendency of venous blood to pool in lower extremities as a result of gravitational pull

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11
Q

Adaptations venous system

A

One way valves that prevent back flow of blood to promote venous return to heart
Systemic veins with thin walls enabling them to collapse and obliterate the lumen as the veins collapse they passively pump blood past one valve at a time
Musculovenous pump

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12
Q

Classes of veins

A

Superficial veins
Deep veins
Perforating veins
Accompanying veins: vaso vasorum, venae commitante(accompany arteries)
Venous blood flows form superficial to deep veins via perforating veins

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13
Q

Sites for venepuncture and saphenous cut down

A

The great saphenous vein (branch of femoral vein) is accessible through:
A hands breath to the medial edge of patella
In front of medial malleolus

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14
Q

Varicose veins

A

Perforating veins drain venous blood from superficial to deep veins
If incompetence in valves preventing backflow in perforating veins- venae stasis
Resulting in varicose veins, clotting in stagnant venous blood, raising probability DVT

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15
Q

Nerve supply to lower limb

A

Lumbar and sacral spinal segments
Lumbar plexus: L1, L2, L3, half L4
Sacral plexus: half L4, L5, S1,S2, S3 S4
The lumbar contribution to sacral plexus is the lumbosacral trunk

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16
Q

The lumbar plexus

A

Forms behind psoas major
Nerves emerge form medial or lateral border psoas major
Lateral: femoral nerve L2-4, iliohypogastric, ilioinguinal, lateral cutaneous nerve of thigh
Medial: obturator nerve L2-4, lumbosacral trunk half L4, L5

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17
Q

The sacral plexus

A

Plexus forms in pelvis cavity
Lies in relation to piriformis
Composed of lumbosacral trunk half L4 and L5
Sacral spinal segmental outflow
Supplies nerves to pelvic region, gluteal region, perineal region, lower limb via sciatic nerve

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18
Q

Sensory supply lower limb

A

Front limb is supplied largely by lumbar segments
Back limbsuppleid mainly sacral segments
Saddle area is sacral segments
Perineal area is sacral segments

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19
Q

Axial lines

A

Line of junction of two dermatomes supplied from discontinuous spinal levels known as axial line
Limbs have anterior and posterior axial lines
Axial lines mark centre of either pre axial or post axial territories of the limbs
Boundaries between pre axial and post axial compartments are marked out by veins

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20
Q

Sensory nerve territories

A

Sensory divisions of terminal nerves of lumbar plexus supply skin in territorial domains
Not dermatomes

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21
Q

Visual assessment of the musculoskeletal system

A

Appearance of skeletal muscles in the body
Symmetry of muscle bulk between left and right sides
Presentation of the limbs and other muscle systems
Posture
Gait etc

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22
Q

Definition of motor unit

A

Constitutes from the alpha motorneuron and all muscle fibres it supplies
It comprises:
1 alpha-motor neurone
Extrafusal muscle fibres it supplies
Minimal functional unit of the motor system

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23
Q

Definition of a reflex

A

Involuntary, unlearned, repeatable, automatic reaction to a specific stimulus that doesn’t require brain to be intact
Reflex arc:
A receptor
An afferent fibre
Integration centre
Efferent fibre
Effector organ

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24
Q

Role the muscle stretch reflex play in neurology

A

Template neural circuit from which all motor circuits are built
Minimal neural circuit that underlies all movements of muscles of body
Neural circuit that sets all motor tone of the body

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25
Q

Muscle Stretch reflex

A

Stretch activated reflex contraction of skeletal muscle
When a muscle is not contracted it relaxes
A relaxing muscles is effectively lengthening
When muscle length receptors detect stretch they fire action potentials via afferent axons to keep CNS appraised of muscle length at all times

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26
Q

How does muscle stretch reflex work

A

Action potentials from muscle length receptors are sent to:
The brain via dorsal column
The cerebellum via spino-cerebellar tracts
A copy of that signal is also sent directly to spinal motorneurones
Results in reflex recruitment of motorneurones

