Week 2 CV Exam & Assessment Flashcards
Chart review
Orders
Medications
Medical events
Teams involved in care
Vitals
Labs
Imaging
Surgeries
Time on bypass/on pump
DNR/I
Family
Prior notes
Infection
I&O
CM/SW notes
Orientation
Premorbid status
# of readmits, LOS, outcomes
Time OOB
Pain
Normal blood pressure
<120/<80
Elevated blood pressure
120-129/<80
Stage 1 hypertension
s 130-139
or
d 80-89
Hypertension stage 2
s >140
or
d > 90
Hypertension crisis
s > 180
and/or
d >120
Normal HR
60-100 bpm
Tachycardia HR
> 100 bpm
Bradycardia HR
< 60 bpm
SpO2 normal
> 90%
SpO2 for COPD
88-92 %
Concerning Vital signs in CV
BP: >180/90 or < 90/60; MAP <60
HR: <50, uncontrolled arrhythmia, >120 at rest
SpO2: <88-90% at rest, change in O2 demand/device
RR: <10 or >30 at rest
What factors might cause bradycardia in a CV patient
Heart block
Adverse drug reaction
Metabolic dysfunction
Post-op status
Medications
Myocarditis
Lab abnormalities
Respiratory pattern
What factors might cause tachycardia in a CV patient
Anemia
Hypotension
Infection
Anxiety
ETOH use
Fear
Pain
Substance use
What factors might cause hypoxia in a CV patient
VQ mismatch
Hypoventilation
Heart/lung disease
Infection (COVID, pneumonia)
Anemia
PE
Sleep apnea
What factors might cause hypertension in a CV patient
Lifestyle factors
Pain
Obesity
Tobacco use
Age
Comorbidities (CKD, diabetes)
What might cause hypotension in a CV patient
Medications – diuretics, pain meds, CV meds
Acute blood loss
Diastolic dysfunction
Bradycardia
Shock
Position changes
Dehydration
Arrhythmias
Vital signs absolute contraindications
New onset a fib
Non-sustained v tach
Complete heart block
Increasing frequency of PVC burden
Increased ventricular arrhythmias
New-onset chest pain
Uncontrolled arrhythmias causing hemodynamic instability or acute/subacute symptoms
Unstable angina
Use of pacing pads or temporary non-sutured pacemaker (especially femoral access)
VT storm (3+ episodes vtach in 24hr)
Pending pacemaker interrogation
Vital signs relative contraindications
Cardiac signs/symptoms dependent on baseline
Decrease in heart rate with activity
Orthostasis with increasing workload or activity level
A fib with rate control, >130-140 bpm
Cardiac enzymes and biomarkers can help indicate
damage to myocardial tissue
B type natriuretic peptide (BNP)
odds ratio ~ 30 for CHF
Creatine phosphokinase
release with muscle tissue damage
CK-MB
Cardiac muscle
Increases 3-6hr after MI; peaks 18-24hrs; normalizes after 2-3 days
Less sensitive than troponin
Can also be elevated with PE, CO poisoning, hypothyroidism
CK-BB
Brain tissue
CK-MM
skeletal muscle
Troponin
often considered gold standard for cardiac biomarker