week 7 Flashcards

1
Q

Vascular System:
Comprised of:
Depends upon:

A
Comprised of:
Arteries and arterioles
Capillaries
Veins and venules
Lymphatic vessels
Depends upon:
Cardiovascular system
Systemic blood vessels
Circulating blood (volume/viscosity)
Nervous & endocrine system activity
Metabolic tissues needs
… lymphatic system
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2
Q

Vascular System:
Function
How?

A

Function:
To meet the circulatory needs of the tissues
Constantly changing according to metabolic requirements
When supply doesn’t meet demand = ischaemia

How?
Blood flow (from high pressure to low pressure)
(from arterial [~100mmHg] venous [~4mmHg])
Flow rate = ΔP/R (pressure difference ÷ resistance)
Capillary Fluid exchange
Hydrostatic (blood pressure)/osmotic pressures (proteins)
Any extra between arterial supply & venous reabsorption lymph

Imbalance = oedema

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3
Q

Peripheral Blood Flow

A

Flow rate = ΔP/R! Affected by:-
Hemodynamic resistance
Blood viscosity
Vessel diameter

Regulation of peripheral vascular resistance
CNS (sympathetic = vasoconstriction)
Hormonal (noradrenaline/adrenaline/angiotensin)
Chemicals/proteins/hypoxia/pH

Movement of fluid across the capillary wall:
To meet metabolic needs
Hydrostatic & osmotic force

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4
Q

Pathophysiology of Vascular Disorder:
Characterised by reduced blood flow
Effect depends on imbalance of demand/supply
Inadequate supply = ischemia

A
  1. Heart failure
    Left
    Pulmonary congestion & reduced cardiac output
    Poor arterial blood supply
    Right
    Venous congestion & ? reduced cardiac output
2. Alteration in vessel supply
Lymph/arterial/venous
Intact 
Rupture – aneurysm/trauma
Patent 
Atherosclerosis/thrombus/valve issues
Responsive 
Vasospasm/arteriosclerosis/aging changes

The ability to develop collateral supply can greatly decrease the ischaemic damage

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5
Q

Peripheral Arterial Disorders

PAD

A
  • Intermittent claudication
  • Pulses diminished or absent
  • Oedema – None/minimal
  • Skin changes: Trophic – cold/dry/shiny/hairless/thick opaque toe nails
  • Pallor when elevated
  • Red when dangling (dependent rubor)
  • Ulcers – tips of extremities/ painful/deep/circular/pale to black base or dry gangrene
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6
Q

Peripheral Venous Disorders

PVD

A
  • Pain: aching to cramp like, relieved by activity/elevation
  • Pulses usually present
  • Oedema – present/increases at the end of day
  • Skin changes: warm/thick/ tough/darkened/? dermatitis
  • Ulcers – medial malleolus/ pain variable/ superficial/irregular border/granulation base
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7
Q

Intermittent Claudication

A
Muscular/cramping (ischaemic) type pain
Precipitated by exercise
Resolves within 10 mins of rest
Reproducible
Area depend on which vessel affected

Lack of blood supply of oxygen/nutrients when increase in demand
Caused by arterial supply failure
Anaerobic cellular metabolism

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8
Q

Assessing peripheral pulses

A

Diagnostic:
Doppler studies
Exercise testing ( with ankle systolic blood pressure)
Duplex ultrasound/CT or MRI (with/without angiogram)
Angiogram
Venous studies
Lymphatic studies

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9
Q

Ankle Brachial Index FYI (with hand-held doppler)

A

Measures degree of PAD

Quantifies stenosis
Systolic BP both arms
Use highest reading
Systolic BP both ankles

Divide ankle systolic/
brachial systolic pressure

Normal ~ 0.90 – 1.30
Mild-mod. – 0.50 – 0.95
Mod-severe – 0 – 0.50

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10
Q

Common Sites of Atherosclerotic Obstruction

A
Coronary arteries
Carotid arteries
Aortic bifurcation
Iliac and common femoral arteries
Distal popliteal artery
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11
Q

Risk Factors for PAD: Modifiable

A
Smoking
Diet
Hypertension
Hyperlipidaemia
Diabetes
Obesity 
Stress
Sedentary lifestyle
C-reactive protein 
    (inflammation)
Hyperhomocysteinemia
	(clotting factor)
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12
Q

Risk Factors for PAD: Non Modifiable

A

Age
Gender
Familial predisposition/genetics

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13
Q

Nursing Process: The care of the patient with PAD — assessment

A

Health history
Medications
Risk factors

Clinical manifestations of arterial insufficiency
Claudication and rest pain
Colour changes
Weak or absent pulses
Skin changes and skin breakdown
Arterial/venous/lymphatic (medical) diagnosis made

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14
Q

Nursing process: The care of the patient with PAD — diagnoses

A

Altered peripheral tissue perfusion
Chronic pain
Risk for impaired skin integrity
Knowledge deficient

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15
Q

Nursing process: Altered peripheral tissue perfusion — planning

A
Major goals include:
Increased arterial blood supply 
Promotion of vasodilatation
Prevention of vascular compression
Relief of pain
Attainment or maintenance of tissue integrity
Adherence to self-care programme
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16
Q