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27
Q

Anatomy of stretch reflex

A

The spinal reflex pathway involves two neurones
A stretch receptor afferent
Efferent neurone
1 synapse in lamina IX of spinal cord

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28
Q

Motor tone

A

In a normal awake neurological status lower motorneurones tonically supply muscles with background electrical impulses
These lead to background minimal contraction of the muscle
This minimal contraction gives the muscle a small amount of force called muscle tone/motor tone
Motor tone allows us to maintain body posture and hold heads up

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29
Q

Muscle tone in babies

A

Present but low in-utero
Suppressed in the new born
Returns within months after birth
Return of muscle tone in the new baby informs of the absence or presence of birth injury of the brain

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30
Q

When is muscle tone inhibited

A

During deep (REM) sleep in all muscles except:
Muscles of breathing
Extraocular muscles
Urinary sphincter
Anal sphincter

31
Q

What happens when muscles generate too little tone

A

Body becomes limp
Body is unable to support its weight
Normal body posture is lost
This is anything between hypotonia and atonia depending on severity

32
Q

What happens when muscles generate too much tone

A

Muscles become stiff
Reciprocal inhibitory relationships between agonists and antagonists is disrupted
Agonists and antagonists become equally as stiff, simultaneously
Joints become stiff
Normal body posture altered
Ability to move affected limb lost
Fluidity of movements of joints will be lost
Spastic paralysis will results

33
Q

Constellation of Lower motor neurone signs

A

Flaccid muscle weakness
Hypotonia or atonia
Hyporeflexia or areflexia
Denervation muscle atrophy
Fasciculations acute phase
Muscle wasting

34
Q

Three types of movement

A

Involuntary or reflex (simplest)
Voluntary (most complex)
Rhythmic ( a combination of reflex and voluntary)
All require muscle contraction

35
Q

Centre of gravity

A

Moves with unconscious (reflex) change in posture or with voluntary goal directed movement but must remain within the area bounded by our feet to avoid falling
The point at which the whole weight of an object can be considered to act and therefore at which all parts of an object are in balance. The position of the centre of gravity varies according to the shape of the object

36
Q

Postural muscles

A

Need to maintain tone in order to stay in a constant state of partial contraction and thus counteract gravity
Corrective reflexes to maintain stability and position
A role for receptors and somatic reflexes

37
Q

Sensory feedback arises from

A

Visual system
Vestibular system- sense of balance and spatial orientation
Skin receptors
Proprioceptors

Skin and proprioceptors—> somatic sensation (somesthesia)
Visual and vestibular—> Vestibulo-ocular reflex and optokinetic reflex

38
Q

Muscles spindles

A

In parallel with muscle fibres, undergo same length change as muscle
Measure length
Intrafusal muscle fibres lying in parallel within extrafusal muscle fibres, greatest density in small muscles serving fine movement and also deep muscles of neck
Innervated by sensory and motor nerves
Detect dynamic and static changes in muscle length essential for detection of muscle length and rate of change of length
Central region: specialised non contractile fibres, primary and secondary sensory afferent endings
Peripheral region: innervated by gamma unmyelinated motor neurones does not contribute to overall force adjusts sensitivity of spindle

39
Q

Golgi tendon organs

A

In series with muscle fibres measure tension
Sensory stretch receptors that are found in tendons near junctions with muscle fibres and respond to tension changes in the muscle they are innervated by myelinated Ib fibres
Ib fibres go to ventral horn and activate interneurons which inhibit (glycinergic) alpha motor neurons opposite muscle spindle but with higher threshold than for muscle spindle
Stimulation of Golgi sensory neuron fibres causes a reflex inhibition in the efferent signal to skeletal muscle so loss in contraction strength, muscle relaxes
Believed to assist in the fine control of muscle length playing a role in the maintenance of posture

40
Q

Proprioceptors

A

Are sensory receptors found in muscle that are important for reflex control of muscle length and tension

