Week 4 - Lecture - Eating Disorders Flashcards

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1
Q

definition of ED

A

Severe, psychiatric disorders characterised by a dysfunctional relationship with food and distorted perceptions about the body, that significantly impairs physical health and/or psychosocial functioning. (APA, DSM-5).

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2
Q

AN in DSM 4 compared to DSM 5

A

DSM-4:
- Refusal to keep body weight above minimal healthy level (85%)
- Fear of weight gain
- Disturbance of body experience
- Amenorrhea

DSM-5:
- Restriction of intake leading to a significantly low body weight in the context of age, sex, developmental trajectory, and physical health.
- Fear of weight gain
- Disturbance of body experience

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3
Q

subtypes of AN

A
  • Restricting
    • Binge-eating/purging
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4
Q

BN in DSM4 and 5

A

DSM-4:
- Recurrent episodes of binge-eating
- Compensatory behaviours
- Bingeing & compensation happen twice per week over at least 3 months
- Self-evaluation is unduly influenced by body shape & weight
- Not simply a phase of anorexia

DSM-5:
- Recurrent episodes of binge-eating
- Compensatory behaviours
- Bingeing & compensation happen once per week over at least 3 months
- Self-evaluation is unduly influenced by body shape & weight
- Not simply a phase of anorexia

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5
Q

binge-purge cycle

A

cycle:

strict dieting

diet slips or difficult situations arise

binge eating is triggered

purging to avoid weight gain

feelings of shame and disgust

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6
Q

binge eating disorder

A
  • Newish binge eating disorder (BED)
    • Recurrent episodes of eating significantly more food in a short period of time than most people would eat under similar circumstances; eating very fast in the absence of hunger.
    • Marked by feelings of loss of control and guilt, embarrassment, or disgust.
    • Bingeing happens once per week over at least 3 months.
    • Mild 1-3, 4-7,8-13, extreme 14+ binge periods a week
    • Can binge over 10000 at a time
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7
Q

Eating disorders: not otherwise specified; EDNOS; DSM-IV

A
  • Atypical anorexia nervosa (significant restriction and fear of weight gain but is of normal weight)
    • Low frequency bulimia nervosa (not meeting the thresholds)
    • Low frequency binge eating disorder (not meeting thresholds)
    • Purging disorder (self-induced vomiting, laxatives, exercise etc but no binging)
    • Night eating syndrome (eat lots of food a night not related to hunger)

Pica (consume non-nutritious substances - non-food)
Rumination disorder (regurgitate part digested food)
Avoidant/restrictive food intake disorder (such a restricted diet they are unable to make their nutritious needs, impact physical and psychological health)

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8
Q

sub-threshold disorders

A
  • 40-50% of cases do not fit neatly into diagnosis
    • Atypical cases (OSFED) are the largest group
    • Many people move from one diagnosis to another
    • Sub-threshold disorders are most common
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9
Q

shift away from rigid diagnoses

model

A
  • Transdiagnostic model (Waller, 1993; Fairburn et al., 2003)
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10
Q

incidence and prevalence of ED

A
  • 62% of people with eating disorders have symptoms before the age of 16 (PwC, 2016)
    • All figures taken from Westernised cultures (similar across countries)
    • Peak age of onset is slightly younger in AN (14-16 years vs 18-20 years; but many cases are younger or older).
    • BED: compared to other eating disorders, more common in males and older people.
    • Female: male ratio (approx. 20:1 > more recent suggestions = 10:1)
    • Anorexia nervosa (0.5-1.0% of teenage girls)
      ○ 21 new cases per 100000 population per year
    • Bulimia nervosa (1-2% of women aged 16-35)
      ○ 30 new cases per 100000 population per year
    • BED (epidemiological studies are lacking. Extrapolation estimates are 3.6% of women and 2.1% of men aged 18-70)
      ○ Similar to BM in terms of new cases per year but not known yet
    • OSFED (2-3% of women aged 16-35)
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11
Q

why might ED rates differ?

