Week 6 - Abdominal Flashcards

1
Q

what are the 5 factors that effect nausea

A
Anxiety
Increased Intracranial Pressure (ICP)
Movement
Gastric changes (stasis, constipation, obstruction)
Biochemical Changes (e.g. drugs)
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2
Q

what are the The Emetic Pathway

A

Vomiting Centre

  • Cerebral Cortex
  • Pressure Receptors
  • Vestibular Nucleus
  • Gastrointestinal Tract
  • Chemoreceptor Trigger Zone
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3
Q

what medication can induce nausea

A

Histamine Antagonists
Acetylcholine (muscarinic) Antagonists
Serotonin Antagonists
Dopamine Antagonists

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4
Q

WHY DO WE NEED a Digestive System?

A

All organisms must obtain nutrients from their environment to survive
Single-cell organisms – no need for GIT
More complex organisms – more cells 
need organisation and coordination

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5
Q

what is the Gastrointestinal tract (GIT)

A

A hollow muscular tube starting from the oral cavity
Passes through the pharynx, esophagus, stomach & intestines to the rectum and anus
Accessory organs assist by secreting enzymes to help break down food into its component nutrients
The salivary glands, liver, pancreas & gall bladder have important functions
Food is propelled along the length of the GIT by peristaltic movements of the muscular walls.

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6
Q

what are the primary Functions of the GIT

A

Ingestion

Digestion

Absorption

Elimination

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7
Q

what function does the mouth serve

A

Oral cavity – teeth and tongue: mechanical processing, moistening, chemical digestion starts
Salivary glands – enzymes and lubrication
Pharynx – muscular propulsion of materials into oesophagus

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8
Q

what are the common disorders of the mouth

A

Stomatitis
Primary or secondary
Can affect any or all parts of the mouth
Symptoms of pain, halitosis, swelling, bleeding, ulceration

Other common disorders of the mouth?

Oral Cancer

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9
Q

what are the common disorders of the oesophagus

A

Gastro –Oesophageal Reflux Disease (GORD)
Definition – the backward flowing of gastric contents into the oesophagus
Pathophysiology
Contributing factors e.g. Increased gastric volume; positioning & increased gastric pressure
Manifestations
Complications

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10
Q

what is the HIATAL HERNIA

A

this is when part of the stomach pokes through the diaphragm

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11
Q

name of stomach areas

A

refer to image on power point Topic 5 abdominal bowel considerations slide 15

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12
Q

what are the disorders of the stomach

A
Gastritis
Definition – inflammation of the stomach lining as a result of irritation of the gastric mucosa.
Two types – acute and chronic
Manifestations
Nursing management
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13
Q

key points of stomach cancer

A

Incidence increases with age
Risk factors – H. Pylori infection –35%-89%
Most common type – Adenocarcimona
Metastatic spread to liver lungs, ovaries and peritoneum
Manifestations – few – similar to gastritis
Treatment – medical / oncology; gastrostomy tube
Nursing intervention

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14
Q

Disorder of the Bowel

A
Bowel Obstruction
Intestinal obstruction is failure of intestinal contents to move through the bowel lumen. 
Can be in the small or large intestine.
Mechanical or functional 
Manifestations
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15
Q

Disorders of the Liver

A
Hepatitis
Inflammatory condition of the liver
Generally caused by one of 5 virus’s
Mode of Transmission
Manifestations: Acute hepatitis
Manifestations: Chronic hepatitis
Can be brief, severe & life threatening
Liver can regenerate tissue - but can lead to cirrhosis & chronic liver dysfunction and cancer
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16
Q

Disorders of the Pancreas

A
Pancreatitis
Inflammation of the pancreas
 Acute : Manifestations
Chronic : Manifestations
Causes – alcohol main cause but not the only one
Nursing interventions
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17
Q

Pancreatic Cancer

A
Accounts for 1/6 of all cancer deaths 
5 year survival rate of less than 6 %
Often advanced on diagnosis – only 30% offered curative treatment
Risk Factors
Manifestations
Nursing Management
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18
Q

what is the GALL BLADDER

A

Site of bile storage
Bile is released into the duodenum in response to the hormone cholecystokinin (CCK)
CCK is produced in the duodenum