41
Q

Somatic motor reflexes

A

Stereotypic movements elicited by specific sensory stimuli
Characteristic somatotopic organisation and large dendritic tree

42
Q

Afferent innervation muscle spindle

A

Primary afferents- innervate central region and respond primarily to velocity of lengthening
Secondary afferents- innervate ends of central region and respond to static length
During a stretch reflex increased firing by the sensory neuron increases signalling by the alpha motor neuron causing the muscle to contract

43
Q

Role of gamma efferents muscle spindle

A

A passive stretch activates gamma efferents and can initiate corrective reflex
If alpha motor axons activated without firing of gamma efferents the spindle loses activity when the muscle contracts - skeletal muscle contracts muscle spindle squashed switched off losing sensitivity of stretch
Alpha-gamma coactivation maintains spindle function when muscle contracts, activated independently
Gamma motor neurons contract end of muscle spindles

44
Q

What is gait

A

Walking
Jogging
Skipping
Running
Sprinting

Children learn to walk between 9-18 months usually 12 months

45
Q

Locomotor unit

A

11 joints
Lumbar spine
2 hip joints
2 knee joints
2 ankle joints
2 subtalar joints
2 metatarsal phalangeal joints

46
Q

Assessment of gait

A

Assess symmetry
Speed: pain slows gait down
Top to bottom analysis:
- do arms swing naturally
-is the lumbar spine hunched or upright and stiff
- are pelvis and hips controlled
-does hip flex and extend normally
-does knee bend normally
-assess ankle and foot position
-step width
-toe out angle- pigeon toe

47
Q

Gait cycle

A

Stance and swing phases
Starts with initial contact- heel strike
A complete gait cycle is a single stride from a heel strike to next heel strike of same limb
A step is half a stride
Stance phase: when the foot that made the heel strike is on the floor
Swing phase: the foot that made the initial hell strike is in the air
Gait cycle then ends with heel strike of same foot
2 double limb stances in cycle:
-beginning and end of stance phase
As walking speed increases period in double stance phase decreases until its omitted in running
Single support: one foot on the ground bearing body weight

48
Q

Function of the stance phase

A

Shock absorption
Stability
Propulsion

49
Q

Function of the swing phase

A

Ground clearance- need enough hip flexion, knee flexion and ankle Dorsiflexion
Step length

50
Q

Sub phases of gait cycle

A

Swing phase: initial swing, mid swing, terminal swing phase
Stance phases:
- initial contact heel strike event: hip is flexed, knee extends ankle is neutral
-loading response; hip flexes, knee flexes, pelvis dips to absorb shock of impact, ankle plantar flexes then dorsiflexes into midstance
-mid stance phase: ankle extends, knee straightens out to allow swing leg to clear ground
-terminal stance phase: extended limb is extended further through hip into pre swing as initial contact is made of other side, heel raises and push off occurs
-pre swing- toe off

51
Q

Rockers of gait

A

There are 4 rockers (pivots):
- the heel rocker
- the ankle rocker
- the forefoot rocker
-the toe rocker
The whole leg rocks over the foot using these pivots in different stages
This is called tibial progression
When the rockers cease to function properly you can perform physiotherapy to mitigate effect

52
Q

Shock absorption

A

Important function of stance phase
Before heel strike the body is in free fall
The shock absorbing response of the foot, ankle, knee and hip reduce intensity of impact
Knee flexion is the largest shock absorbing mechanism
Weight loss is also benefit joint function as it reduces shock

53
Q

Energy cost gait

A

Normal walking uses 40% of maximal aerobic capacity
Reduce energy expenditure:
-selective muscular control is crucial to efficiency of gait
-centre of mass control

54
Q

The major determinants of gait

A

Maintain body centre of mass at constant height above ground to reduce energy expenditure
6 major displacements:
- pelvic rotation
-pelvic tilt
- stance knee flexion
- foot and ankle mechanisms
-tibiofemoral angle
-pelvic lateral displacement

