week 7, GUT Flashcards

1
Q

What is digestion?

A

The gastrointestinal process of breaking down food and absorbing its components into the body.

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2
Q

What are the three ways energy is delivered into the body?

A

Lipids (fats), Amino acids (breakdown of proteins) and glucose

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3
Q

How is energy stored in the body (3).

A

Fats, glycogen and proteins.

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4
Q

wht are the three phases of energy metabolism?

A

cephalic phase-prepatory phase, anticipation of food.
absorbative phase: energy in bloodstream.
Fasting phase: unstored energy has been used and organism uses energy reserves.

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5
Q

What does insulin have to do with anything?

A

Insulin is released during Cephalic and absorbative phases, and converts glucose to glycogen, and fat n amino into proteins.

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6
Q

What is glucogen?

A

Glucogen is released in fasting, when insulin is low. this causes gluconeogensis, when protein gets converted to glucose. high glucogon stimulates conversion of fatty acids to ketones. stops storage of fat.

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7
Q

Lipostatic theory

A

theory that every person has a set point for fat, and deviations from this mean ppl eat less or more so they return to the set point, developed bc body weights of adults remain fairly constant (lol, they don’t)

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8
Q

glucostatic theory

A

eating is regulated by blood sugar levels bc our brain needs it.

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9
Q

Set point assumption

A

that after a meal, energy resources hit their set point and then decline, so we get hungry again. incorrect.

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10
Q

problems with set point theory

A

doesn’t account for unpredictable food suppy, obesity, eating disorders, taste, learning, social influences.

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11
Q

positive incentive perspective

A

we eat bc it is anticipated pleasure

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12
Q

settling point theory

A

body weight tends to drift around a natural settling point, the level at which various factors that infleunce body weight achieve an equilibirum, remains stable. so, you can permenently change your body weight, but it has to be a permement change that does it (stopping eating fatty food forever etc).

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13
Q

what happens to glucose levels before eating

A

they drop sharply, and if the meal isn’t served they go back to previous leves.

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14
Q

Lateral nucleus of the hypothalamus

A

controls lack of eating

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15
Q

ventromedial nucleus of hypothalamus

A

uncontrolled eating

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16
Q

ghrelin

A

hunger hormone, stimulates eating by singalling hypothalamus

17
Q

prader-willi syndrome

A

genetic disorder, resulting in insatiable hunger, little or no satiety, and an exceptionally slow metabolism.

18
Q

anorexia nervosa

A

healththreateningweight loss, distorted body image. In ~4% of cases, complications from starvation result in death, and there is a high rate of suicide.

19
Q

Bullimia nervosa

A

periods of not eating interrupted by bingeingfollowed by efforts to immediately eliminate the consumed calories from the body by voluntary purging or by extreme exercise. Bulimia sufferers may be obese or of normal weight. If they are underweight, they are diagnosed with binge-eating/purging anorexia. The mortality rate is ~4%.