Week 3B: Initial Assessment of CAD Flashcards

1
Q

WWYD: a pt presents with what you expect is a heart attack. What would you focus on in your initial assessment? (5)

A

-Baseline VS + 12-lead ECG in 10’
-Assessment of chest pain
-Associated symptoms
-Physical assessment
-Medications

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2
Q

What are some other steps in an initial assessment of a pt with a suspected heart attack (following the initial 5)? (4)

A

Personal and family Hx, environmental factors, psychosocial Hx, pt attitudes and believes about health and illness (do not get these while they are unstable, this is not top of priority list)

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3
Q

How many minutes must you do you ECG within once the patient presents to the ER?

A

10

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4
Q

How frequently do you take a patients EKG?

A

15-30’ over 2-4hr

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5
Q

What is an ST depression?

A

Lack of oxygenation, permanent damage can be avoided if we respond appropriately!

Lack of “motivation” because you’re depressed

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6
Q

What is an ST elevation?

A

More than just schema, this is indicative of infra and permenant death of heart tissue. We would proceed with an echo

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7
Q

What are the stages of an assessment and the diagnostic studies we perform?

A

12-lead ECG (in 10’ q15-30/2-4h)
Cardiac monitor
Chest Xray
Coronary angiography
Exercise stress test
Echocardiogram

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8
Q

Explain a cardiac angiography

A

Assess: Coronary arteries
Looks at: Pressure in cardiac chambers, valve function, ventricular function

Use a dye to take photos via radial or femoral access threaded up to the aorta.

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9
Q

What does a stress test tell us?

A

Ischemia, ST segment changes, arrhythmia, functional capacity, efficacy of surgical intervention

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10
Q

Who cannot receive a stress test?

A

Elderly, with mobility issues

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11
Q

What does an echo assess?

A

Myocardial structures, ventricular function, effusions, thrombus, ischemia

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12
Q

What are the lab values we’re assessing in our cardio work-up?

A

Serum cardiac markers, C-reactive protein, lipid profile, blood glucose, electrolytes, kidney function

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13
Q

What are the serum cardiac markers?

A

Troponin, serum creatinine kinase, myoglobin

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14
Q

Do serum cardiac markers rapidly change in your system?

A

No. They take time to elevate and they’re examined in a “trending” manner

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14
Q

Troponin

A

Two subsets: cTnT, cTn1

Greater specificity than CK-MB

Levels rise 3-12hr
Peak 1-2days
Return to normal over 5-10 days

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15
Q

Serum Creatinine Kinase

A

Fractionated into bands

Rises 3-12hrs, peaks in 24

Return to normal after 2-3 days

16
Q

Pt presents with chest pain, what could be the diagnosis

A

Stable or unstable angina, myocardial infarction

17
Q

A patient presents with pain that lasts 3-5 minutes and responds well to nitro. There is usually no pain at rest and there is a ST segment depression on the ECG.

A

Chronic stable angina: Episodes are predictable and Brought on by precipitating factors, so we can time medications well. A disease grounded in supply and demand. Decrease load.

18
Q

What are the variants of stable angina?

A

Silent ischemia, nocturnal angina, angina decubitus, and variant

19
Q

Silent ischemia

A

Asymptomatic, diabetes mellitus

20
Q

Nocturnal Angina

A

At night, not always while sleeping. Wear nitro patches at night

21
Q

Angina decubitus

A

Chest pain while lying down. Relieved by standing or sitting

22
Q

Variant angina

A

At rest or in response to spasm of major artery. Seen in clients w. Hx migraine, raynaids, not necessarily CAD, relieved by moderate exercise

23
Q

A pt presents with chest pain that is new, occurs at rest and is also becoming worse. It is not relieved by rest and nitro does not work. This patient also has anxiety and indigestion.

A

Unstable angina. Associated with deterioration of plaque, can lead to MI or return to a stable lesion. Increases in frequency, duration, or severity.

24
Q

What causes a myocardial infarction?

A

Severe and prolonged decrease in oxygen supply. This leads to necrosis. Usually associated with acute coronary thrombosis.

25
Q

Is it more severe the lower on an artery the area of necrosis is?

A

No. The higher up on the artery it is, the more muscle it feeds and the worse the situation.

26
Q

Zone of injury vs. zone of ischemia

A

Ischemia is the area deprived of oxygen but it is salvageable. The zone of injury is an area with compromised oxygen delivery but this is still salvageable.

27
Q

A Pt presents with severe and immobilizing chest pain, position change and NO does not relieve their discomfort. They are not diabetic. There is initially an increase in glucose, BP and HR, and then there is a decline. There is pulmonary edema as you can hear crackles in their lungs. There are S3 and S4 lung sounds as well. They also have a fever.

28
Q

If a diabetic patient is having an MI, they will experience more pain than a non-diabetic patient (T/F)

A

False. A diabetic patient might not experience any pain at all!

29
Q

What is the diagnostic criteria for a patient with an MI?

A

2/3 needed
-Chest pain more than 30’
-ECG-Q waves/ST elevation/Twave inversion
-Serum cardiac markers (troponin T, Creatinine kinase)

30
Q

Which has an ST elevation: STEMI or NSTEMI?

A

STEMI (duh)

31
Q

Which has elevated serum markers, STEMI or NSTEMI?

32
Q

Which leads to an elevation in serum markers, NSTEMI or unstable angina?

A

NSTEMI (eventually) and in NSTEMI there is an ST depression or T-wave inversion