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19
Q

Cholecystitis

A

Cholecystitis

  • Commonly caused by gall stones lodged in the common bile duct
  • May produce jaundice
  • Extremely painful; often mistaken by sufferers for a heart attack
  • Episodic in nature; acute episodes often brought on by an influx of high-fat food
  • Typical patient fits the ‘three Fs’: ‘fair’, ‘fat’ and ‘forty’
  • Affects 20% of people over 40 years; more in women
  • Treated surgically
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20
Q

how can Common Disorders of the GIT occur

A

Results from food intolerance, psychological distress or increased gastric juices

Signs &symptoms - constipation & diarrhoea, abdominal distention & pain, mucous in stools

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21
Q

Gastroenteritis

A

Inflammation of the stomach and intestines
Causes
Manifestations
Can be dangerous due to loss of fluids and electrolytes
Nursing Management

22
Q

Appendicitis

A

Inflammation of the appendix
Manifestations
Complications
Nursing Management

23
Q

Inflammatory Bowel Disease

A

Ulcerative colitis and Crohn’s disease are caused by abnormal activation of the immune system in the intestines
Ulcerative colitis : Manifestations

Crohn’s disease : Manifestations

About 25 to 40 % of ulcerative colitis patients must eventually have their colons removed because of massive bleeding, severe illness, rupture of the colon or risk of cancer.

