week 5: skeletal muscle structure and function Flashcards

1
Q

outline the 3 muscle tenodn units

A
  • contractile element
  • series elastic element
  • parralell elastic element
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2
Q

outline the function of contractile unit?

A

active force generation
active tension

capacity for tension impacted by
- length
- fibre profile
- CSA
- fasicle length
- (stiffness)

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3
Q

function of series elastic unit

A
  • provides longditudinal energy transfer from CE to PEE
  • ‘spring storage’ of energy when muscle is tensed + stretch in SSC

provides passive tension
- stiffness
- compliance

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4
Q

function of parralell elastic unit

A
  • radioal energy transfer
  • longditudinal transfer
  • provides passive tension, eg. resistance when a muscle is passivley stretched
  • will become more stiff when a muscle is at length
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5
Q

describe the difference between structural and nueral determinants of strength

A

structural: how it is put together
- CSA
- fibre type
- pennation angle
- fasicle length
- tissue quality

nuerological: improves how it is used
- muscle unit firing frequency
- inhibition vs. frequency
- motor unit recruitment
- MU activation of antagonist/ synergists
- MU type
- central/nueral drive

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6
Q

relationship of CSA/ pennation and strength?

A
  • force genration capacity is proportional to PCSA
  • increase of pcsa = increase of strength
  • increase pennation = increase force (due to angle)

resistance training can
- increase CSA of type 1, but more type 2 muscle fibres (can happen quickley but then stabilise)
- results in a higher fibre pennation which is more relative to the LOP and has higher force production
- therfore higher strength

  • growth happens outward from the muscle belly
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7
Q

relationship of fasicle length and strength?

A

fascile length (number of sacromeres in series) can impact the force:length, and force:velocity
- longer fasicles = greater velocity
- longer fasicles = greater capacity to generate optimal force at larger capacities

  • good for rapid large length-excursions of muscles. eg. sprinting, jumping
  • ecc training increases FL
  • increased ROM of excercise increases FL
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8
Q

muscle fibre type and force relationship?

A

evidence for shifting fibre types is elated to MHC (muscle heavy chain) expression
- type 2a and 2b/x have largest force production
- conversion form type 2a to 2b is evidenced

  • high velocity training has incresed expression for type 2a muscle fibres, and decrease of type 1
  • endurance traing shows an increase of type 1

however studies are not strong/long enough

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9
Q

Explain the typical time-course of adaptation of structural vs neural determinants of Strength

A

early stages of training:
- rapid changes in nueral improvements

intermediate stages of training:
- sturctural adaptations follow

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10
Q

what is the structural-nueral time course of detraining

A

within days - weeks of no training:
- nueral adaptations decline quickly

weeks-months:
- structural adaptations such as hypertropojhy of bone and muscle degrade

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11
Q

define hypertrophy

A
  • an increae of muscle fibres
  • when the rate of protien synthesis exceeds protein breakdown (anabolism>catabolism)
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12
Q

what are the major inputs that can result in hypertrophy?

A

gene transcription factors
- load changes how our genes are expressed
- up regulates myonuclei and satellite cells
- increases protien synthesis

myofibrils and mitochondria:
- myofibrils increase size and number by 30%
- mitochondria increase size and number by 30%
- increases sarcoplasmic volume and this increases cell size

satellite cells:
- are activated after a mechanical stimmulus
- creation of new myofibres
- tissue repair and growth

anabolic cell signalling:
- drives the hypertrophic response

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13
Q

describe the stucture of the MTJ and how it can be adapted?

A

the sarcolemma has extensive folding at its end, these folds interdigitate with folds in connective tissue

adaptations:
- increase in length and thickness of invaginations
- increased fibre branching
- increased collagen and structural protien content
- increased cell proliferation

results in:
- increased SA
- increases the attatchment strength
- and there is greater force distribution

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14
Q

the importance of adaptations to tendons?

A

habitual chronic load leads to:
- increase of tendon dimensions
- increase of CSA in response to loading
- improves load bearing capacity becuase there is less stress of a given force place upon it becuase stress = F/CSA

  • also increses tendon stiffness
  • which improves the effciency of the MTJ in the SSC
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15
Q

adaptations of concenctric loading during excercise?

A
  • increase in muscle size
  • increase of pennation
  • increase of strength
  • may elicit a central hypertrophic response
  • increase fibre area
  • but may result in muscle shortening
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16
Q

adaptations to eccentric loading only

A
  • increase of fibre length
  • increase in single fibre area
17
Q

adaptations to isometric loading only

A
  • significant hypertrophy
  • adaptation is affected by the length of the tissue loaded
  • larger lengths = increased hypertrophy, becuase it shifts the length at peak F generation to longer
  • increase in fasicle length
  • increase in CT stiffness
18
Q

how do different excerciese types result in nueral and structural adaptations

A

convnetional excerice using both CON and ECC loading results in specific positive adaptations

19
Q

isometric loading’s affects on nueral vs. structural adaptations

A

nueral = ++

structural= ++

20
Q

heavy loading’s impact on nueral vs. structural adaptations

A

nueral: +++

structural: ++++

21
Q

speed strength loading’s impact on nueral vs. structural adaptations

A

nueral: ++++

structural: ++

22
Q

intentionally slow (strength endurance)

A

nueral: ++

strength: ++++