A

○ Definitions used for diagnosis (DSM-4 vs 4)
○ Individual assessors/therapist opinion
○ Sample - clinical cases only or self-reported
○ Other researcher variables, e.g. new cases only, not re-referrals
○ Across cultures; less ED? Less acceptable? Fewer treatment opportunities?

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12
Q

covid 19 and ED

A
  • Systematic review of 53 studies (Devoe et al., 2022)
    • Evidence to suggest that the pandemic led to:
      ○ increased diagnoses of EDs (↑15%);
      ○ increased hospital admissions for EDs (↑48%)
      ○ and increased ED symptoms.
    • Increased co-morbidity (e.g., anxiety, depression, suicidal ideation; Taquet et al., 2021)
    • Decreased access to care and treatment, changes to routine and loss of structure, social isolation.
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13
Q

diagnosis and treatment

A
  • Takes 15 months between spotting symptoms and starting treatment on average (PwC, 2015)
    • Hamilton et al., (2022) – 5.28 years between symptom onset and treatment seeking in Australian sample (n = 119).
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14
Q

death and ED

A
  • Meta-analysis from Arcelus, Mitchell, Wales & Nielsen (2011).
    ○ Elevated risk of death for all patients with eating disorders.
    § Significant increase for those with AN.
    ○ In practice: If I see 1000 patients with AN a year in my clinic, 5 will die and 1.3 will die of suicide
    ○ Patients with AN have nearly 6 times more chances of dying than people without AN
    ○ Older on assessment →higher mortality
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15
Q

disability-adjusted life years

A
  • Australian Institute of Health & Welfare
    ○ EDs are 6th in terms of disability-adjusted life years: from early mortality and reduced quality of life for ages 15-24.
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16
Q

risk factors for ED

A
  • Female
    • Adolescent/young adult
    • Sociocultural pressures and expectations “thin ideal”
    • Biological: genetic predisposition
    • Biological: serotonin dysfunction
    • Family history of depression (or other psychological illness), substance/alcohol abuse, eating disorder, obesity, chronic dieting
    • Experiences: parenting (invalidating environment), abuse, critical comments, pressures to be slim
    • Individual characteristics: low SE, perfectionism, anxiety problems, obesity, early menarche
17
Q

genetic model of ED

A

○ Eating disorders appear to run in families.
○ Strober et al (2000): female relatives of AN patient x11 more likely to develop AN.
○ Some specificity for ED type; but risk for all EDs is higher among first degree family members.
○ Heritability estimates up to 0.74 for AN
○ 55% identical twins concordant for AN.
○ Much weaker for BN (less is known about the genetic pathways for other EDs).
○ Significant genetic correlations between AN and other psychiatric disorders (e.g., schizophrenia).
○ Large genetic studies identified certain patterns of genes
that seem to be important.

18
Q

psychoanalytic (psychodynamic) model of ED

A

○ Way of understanding patients’ experiences
○ Emphasise meanings attached to symptoms and function of them
§ Restriction = success / personal effectiveness / avoidance of sexuality (Bruch, 1974)
§ Vomiting = rid oneself of traumatic sexual experience or fantasy pregnancy
§ Hunger = greed (Dare & Crowther, 1995)
○ Emphasise role of infancy and subsequent experiences in shaping a person.
§ Control
§ Avoidance of maturation

19
Q

cognitive behavioural model of ED - transdiagnostic cognitive-behavioural model

A

○ Both AN and BN (transdiagnostic)
○ Core behaviours established via positive and negative reinforcement
○ Can include cognitive state
○ Transdiagnostic = not eating disorder specific and can be applied to all eating behaviours
○ Core self-esteem = overevaluation of importance on the body and eating etc.