Nursing Management

24
Q

what is a STOMAS

A

Stoma is the Latin term for
Opening, and it is artificially made by a surgeon

There are 3 types of “ostomy” 
Colostomy
Ileostomy
Urostomy 
Name usually depends on the 
site it is situated.
25
why are stomas necessary
Approximately 36,000 Australians receiving ostomy support. Newborns may require a stoma due to congenital deformities. They may be necessary due to trauma or disease They may be permanent; or temporary
26
what are the clinical observations a nurse should observe
Observe for signs of nutritional alteration. General appearance- alert/responsive or listless/confusion Muscles- tone, development, presence of fat CNS- reflexes, attention span, sensation CVS- HR, BP, rhythm GI – appetite, digestion Hair – lustre, scalp Skin/nails – colour, dry, scaly Eyes – lustre, membranes Oral cavity – mucous membranes, teeth, lips
27
what are the relevant health information
Past medical/ health history chronic and acute Surgical procedures Medications & Allergies Developmental problems – growth, birth history, milestones, functional abilities if applicable Obstetric and reproductive history Genito-urinary problems Substance use- Alcohol and cigarettes
28
what is the importance of the renal system
``` Prevent build up of waste • Prevent build up of fluid • Maintain electrolyte balance • Produce hormones for BP regulation • Stimulate production of red blood cells • Maintain strength of bones ```
29
GLOMERULAR FILTRATION | RATE (GFR)
Glomerular Filtration Rate (GFR) - the rate at which fluid filters from the glomerulus into the Bowman’s capsule • GFR provides a measure to assess renal function • Creatinine clearance is used in clinical practice to estimate GFR • Normal GFR 90-140 ml/min (men), 80-125 ml/min (women)
30
Renal system - Terminology
``` Renal • Nephrology • Urology • Glomerular filtration rate • Diuretic • Diuresis ```
31
what considered RENAL FAILURE (RF)
``` Severe decrease in GFR.( Glomerular Filtration Rate) • Kidneys fail to remove metabolic end nitrogenous products from the blood (azotemia). • Failure to regulate electrolyte & acid – base balance of the ECF. • Two types of renal failure – Acute renal failure (ARF) – Chronic renal failure (CRF) ```
32
what is considered ACUTE RENAL FAILURE
School of Nursing & Midwifery • Sudden decline in renal function sufficient to increase plasma levels of nitrogen waste products (azotaemia) and impair water-electrolyte balance • Sudden decline in renal function with a Decrease in urinary output to less than 400ml / day
33
what are the 3 classifications of acute renal failure
``` – 1. Pre-renal - Marked decrease in renal blood flow – 2. Intra-renal - Damage of kidney structures – 3. Post-renal obstruction ```
34
what causes Pre-renal failure
``` – Hypovolemia • Massive haemorrhage • Dehydration • Excessive fluid loss due to burns – Heart failure (myocardial infarction) – Decreased vascular filling • Anaphylactic shock – Drugs and toxins • E.g. NSAIDs, endotoxins ```
35
what causes INTRA-RENAL FAILURE
– Glomerulonephritis (common) – Pyelonephritis – Acute tubular necrosis – Transfusion reactions – Nephrotoxic agents (methanol, lead, arsenic) – Antibiotics (Gentamycin and other aminoglycosides)
36
what is Glomerulonephritis
There is active proliferation of glomerular cells & an extensive inflammatory process. • The inflammation leads to a decrease in GFR, it may be transient or progress to renal failure • Manifestations: haematuria, proteinuria, salt and water retention
37
what is PYELONEPHRITIS
Refers to inflammation of the renal pelvis Mainly caused by bacterial infection Ascending infection from the bladder is the most common cause
38
what are the symptoms of ACUTE PYELONEPHRITIS
``` Fever, chills Leukocytosis, pyuria common Haematuria may be present May follow cystitis May subside w/o treatment (organisms may present in the urine for wks/mths) ```
39
ACUTE TUBULAR NECROSIS
- Extensive necrosis of tubular epithelial cells | - Most common cause of acute renal failure
40
what causes ACUTE TUBULAR NECROSIS
``` • Causes – Ischaemia •Due to massive haemorrhage  shock – Nephrotoxins •Antibiotics, heavy metals ```
41
what are the 3 phases of ACUTE TUBULAR NECROSIS
• Initiating phase – First 24-48 hours – Clinical causing event (e.g. haemorrhage, poisoning) – Abrupt decrease in urinary output  oliguria  anuria • Maintenance phase – Few days – tubular epithelium needs time to regrow – Requires supportive care ± dialysis • Recovery phase – Urinary output increases  may be excessive initially (polyuria) as new epithelium cannot yet fully reabsorb components of filtrate  normal urine output
42
what are the causes of POSTRENAL FAILURE
``` – Obstructive disorders – Calculi and strictures in the ureters – Bladder tumours, neurogenic bladder – Prostatic hyperplasia  urethral obstruction ```
43
what are the obstructive disorders
- Pelvis >Calculi >Tumors >Ureteropelvic ``` - Ureter-intrinsic >Calculi >Tumors >Clots >Sloughed papillae ``` - Ureter-extrinsic >Pregnancy >Tumors >retrioeritoneal fibrosis - Vesicoreteral reflux - Bladder >Calculi >Tumors >Functional - Urethra >Tumors >Posterior valve structure - Prostate >Hyperplasia >Carcinoma >Prostatitis
44
what are the 4 types of RENAL CALCULI
1. Calcium Stones 2. Struvite Stones 3. Uric acid stones 4. Cystine stones
45
key points of calcium stones
1. Calcium stones : 70 – 80 % ``` • Calcium oxalate, calcium phosphate or combination of the two. • Associated with concentrated calcium in the blood & urine. • Risk factors • Management ```
46
key points of struvite stones
2. Struvite stones : magnesium ammonium phosphate stones formed in alkaline urine. • These stones are always associated with urinary tract infections. • They are often called staghorn stones due to their shape • Risk factors & Managemen
47
key points of Utic acid stones
3. Uric acid stones: due to increase in uric acid in the urine • Risk factors – Gout and genetics * Not visible on x-ray films. * Management
48
what are the key points of Cystine stones
4. Cystine stones : are rare. * Genetic defect in renal transport of cystine * can be Managed
49
what are the CLINICAL MANIFESTATIONS | OF ACUTE RENAL FAILURE
• Oliguria and anuria > diuresis > recovery • ↓GFR > azotaemia (↑plasma creatinine, ↑ BUN (blood urea nitrogen) and ↑ urea) • Outcomes – Complete recovery – Progress to chronic renal failure – Death
50
Treatment Goals
Treatment goals are: 1.Identify the correct underlying cause 2.Prevent additional kidney damage 3.Restore urine output and kidney function 4.Compensate for renal impairment until kidney function is restored