Nursing process: Altered peripheral tissue perfusion — implementation

A

Exercises and activities:
Walking (stop with pain – gradually increase tolerance)
Graded isometric exercises.
Promote circulation & development of collateral circulation
Specialist advice before commencement
-Contraindications include leg ulcers/cellulitis/thrombotic occlusions

Positioning strategies

Temperature
Effects of heat (vasodilation) & cold (avoid)

Stop smoking

Stress reduction (counselling)

17
Q

Peripheral Arterial Disease (PAD)

Complications

A

Atrophy of skin/nerves /muscles

Delayed healing/wound necrosis/infection/gangrene

18
Q

PAD – Medical treatment

A

Medical (relieve symptoms/improve arterial supply)
Risk factor modification (smoking/diet/exercise)
Drugs (aspirin …/prostaglandin)
Management of diabetes/hypertension/obesity
Refer to podiatrist/physio/rehab. speciallist

Surgical (relieve symptoms/improve arterial supply)
PTBA (balloon angioplasty/stent)
Peripheral arterial bypass graft (femoral-popliteal)
Endarterectomy/patch graft
Amputation (last resort)

19
Q

Nursing Management – Peripheral arterial bypass graft

A
Vital signs/neurovascular observations (pulses)
Graft patency obs (doppler)
Ankle brachial index (8hrly)
IDC 1hrly measures
Complex invasive line care
Wound care (bleeding/haematoma)
Elevate extremity/gently exercise
Graduated compression stockings(?)
Analgesia
Discharge advice/patient education
Care of co-morbidities
20
Q

Arterial Aneurysms

A

An aneurysm is a localised sac or dilation formed at a weak point in the wall of the aorta

21
Q

Aortic Aneurysm: Classified

A
Classified:-
Type 
True – wall of artery forms the aneurysm
False – disruption of all artery layers (trauma/infection)
Shape
Position (thoracic 85%)
22
Q

Aortic Aneurysm: Risk factors: (exact pathophysiology unknown)

A
Atherosclerosis in most cases
Genetic link/congenital vessel wall weakness
Trauma/disease/inflammation
After formation 		tends to enlarge
Smoking/hypertension
23
Q

Aortic Aneurysm: Clinical manifestations

A

Clinical manifestations (often asymptomatic/varied)
Pain/throbbing/”beating”
Ascending – voice hoarseness/dysphagia/venous return interruption (distended neck vessels/oedema)
Thoracic – deep spreading chest pain
Abdominal –back pain/bowel pressure/bruit/palpable
From thrombosis of smaller vessels

24
Q

Aortic Aneurysm: Diagnosis

A

Routine examination
Pulsatile mass in 80% cases/bruit
Chest x-ray – calcification/widening of aorta
ECG to rule out MI
Echocardiology – aortic insufficiency
Ultrasound/ CT scan/MRI scan
Angiography – useful to assess other vessel involvement

25
Q

Medical Management - Aortic Aneurysm

A

Depends on symptoms/prognosis/position/ patient co-morbidities
High rate of surgical death/complications
Prevent rupture/early detection essential
Conservative (small aneurysm/poor surgical risk)
Risk factor/behaviour modification (hypertension/smoking/sedentary lifestyle)
Manage co-morbidities & other atherosclerosis

Operative
Surgical - graft or primary closure
Endovascular – femoral/iliac artery catheter accessed
minimally invasive/strict criteria

26
Q

Open Repair of an Ascending Aortic Aneurysm- Postop Nursing Care

A

Complex major surgery/surgical ICU/HDU area
Patient may have complex co-morbidities
Vital signs/neurovascular – frequent/regular
Monitoring for signs of occlusion/thrombosis/emboli
Monitor all systems (respiratory/renal – IDC 1 hrly)
Monitor T° 4 hrly (graft rejection)
Assess site/wound – haematoma/ooze/inflammation
Often first or second day post op walking
Surgeon/facility dependent

27
Q

Thromboembolism(VTE) Prevention & Care

A

“Deep vein thrombosis (DVT) is one of the most common, preventable complications of surgery

28
Q

DVT - Prevention/prophylaxis

A

often medically prescribed - though not always
nothing stopping us from initiating interventions (Dr have you considered …)
Prevention is better than cure!
Early mobility/ambulation/SOOB
Bed exercises/deep breathing & coughing/alter position

Compression stockings/(good for distal DVT but what about proximal)? –
Fit correctly!!!!
(some contra-indications – see the readings)
Pneumatic compression devices - SCDs/ICDs
(some contra-indications – see the readings)
Drugs
Anticoagulants - Heparin(LMWH)/enoxaparin/warfarin (phasing out)/others

29
Q

DVT treatment

A

Treatment (of diagnosed DVT)
Prevent further growth & fragmentation (into PE)
Bed rest with limb elevation
Anticoagulants (prevent further thrombi)
Thrombi resolves naturally (not through anticoags)
Then mobilise with quality stockings (not the cheapies)
Drug Therapy
Heparin – IV infusion/SCI/Warfarin/more modern types
Thrombolytics
Previously (with DVTs) only for limb threatening situations
Now being trialled to ascertain treatment as more routine
Surgery (uncommon)
Vena cava filter
Open thrombolectomy