55
Q

Springing pendulum theory of human gait

A

Pivoting over a stiff stance leg
Passive movements of the swing leg
Propulsion from the ankle propelling the swing leg
Rotation of the hips in axial plane to increase stride length
Tilting of the hips in coronal plane to improve balance during stance

56
Q

Muscular anatomy of walking stance phase

A

Stance phase:
Pelvis- stability tensor fascia latae
Hip- flexion (iliopsoas, sartorius, rectus femoris) extension (semitendinosus, biceps femoris, gluteus maximus, adductor magnus)
Knee- flexion( rectus femoris, vastus lateralis and vastus medialis eccentric contraction),
Ankle: plantarflexion (gastrocnemius, soleus), Dorsiflexion( tibialis anterior, extensor digitorum longus, gastrocnemius, soleus)

57
Q

Muscular anatomy of walking swing phase

A

Pelvis: stabilisation tensor fascia latae
Hip: flexion (semitendinosus and biceps femoris)
Knee: flexion (sartorius), extension ( rectus femoris, vastus lateralis, vastus medialis)
Ankle: Dorsiflexion (tibialis anterior, extensor digitorum longus), plantarflexion (gastrocnemius, soleus)

58
Q

What influences gait

A

Bony Skeleton
Muscle function
Other soft tissues
Other organ systems
Trauma
Pain
Inflammation
Congential abnormality
Neurological function
Weakness
Psychology

59
Q

Antalgic gait

A

To counteract pain
Classic limp
There is decreased time spent in stance phase to avoid weight bearing
Many causes

60
Q

Circumduction gait

A

Affected leg excessively abducts in swing phase
Cause long or stiff leg

61
Q

Spastic gait

A

On affected side: Circumduction hip, medial rotation leg, feet are dragged, stiff knee and hip increased swing phase and decrease stance phase
Cause: neurological upper motor neurone pathology, stroke or cerebral palsy

62
Q

Ataxic gait

A

Unstable gait pattern
Similar to drunken gait
Uncoordinated movement
Broad base gait- provides stability
Cerebellar

63
Q

Trendelenburg gait

A

Weak hip abduction
Hip is not stabilised in stance phase so pelvis tilts down
Persons weight is then used to maintain pelvis position resulting in waddling

64
Q

Toe walking gait

A

Common
Can be habitual

65
Q

Foot drop gait

A

High step to clear the ground
Whole leg being lifted with excessive hip flexion to keep foot clear off the ground in the swing phase
Knee also flexes excessively
Ankle is usually plantar flexed
Cause: polio, spina bifida

66
Q

Parkinsonian gait

A

Hurry along with short fast steps (shuffling gait)
Stooped posture
Walking is on forefoot rather than heal
Lack of arm swing
Cause: Parkinson’s

67
Q

Crouch gait

A

Occurs with weakness of quadriceps
Increased Dorsiflexion of ankle
Excessive knee flexion
Cause: cerebral palsy

68
Q

Bizarre gait

A

Psychogenic cause

69
Q

Clumsy gait

A

Caused: dyspraxia

70
Q

Rapid clinical gait analysis

A

Is there pain
Is there asymmetry
Are there clues to a diagnosis
Head to toe observation of joint movement

71
Q

Assessing a acutely limping child

A

Consider broad differential
Rule out: infection, tumour, non-accidental injury
Be systematic: history, examination, investigations
Have a clear diagnosis and plan

72
Q

Red flags in gait assessment of a child

A

Duration>7 days
Unusual story
Severe localised pain
Change in bowel/bladder
Unable to weight bear
Nocturnal pain
Systemic symptoms
Wasting fever
Rash
Kocher criteria: signs for septic arthritis:
- non weight bearing
-ESR>40 con
-WCC> 12
-Temp>38.5

73
Q

Physical examination gait in child

A

Musculoskeletal system
Systemic exam