Week 3C Flashcards

Management of Coronary Disease

1
Q

What are the goals of nursing care for a patient with ACS?

A

-Ischemic pain relief
-Effective coping with illness-anxiety
-Preservation of myocardium
-Participation in rehab plan
-Immediate treatment of ischemia
-Reduction of risk factors

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2
Q

What signs and symptoms should be addressed immediately in collaborative care of CAD?

A

ABC, hemodynamic stability, preliminary history

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3
Q

What should be done as an acute intervention for ACS (acute coronary syndrome)

A

-S/S
-12-lead ECG
-BLoodwork
-oxygenation (greater than 90)
-IV access
-initial meds
-reperfusion therapy

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4
Q

What are the 4 initial meds we would give to an ACS patient ?

A

ASA/Plavix/Ticagrelor

Oxygen

Nitro

Morphine

MONA Framework

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5
Q

What drugs/doses should be given to prevent platelet activation and interfere with platelet adhesion?

A

ASA (160-325 TAB)

Plavix (600mg)

Ticagrelor (180mg)

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6
Q

What drugs/doses should be given to hypoxic patients in respiratory distress (SPO2 90»)

A

Oxygen, titrate it to SAo2

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7
Q

Can high flow rates (8L/min) improve the size of infarct?

A

No. False. High flow oxygen can actually worsen size of infarct.

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8
Q

What TAB is given sublingually followed by an IV when a patient is in persistent chest pain, HTN, or heart failure?

A

Nitro. Can give x3

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9
Q

What drug should you give a pt if nitro is ineffective?

A

Initially, you’ll have increased O2 demand, BP, HR, and contractility. Then you’ll give the patient morhpine. This will subsequently decrease all of these values.

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10
Q

Explain the MONA framework

A

Morphine, Oxygen, Nitro, Aspirin- how to treat ACS

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11
Q

List some additional medications you could give to a pt with ACS

A

B-adrenergic blockers, LMWH or IV hep, ACE-inhibitors, O2Y12 inhibitors, anti-dysrhythmic medications, cholesterol lowering, stool softeners

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11
Q

When would you give pt B-blockers?

A

Within 24h, if they’re very hypertensive or bradycardia

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12
Q

When do you give a pt LMWH or IV heparin?

A

Minimally 48hr after MI, to prevent rethrombosis or acute stent thrombosis

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13
Q

When and why would you give ACE inhibitors?

A

To lower their BP and reduce vasoconstriction and fluid retention

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13
Q

What are the two types of repercussion therapy?

A

Mechanical or pharmacologic

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14
Q

Mechanical reperfusion

A

Primary percutaneous coronary intervention

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15
Q

Primary PCI

A

Angiogram, insert STENT to re-establish perfusion distal to the occlusion. Can elect for this is there is a significant occlusion and they want to take preventative action to prevent an MI.

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16
Q

Pharmacologic Reperfusion

A

Fibrinolytic Therapy

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17
Q

Fibrinolytic Therapy

A

“systemic, risk for bleeding and/or stroke”
-Steptokinase, alterplase, reteplase, tenecteplase
-STEMI only

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18
Q

TIs for PCI (percutaneous coronary intervention)

A

-Electively for chronic, stable, angina
-Urgent for unstable angina
-Emergent for MI
-1-2 vessel disease
-Perform within 120’ of first medical contact, ideally within 90’

19
Q

What are some complications of PCI ?

A

-Restensois
-Coronary artery spasm
-Puncture or damage
-Irritation to the heart
-Bleeding
-Risks of heparin use

20
Q

What are the nursing interventions for follow-up care post-PCI?

A

-Angina
-Vascular site care
-Peripheral ischemia
-Renal protection

20
Q

What meds are given 120’ following a PCI?

A

ASA 160mg
Fibrinolytic IV (STEMI only)
Plavix 300mg
UF Hep bolus 60, + cont. drip 12u/kg/hr

21
Q

What meds are given immediately for a PCI?

A

ASA 160, Plavix 300, UF hep 70U/kg, standing by for transfer to Cath lab

22
Q

Are we always worried about angina following an MI?

A

No. This may be caused by a transient coronary vasospasm, but it can also be a more serious complication

23
Q

How do we assess for kidney function and why are we worried following a PCI?