20
Q

significant events and ED

A

○ Sexual abuse
§ About 30% of ED patients (Connors and Morse, 1993)
□ But may be more significant in men
○ Childhood loss
§ Face validity but not supported by evidence

21
Q

family systems approach and ED

A

○ Family not the cause, but context in which ED is embedded (Eisler, 1995)
§ Symptoms as communicative acts
§ The homeostatic family - families may be resistant to change so recovery may be difficult.
§ Boundaries
§ Conflict avoidance

22
Q

sociocultural model and ED

A

○ Expression of social values
○ Culture-bound or ethnic disorder
§ Gender role conflicts – traditional feminine role may be protective (Silverstein et al., 1986); Japan.
§ Superwoman Syndrome (Thornton et al., 1991)
§ Identity: child vs independent
§ Strong but look tiny
§ Representations of beauty through unattainable levels of thinness.
§ Evidence to suggest increase in EDs when exposed to westernised ideals (e.g., introduction of TV in Fiji; Becker, 2004; social networking; Becker et al., 2011).

23
Q

neural model and ED

A

○ AN may have lower levels of chemicals in brain that might be involved in intake regulation,
§ I.e. Do not feel hungry and so do not eat.

24
Q

comorbidities

A
  • Self-harm
    • Anxiety disorders
    • Depression and other mood disorders
    • Substance use
    • Personality disorders
    • Temporal relationships are not always clear
25
Q

medical treatments

A
  • Re-feeding for nutritional deficits
    ○ Life-saving
    ○ Mood stabilisation
    ○ Inpatient care for the most unwell patients
26
Q

treatment for AN (NICE)

A
  • Individual CBT-ED (40 sessions over 40 weeks)
    • Maudsley AN treatment (MANTRA; 20 sessions)
    • Specialist supportive clinical management (20 weekly sessions)
    • ED focused focal psychodynamic therapy (40 sessions, 40 weeks)
    • AN-focused family therapy for children and young people (20 sessions over 1 year).

Focus on:
- Reaching a healthy body weight or BMI for their age
- Psychoeducation
- Monitoring of weight, mental and physical health.
- Multidisciplinary care, coordinated between services.
- Nutrition, cognitive restructuring, mood regulation, social skills, body image concern, self-esteem and relapse prevention.
- Enhancing self-efficacy

27
Q

treatment for BN (NICE)

A
  • First line of treatment is BN-focused manualised guided self-help
    • Or: Individual CBT-ED (20 sessions over 20 weeks, twice weekly sessions in the first instance).
    • BN focused family therapy for young people (or individual CBT-ED).

Focus on:
- Establishing a regular pattern on eating
- Addressing eating psychopathology (e.g., urge to binge eat in response to difficult thoughts/feelings, concerns about body shape and weight, dietary restraint).
- Psychoeducation and engagement

28
Q

treatment for BED (NICE)

A
  • First line of treatment is BED-focused manualised guided self-help
    • Or: Group CBT for EDs (16 weekly sessions over 4 months)
    • Or: Individual CBT (16-20 sessions)

Focus on:
- Psychoeducation, self-monitoring of the eating behaviour, analyse problems and goals
- Identifying binge eating cues
- Daily food intake plans
- Body exposure training (to identify and change negative beliefs about the body)
- Coping with risks and triggers

29
Q

treatment outcomes

A
  • Remission rates for EDs are poor
    ○ at 1-2 year follow up for AN, remission rates range from 13-50% (e.g., Brockmeyer et al., 2018); 30-40% in BN (Quadflieg & Fichter, 2019; Gorrell et al., 2020)
    ○ AN – if treatment efforts fail during adolescence, then at risk of “severe and enduring anorexia nervosa” (Wonderlich et al., 2020).
    ○ Equivalent outcomes for males and females (e.g., Strobel et al., 2019)
    • Lack good evidence of superiority for any therapies in:
      ○ multi-impulsive eating disorders
      ○ eating disorders characterised by exercise
      ○ most ‘atypical’ cases
      ○ the majority
    • And that is where the research is going…