Week 2 Arterial Insufficiency Flashcards

1
Q

what is AI and what is an example

A

decreased arterial blood flow either by arteriosclerosis (thickening and hardening) or atherosclerosis (lumen is encroached by fatty walls)

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2
Q

what is intermittent claudication

A

activity specific discomfort, that goes away within 1-5 minutes of stopping activity. you can repeat this and reproduce this

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3
Q

how can you differentiate intermittent claudication with spinal stenosis

A

AI: s/s relief with cessation of activity, and predictable with the same level of activity
SS: s/s relief with change of position

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4
Q

what is the etiology of the AI

A

ischemic rest pain, and burning when the leg is elevated, and relieved with dependency , ischemic ulcers and even gangrene

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5
Q

what are some potential risk factors

A

hyperlipidemia

hypertriglyceridemia, smoking, DM, HTN, Trauma (shoes, bites bump injury, burn, MVA), age, PAD, obesity

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6
Q

what does smoking cessation do to circulation and CAD risk

A

circulation improves within 4 weeks, and CAD risk decreases by 1/2 in 1 year.

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7
Q

how does normal DM affect AI

A

you may not feel the pain associated with AI because of neuropathy

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8
Q

AI: how does the skin look

A

dry, withered, shiny, taut, thin

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9
Q

how is the skin temp with AI

A

cooler

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10
Q

Hair?

A

no

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11
Q

how does the limb surrounding look

A

pale and dusky, with pallor with elevation and rubor with dependency

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12
Q

AI and sensation?

A

decreased

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13
Q

muscles and AI

A

atrophy, and weakness, and claw toes from weak intrinsics

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14
Q

nails and AI:

A

brittle and yellow, hard and thick

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15
Q

do you have edema with AI

A

not usually

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16
Q

what are the 6 tests you can do with your clinical vascular exam

A
palpation for temp and pulses 
capillary refill
rubor of dependency 
claudication time 
ABI
venous filling rate
17
Q

what is the pain pattern associated with AI

A
increased with elevation, and exertion (walking)
numb, tingling, cold ache with exertion 
worse at night 
wakes with pain 
needs rests when walking
18
Q

what is increasing pain indicative of

A

capsular consult, and meds, education, lifestyle modifications, revascularization.

19
Q

what are two factors that might lead to amputation

A

uncontrolled pain and poor QOL

20
Q

TF: in neuropathy with DM, you will always get pain

A

false the neuropathy can mask it

21
Q

where are AI wounds usually found

A

below the ankle, not he foot, heel, met heads, tips of the toes and in bunion areas. sometimes superior to the lateral malleolus or on the anterior leg

22
Q

what is the presentation of an AI wound

A

shallow then deep
punched out look
usually round

23
Q

drainage of an AI wound

A

minimal to none, usually dry and hard

24
Q

what is the tissue like in an AI wound

A

black or brown eschar

pale granulation tissue or a mix

25
Q

dry gangrene (drainage, odor, demarcation)

A

mummification, and no drainage, and hard, little to no odor and clear demarcation

26
Q

what is the wet gangrene like (drainage, odor, demarcation)

A

drainage, odor, flactuance/edema, erythema, less clear demarcation

27
Q

what do you do for dry gangrene

A

protect and off load and monitor for conversion to wet and wait for it to auto amputate

28
Q

what do you do for wet gangrene

A

urgent referral and vascular surgeon.

29
Q

what do you do if you think they have AI

A
  • MD refer for vascular testing, and cardiac workup
  • PT education about the disease and self care
  • safe, graded exercise
30
Q

what kinds of things do you teach the patient about self care

A
  • skin care and protection (checking shoes, keeping them warm)
  • hot water bottle at the groin for gentle warming (warmth vasodilator and can bring blood in)
  • behavior modification (smoking, diet, exercise, meds)
  • sleep (positioning)
  • wound management (protection, off-loading and care)