A

The dye used is bad for kidneys, as is metformin. Look at hydration, fluids, D/C of some meds

23
Q

Timing of fibrinolytic therapy

A

30’, ideally in first hour, less than 6hrs otherwise we just won’t give it because the risks are too great.

24
Q

Do we treat dysrhythmias in fibrinolytic therapy ?

A

No, they are self limited. NO tx

25
Q

What are some relative CIs for fibrinolytic therapy

A

-Active peptic ulcer disease
-Anticoagulant
-Pregnant
-Hx ischemic stroke not last 3month
-Dementia
-Sugery in last 3week
-Internal bleeding last 204wek
-Severe HTN
-Traruma or prolonged cardiopulmonary resuscitation

25
Q

What is the major complication in fibrinolytic therapy?

A

Bleeding. We are looking at superficial bleeding (IV site) or internal bleeding where we would then stop the infusion and observe for stroke symptoms.

26
Q

Who is eligible for fibrinolytic therapy?

A

-Recent onset (12hr) persistent ST-elevation, Bundle Branch blocks (BBB’s), chest pain UR Nitro, NO conditions w. predisposition to hemorrhage

27
Q

Who are we NEVER giving fibrinolytic therapy to?

A

-Internal bleeding
-Hx cerebral aneurysm/AV malformation
-Hx cerebral hemorrhage
-Ischemic stroke Hx last 3mth
-Closed head/facial trauma 3mth
-Suspected aortic dissection

28
Q

When would you elect for a coronary artery bypass graft?

A

When there are blockages but a stent is not appropriate. Not usually an emergency surgery.

29
Q

What vein is used for a coronary bypass graft?

A

Saphenous from the Breast or leg. Placed into the heart to bypass the area where there is an occlusion. Allows us to re-establich perfusion distally.

30
Q

TI for CABG

A

Left main disease, multi vessel disease, satisfactory improvement is not reached with medical management, patient is not a candidate for PCI, lifestyle limiting angina unresponsive to therapy or PCI

31
Q

Post MI ongoing assessment and care

A

1) PAIN
2) Bleeding/skin/tubes/wires
3) catheter, extremeties
4) Monitor cardio, resp, VS, O2
5) rest/sleep
6) Anxiety
7) Efficacy
8) Psychological response

32
Q

What ventricle are we US to check ejection after an MI?

33
Q

How long to discharge most patients after an MI?

34
Q

How would you educate your patient prior to discharge?

A

1-manage pain and ID PF (precipitating factors)
2-ID personal risk and lifestyle changes to make
3-Sexual activity may resume when they can climb 2 flights of stairs
4- stick to ur drug regime (duh)

35
Q

Which antiplatelet will a pt be on for the rest of their lives?

36
Q

When would you prescribe plavix in LT drug therapy?

A

Allergic to ASA or if they have a stent

37
Q

Describe dual antipltelet therapy ?

A

For one year, then change in protocol. ASA + Tricagrelor//plavix

38
Q

What is recommended for all patients with ACS ?

A

Statin therapy

39
Q

What is the statin protocol for patients?

A

Manages cholesterol. Atorvastatin 80mg daily of Rosuvastatin 20-40mg daily.

40
Q

Why do we prescribe ACE inhibitors in LT management ?

A

Regulare BO, prevent remodelling of Left ventricle,

ramipril, captopril

41
Q

Why do we prescribe beta-blockers to pts for LT drug therapy ?

A

decrease: contractility, heart rate, after load, lower O2 demand and increase supply

Metoprolol, atenolol

42
Q

Describe why and how you would take nitro in LT disease management

A

-PRN
-Before activity

Promotes peripheral vasodilation and enhances collateral flow. Increase BF to ischemic zones

43
Q

When and why do we prescribe calcium channel blockers?

A

Coronary and peripheral vasodilation, reduced contractility,used in combination with beta boclkers,

Diltiazem, verapamil, nifedipine

44
Q

What are some complications you may see in patients following an MI?

A

Arryhtmias, CHF, cariogenic shock, papillary muscle dysfunction, ventricular aneurysm, pericarditis, pulmonary embolism