week 13 Flashcards

1
Q

factors that influence EDV/preload

A

venous return
skeletal muscle pump
filling time
venous tone
venous BP
blood volume

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2
Q

factors that influence afterload

A

arterial BP
vasodilation/constriction
vascular resistance

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3
Q

factors that influence contraction force

A

preload
contractility

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4
Q

during extended heavy exercise, cardiac output remains stable but…

A

HR increases and SV decreases

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5
Q

how does decreased blood volume lead to decreased stroke volume?

A
  1. decreased blood volume leads to decreased venous return
    • decreased prelaod/decreased EDV
      • decreased SV beause less blood in the heart before contraction
    • decreased contraction force
      • lower SV bc less of the blood that is in the ventricle gets squeezed out
  2. decreased blood volume leads to lower mean arterial pressure
    • lower afterload
      • increased SV bc the heart needs to overcome less force to eject blood

overall: the effect of lower venous return outweighs the effect of lower MAP and SV drops

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6
Q

if lower blood volume leads to decreased SV, what would happen to HR?

A

HR increases in order to keep Q constant

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7
Q

copy down second slide on page 5 of CVII

A
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8
Q

review first slide on page 6 of CVII

A
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9
Q

how do we mitigate cardiovascular drift and its functional consequences?

A

stay hydrated and cool

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10
Q

write down page 7 of CVII

A
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11
Q

what happens in acute exercise?

A
  1. numerous changes (increased mvmt, emotions, body temp, local metabolites, etc…) are detected by receptors throughout the body and are communicated to the cardiovascular control center in the medulla
  2. the cardiovascular control center integrates the inputs and responds by reducing in PSNS outflow to the SA node via the vagus nerve. if needed, SNS outflow to the SA node via the accelerator nerve will also be increased
  3. the result is an increase in HR up to HRmax at max SNS stimulation
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12
Q

what are some cardiovascular challenges of exercise?

A
  • we need to pump a lot more blood per minute
  • we need to direct blood flow to the right places
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13
Q

copy down second slide on page 9 of CVII

A
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14
Q

will MAP always be closer to or further from DBP than SBP

A

MAP will always be closer to DBP than to SBP

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15
Q

diastolic vs systolic BP

A

diastolic BP: intra-arterial pressure during ventricular relaxation

systolic BP: intra-arterial pressure during ventricular contraction

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16
Q

what do SBP and HR influence?

A

SBP (force of contractions) and HR (how many contractions) influence myocardial work and myocardial VO2

17
Q

what is the formula for rate pressure product (RPP)

A

RPP provides an estimate of myocardial work aka “strain on heart”

rate pressure product (RPP) = SBP x HR / 100

18
Q

how do we selectively vasodilate/vasoconstrict different vessels?

A

smooth muscle in arterioles is regulated via:
- autonomic nervous system control (dilation/constriction in response to SNS/PSNS innervation)
- hormonal control (constriction in response to epinephrine/norepinephrine)
- intrinsic metabolic control (dilation/constriction in response to local metabolic activity)

19
Q

copy down page 12 of CVII

A
20
Q

how do patterns of blood flow distribution change during exercise?

A
  • increased O2 delivery to skeletal muscle, heart and brain
  • decreased O2 delivery to viscera
  • increased blood flow to skin at lower intensities (to help thermoregulate) but skeletal muscle takes priority at max exercise so blood flow to skin decreases
21
Q

review page 14 of CVII

A
22
Q

acute vs. chronic responses to exercise

A

acute responses: how the body responds to a single bout of exercise

chronic: how the body changes in response to chronic (repeated) exposure to exercise - training adaptations

23
Q

what are the 5 training principles

A
  1. overload
  2. progression
  3. individual differences
  4. reversibility
  5. specificity
24
Q

aerobic exercise imposes a high ___ on the heart and resistance exercise imposes a high ___ on the heart

A

volume load

pressure load

25
Q

why do endurance athletes have bradycardia (low HR) at rest?

A
  • cardiac adaptations + increased blood volume = increased SV
  • can achieve the same Q with lower HR
26
Q
A