Week 5 Flashcards

1
Q

What does contraction lead to?

A

Muscle tension

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2
Q

Define muscle tension

A

The Force generated by contracting muscle

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3
Q

Define load

A

The external force exerted on the muscle

Can be influenced by:
- external load + gravity
- body weight + gravity
- another muscle

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4
Q

What happens when tension is greater than the load?

A

There will be a concentric contraction and the muscle length will shorten

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5
Q

What happens when tension is less than load?

A

There will be an eccentric contraction and muscle length will lengthen

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6
Q

What happens when tension is equal to load?

A

There will be an isovolumetric contraction and muscle length will stay the same

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7
Q

What is a dynamic contraction and what are its two subcategories?

A

A dynamic contraction is one that moves the skeleton

two subcategories:
1. Isotonic: tension stays the same throughout contraction (velocity changes)
2. Isokinetic: velocity stays the same throughout contraction (tension changes)

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8
Q

What is a static contraction?

A

A contraction where no movement occurs - isometric

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9
Q

What results from more crossbridges?

A

More tension

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10
Q

What does max tension differ between?

A
  • movements due to mechanical factors
  • muscles due to morphological factors
  • contractions due to neural factors
  • physiological contexts (nutritional status, fatigue, etc…)
  • people
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11
Q

What are 3 mechanical factors that influence muscle tension

A
  1. Contraction velocity
  2. Muscle action
  3. Joint angle
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12
Q

Draw and explain the contraction velocity graph on page 4 of muscle tension I

A
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13
Q

In terms of strength, what is strongest for muscle contractions

A

Eccentric > isometric > concentric

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14
Q

How does joint angle affect tension?

A

Joint position (aka joint angle) influences muscle length

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15
Q

What else influences the relationship between force and angle other than muscle length?

A
  • other biomechanical factors
    • type of joint/level
    • location of muscle insertion
    • length of limb segments
  • morphological factors
    • xc area, pennation angle
  • individual factors
    • age, sex, training status
  • fatigue
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16
Q

What are the strongest contractions usually?

A
  • mechanically optimal
  • morphologically optimal
  • neurally optimal
  • metabolically optimal
  • psychologically optimal
  • in a body that is not experiencing fatigue
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17
Q

What is fatigue?

A

The inability to maintain a power output or force during any type of repeated or sustained muscle contractions

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18
Q

Why is understanding mechanisms of fatigue important?

A
  • to prevent/delay it in critical situations
  • to improve fatigue resistance through training
  • to optimize recovery in critical situations
  • to improve recovery rate/process through training
19
Q

What are the peripheral factors of fatigue (mainly local effects)

A
  • phosphagen depletion
  • substrate depletion
  • metabolite accumulation/ion imbalance
20
Q

What are the central factors of fatigue (global effects)

A
  • psychological factors
  • central and peripheral neural factors
  • oxygen supply
21
Q

Describe depletion of phosphagens in terms of fatigue

A

THE PRIMARY FATIGUE MECHANISM FOR PHOSPHAGEN SYSTEM

ATP and PCr deplete during very high intensity activity
- ATP can deplete to 40% of resting values
- PCr can deplete to 15% of resting values

To recover, ATP and PCr must be replenished

For half time, this takes ~20-25 seconds and for full time this takes 3-5 minutes

22
Q

Describe the accumulation of metabolites/ion imbalance in terms of fatigue

A

PRIMARY FATIGUE MECHANISM FOR ANAEROBIC LACTIC SYSTEM

Many metabolites/ions can interfere with ATP synthesis and/or muscle contraction when their quantities are too high/too low
Ex: H+, Ca2+, Ammonia/ammonium, Na+/K+, Pi

In order to recover, the body needs to reset

For half time, recovery takes ~5-8 mins and for full time, recovery takes 12-20 mins

23
Q

Describe substrate depletion (glycogen) in terms of fatigue

A

PRIMARY FATIGUE MECHANISM FOR AEROBIC SYSTEM

  • reduces rate of glycolysis which leads to reduced pyruvate available for CAC which means reduced ATP resynthesis
    • fat is probably available but fat burns in a CHO flame - can’t make oxaloacetate without burning CHO
  • reduces ability to maintain blood glucose concentration
    • can lead to CNS impairment/loss of coordination
    • bonking or hitting the wall

In order to recover, the muscle glycogen must be replaced

For half time, recovery takes ~5-6 hours w/ proper nutrition and for full time, recovery takes 1-2 days

24
Q

Describe neural fatigue in terms of central AND peripheral

A

POSSIBLE FATIGUE MECHANISM FOR ANY/ALL SYSTEMS

Central:
- occurs in brain and spinal cord
- loss of motivation and/or concentration
- reduced motor drive
- pain intolerance

Peripheral:
- occurs at neuromuscular junction
- decreased muscle excitability

25
what are 3 morphological factors that influence muscle tension?
1. cross-sectional area 2. pennation angle 3. muscle fiber type
26
what does endurance mean
the ability to sustain/repeat contractions
27
what relationship will there always be between force/power and duration/endurance
there will always be an inverse relationship
28
what is max tension directly proportional to
muscle cross-sectional area (CSA)
29
at the level of the muscle fiber, what does bigger muscle fibers mean?
- more myofibrils - more myofilaments (actin + myosin) - more possible cross-bridges
30
at the level of the muscle, what does bigger muscles mean
could mean bigger muscle fibers and/or MORE muscle fibers (+ more connective tissue) - more actin + myosin - more possible cross-bridges
31
what does physiological cross-sectional area depend on?
the arrangement of fibers - fascicles are arranged differently in different muscles - in pennate muscle, fascicles attach obliquely to tendon - direction of tension is oblique to tendon increased pennation angle means more fibers pulling in the direction of tension which means more potential cross-bridges - there is a tradeoff with shortening potential / velocity
32
what can muscle fiber types be categorized according to?
contractile properties - slow twitch (type I) - fast twitch (type II) metabolic properties - oxidative (aerobic) - oxidative/glycolytic (both) - glycolytic (anaerobic)
33
what are fibers typed via
1. muscle biopsy 2. staining
34
complete the tables on page 6 of muscle tension II
35
complete the table on page 8 of muscle tension II
36
can we make muscles stronger? if so how?
Yes we can make muscles stronger - strength gains related to dose of training - increased gains over time - increased gains with increased time under tension - increased gains with increased intensity (increased load) - gains of 25-100% are common - gains evident at: - whole muscle level - single fiber level gains are specific to muscle groups that are trained
37
can we make muscles bigger?
Yes we can make muscles bigger - muscle CAS increases 7-15% within 10-14 weeks - training leads to hypertrophy
38
what does fiber hypertrophy include?
- increased size and number of myofibrils - increased contractile protein (more XBs = more tension) - increased connective tissue
39
what is hypertrophy specific to?
muscle fiber types that are trained
40
can we increase pennation angle?
yes we can adaptations are specific to muscles that are trained
41
can we convert fiber types into the type we want?
no we cant fiber type distribution is genetically-determined and varies from muscle to muscle (highest % SO in postural muscles) we cant turn slow-twitch fibers into fast-twitch fibers or vice versa we CAN make FG better at oxidative metabolism - FOG -> FG
42
complete the table on page 11 of muscle tension II
43
complete table on page 12 of muscle tension II